نتایج جستجو برای: chronic lymphocytic leukemia

تعداد نتایج: 733768  

Journal: :Haematologica 2012
Viola Biberacher Thomas Decker Madlen Oelsner Michaela Wagner Christian Bogner Burkhard Schmidt Robert J Kreitman Christian Peschel Ira Pastan Christian Meyer Zum Büschenfelde Ingo Ringshausen

BACKGROUND In spite of potent first-line therapies for chronic lymphocytic leukemia, treatment remains palliative and all patients frequently relapse. Treatment options for these patients are more limited. BL22 is a recombinant protein composed of the variable region of a monoclonal antibody that binds to CD22 and of PE38, a truncated Pseudomonas exotoxin. BL22 is a very potent drug already use...

MicroRNAs are small, non-coding sequences that regulate gene expression by inducing degradation or translational inhibition of target mRNAs. These molecules control many intracellular physiological and pathological processes.Abnormal expression of these moleculs has been described in different cancers including hematopoietic cancers. According to the type of cancer and the stage, miRNA’s expres...

Journal: :Haematologica 2013
Robin Foà Ilaria Del Giudice Anna Guarini Davide Rossi Gianluca Gaidano

Genetics and molecular genetics have contributed to clarify the biological bases of the clinical heterogeneity of chronic lymphocytic leukemia. In recent years, our knowledge of the molecular genetics of chronic lymphocytic leukemia has significantly broadened, offering potential new clinical implications. Mutations of TP53 and ATM add prognostic information independently of fluorescence in sit...

Journal: :Klinicka onkologie : casopis Ceske a Slovenske onkologicke spolecnosti 2015
M Špaček

Chronic lymphocytic leukemia is one of the most common lymphoid malignancies and is characterized by a highly heterogeneous clinical course. Combined regimens, such as fludarabine, cyclophosphamide, and rituximab have led to improvements in survival in younger patients with chronic lymphocytic leukemia and have become the standard of care in fit patients. However, the majority of chronic lympho...

Journal: :Haematologica 2013
Laia Rosich Ifigènia Saborit-Villarroya Mónica López-Guerra Sílvia Xargay-Torrent Arnau Montraveta Marta Aymerich Neus Villamor Elias Campo Patricia Pérez-Galán Gaël Roué Dolors Colomer

Phosphatidylinositol-3-kinase pathway is constitutively activated in chronic lymphocytic leukemia mainly due to microenvironment signals, including stromal cell interaction and CXCR4 and B-cell receptor activation. Because of the importance of phosphatidylinositol-3-kinase signaling in chronic lymphocytic leukemia, we investigated the activity of the NVP-BKM120, an orally available pan class I ...

2014
Katarina Holanda Antonio Roberto Lucena-Araujo Adônis Quintas Taciana Mendonça Aleide Lima Luydson Richardson Vasconcelos Patrícia Moura Maria Cavalcanti Cíntia Machado Aderson Silva Araújo Marcos Andre Bezerra

BACKGROUND Infectious complications represent the main cause of morbidity and mortality in chronic lymphocytic leukemia. It has been reported that polymorphisms of the mannosebinding lectin 2 (MBL2) genes are correlated with MBL protein serum levels and, consequently, are associated with the development of infectious diseases. OBJECTIVE The purpose of this study was to investigate the possibl...

Journal: :Haematologica 2013
Jennifer R Brown Bradley Messmer Lillian Werner Matthew S Davids Evgeny Mikler Jeffrey G Supko David C Fisher Ann S LaCasce Philippe Armand Eric Jacobsen Virginia Dalton Bethany Tesar Stacey M Fernandes Sean McDonough Jerome Ritz Laura Rassenti Thomas J Kipps Donna Neuberg Arnold S Freedman

This study assessed the safety and preliminary efficacy of escalated dose subcutaneous alemtuzumab in combination with rituximab in chronic lymphocytic leukemia. Twenty-eight patients with relapsed refractory chronic lymphocytic leukemia were treated on four dosing cohorts of weekly rituximab at 375 mg/m(2) and alemtuzumab doses that started at 30 mg three times per week and escalated to weekly...

2017
Mika Shapiro Yair Herishanu Ben-Zion Katz Nili Dezorella Clare Sun Sigi Kay Aaron Polliack Irit Avivi Adrian Wiestner Chava Perry

A novel therapeutic approach in cancer, attempting to stimulate host anti-tumor immunity, involves blocking of immune checkpoints. Lymphocyte activation gene 3 (LAG3) is an immune checkpoint receptor expressed on activated/exhausted T cells. When engaged by the major histocompatibility complex (MHC) class II molecules, LAG3 negatively regulates T-cell function, thereby contributing to tumor esc...

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