نتایج جستجو برای: epidermolysis bullosa

تعداد نتایج: 3618  

Journal: :British journal of nursing 2008
Elizabeth Pillay

This article is the first in a series of three focusing on the causes, clinical presentation, complications and care of adult patients affected by epidermolysis bullosa (EB), a group of rare genetic skin fragility disorders. Although the condition is rare, in some cases it presents extreme challenges both to those affected and those involved in the care of the EB patient; therefore, these artic...

Journal: :The Kurume medical journal 1995
H Hachisuka N Yamamoto H Sakihama Y Sasai

Epidermolysis bullosa is a group of inherited blistering diseases classified into three main sub-groups on the basis of the level of cleavage within the skin. In dominant dystrophic epidermolysis bullosa, characterized by cleavage below the basal lamina, two variants can be distinguished by the presence (Pasini form) or absence (Cockayne-Touraine form) of albo-papuloid lesions. The present stud...

2012
Catherine S Yang Yin Lu Anita Farhi Carol Nelson-Williams Michael Kashgarian Earl J Glusac Richard P Lifton Richard J Antaya Keith A Choate

Epidermolysis bullosa pruriginosa (EBP) is a rare subtype of dystrophic epidermolysis bullosa (DEB) characterized by intense pruritus, nodular or lichenoid lesions, and violaceous linear scarring, most prominently on the extensor extremities. Remarkably, identical mutations in COL7A1, which encodes an anchoring fibril protein present at the dermal-epidermal junction, can cause both DEB and EBP ...

2016
Valeria Carolina Alvarez Sini Tellervo Penttilä Valeria Luján Salutto Bjarne Udd Claudio Gabriel Mazia

Epidermolysis bullosa simplex with muscular dystrophy (EBS-MD; OMIM #226670) is an autosomal recessive disorder characterized by neonatal blistering and later-onset muscle weakness.

Journal: :The New England journal of medicine 2010
John E Wagner Akemi Ishida-Yamamoto John A McGrath Maria Hordinsky Douglas R Keene David T Woodley Mei Chen Megan J Riddle Mark J Osborn Troy Lund Michelle Dolan Bruce R Blazar Jakub Tolar

BACKGROUND Recessive dystrophic epidermolysis bullosa is an incurable, often fatal mucocutaneous blistering disease caused by mutations in COL7A1, the gene encoding type VII collagen (C7). On the basis of preclinical data showing biochemical correction and prolonged survival in col7 −/− mice, we hypothesized that allogeneic marrow contains stem cells capable of ameliorating the manifestations o...

Journal: :Journal of cell science 1995
S M Morley S R Dundas J L James T Gupta R A Brown C J Sexton H A Navsaria I M Leigh E B Lane

Point mutations in the keratin intermediate filament genes for keratin 5 or keratin 14 are known to cause hereditary skin blistering disorders such as epidermolysis bullosa simplex, in which epidermal keratinocytes are extremely fragile and the skin blisters on mild trauma. We show that in 2 phenotypically diverse cases of epidermolysis bullosa simplex, the keratin mutations result in a thermoi...

Journal: :Anais brasileiros de dermatologia 2010
Zilda Najjar Prado de Oliveira Alexandre M Périgo Lígia M I Fukumori Valéria Aoki

Immunological mapping, an immunofluorescence technique, is currently the method most used to diagnose and differentiate the principal types of hereditary epidermolysis bullosa, since this technique is capable of determining the level of cleavage of this mechanobullous disease.

Journal: :Journal of neonatal surgery 2015
Mahdi Ben Dhaou Saloua Ammar Hamdi Louati Hayet Zitouni Mohamed Jallouli Riadh Mhiri

Epidermolysis bullosa (EB) is an inherited blistering disorder characterized by the fragility of the skin and mucous membranes. Extracutaneous manifestations can be associated. We report a unique concomitant occurrence of EB and hypertrophic pyloric stenosis in a newborn.

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