نتایج جستجو برای: extracellular atp

تعداد نتایج: 182107  

Journal: :The Journal of experimental biology 2001
D L Knickerbocker P L Lutz

For most vertebrates, cutting off the oxygen supply to the brain results in a rapid (within minutes) loss of ATP, the failure of ATP-dependent ion-transport process, subsequent anoxic depolarization of neuronal membrane potential and consequential neuronal death. The few species that survive brain anoxia for days or months, such as the freshwater turtle Trachemys scripta, avoid anoxic depolariz...

Journal: :Circulation research 2006
Holger K Eltzschig Tobias Eckle Alice Mager Natalie Küper Christian Karcher Thomas Weissmüller Kerstin Boengler Rainer Schulz Simon C Robson Sean P Colgan

Extracellular ATP liberated during hypoxia and inflammation can either signal directly on purinergic receptors or can activate adenosine receptors following phosphohydrolysis to adenosine. Given the association of polymorphonuclear leukocytes (PMNs) with adenine-nucleotide/nucleoside signaling in the inflammatory milieu, we hypothesized that PMNs are a source of extracellular ATP. Initial studi...

2017
Yuta Shiozaki Masaki Sato Maki Kimura Toru Sato Masakazu Tazaki Yoshiyuki Shibukawa

ATP modulates various functions in the dental pulp cells, such as intercellular communication and neurotransmission between odontoblasts and neurons, proliferation of dental pulp cells, and odontoblast differentiation. However, functional expression patterns and their biophysical properties of ionotropic ATP (P2X) receptors (P2X1-P2X7) in odontoblasts were still unclear. We examined these prope...

Journal: :American journal of physiology. Heart and circulatory physiology 2011
Francesco Vetri Haoliang Xu Lizhen Mao Chanannait Paisansathan Dale A Pelligrino

ATP is thought to be released to the extracellular compartment by neurons and astrocytes during neural activation. We examined whether ATP exerts its effect of promoting pial arteriolar dilation (PAD) directly or upon conversion (via ecto-nucleotidase action) to AMP and adenosine. Blockade of extracellular direct ATP to AMP conversion, with ARL-67156, significantly reduced sciatic nerve stimula...

Journal: :Journal of immunology 2000
J M Sanz F Di Virgilio

Endotoxin-dependent release of IL-1 beta from mouse microglial cells is a very inefficient process, as it is slow and leads to accumulation of a modest amount of extracellular cytokine. Furthermore, secreted IL-1 beta is mostly in the procytokine unprocessed form. Addition of extracellular ATP to LPS-primed microglia caused a burst of release of a large amount of processed IL-1 beta. ATP had no...

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