نتایج جستجو برای: cardiac fibrosis

تعداد نتایج: 376764  

Journal: :research in cardiovascular medicine 0
zahra alizadeh-sani rajaei cardiovascular medical and research center, tehran university of medical sciences, tehran, ir iran; rajaie cardiovascular medical and research center, vali-asr ave, niayesh blvd, tehran, ir iran, tel: +98-2122017669 , fax: +98-2122017669 anoushiravan vakili-zarch rajaei cardiovascular medical and research center, tehran university of medical sciences, tehran, ir iran majid kiavar rajaei cardiovascular medical and research center, tehran university of medical sciences, tehran, ir iran behdad bahadorian rajaei cardiovascular medical and research center, tehran university of medical sciences, tehran, ir iran abas nabavi rajaei cardiovascular medical and research center, tehran university of medical sciences, tehran, ir iran

a 64-year-old female with history of previous aortoiliac occlusion and aortoiliac bypass operation four months ago presented with dyspnea, ascites and leg edema. she has been suffering from bloody diarrhea since two weeks earlier. laboratory data showed important eosinophilia and stool examination was positive for strongyloides stercoralis. patient had clinical signs of heart failure. a cardiac...

Journal: :American journal of physiology. Heart and circulatory physiology 2012
Xiangbin Xu Fan Ding Jinjiang Pang Xue Gao Rong-Kun Xu Wei Hao Ji-Min Cao Chen Chen

Cardiac fibrosis is a hallmark of heart disease and plays a vital role in cardiac remodeling during heart diseases, including hypertensive heart disease. Hexarelin is one of a series of synthetic growth hormone secretagogues (GHSs) possessing a variety of cardiovascular effects via action on GHS receptors (GHS-Rs). However, the role of hexarelin in cardiac fibrosis in vivo has not yet been inve...

Journal: :Journal of the American College of Cardiology 2004
Alan R Collins Janet Schnee Wei Wang Sarah Kim Michael C Fishbein Dennis Bruemmer Ronald E Law Susanne Nicholas Robert S Ross Willa A Hsueh

OBJECTIVES Osteopontin (OPN) is upregulated in left ventricular hypertrophy and is stimulated by angiotensin II (AngII). Our objective was to determine whether mice deficient in OPN would be protected from AngII-induced cardiac fibrosis. BACKGROUND Interstitial fibrosis can lead to myocardial dysfunction and ultimately heart failure. Osteopontin activates integrins that regulate cell adhesion...

Journal: :Current Opinion in Physiology 2021

2016
Jin Yang Konstantinos Savvatis Jong Seok Kang Peidong Fan Hongyan Zhong Karen Schwartz Vivian Barry Amanda Mikels-Vigdal Serge Karpinski Dmytro Kornyeyev Joanne Adamkewicz Xuhui Feng Qiong Zhou Ching Shang Praveen Kumar Dillon Phan Mario Kasner Begoña López Javier Diez Keith C. Wright Roxanne L. Kovacs Peng-Sheng Chen Thomas Quertermous Victoria Smith Lina Yao Carsten Tschöpe Ching-Pin Chang

Interstitial fibrosis plays a key role in the development and progression of heart failure. Here, we show that an enzyme that crosslinks collagen-Lysyl oxidase-like 2 (Loxl2)-is essential for interstitial fibrosis and mechanical dysfunction of pathologically stressed hearts. In mice, cardiac stress activates fibroblasts to express and secrete Loxl2 into the interstitium, triggering fibrosis, sy...

Journal: :Frontiers in Bioscience 2009

Journal: :Современные проблемы науки и образования (Modern Problems of Science and Education) 2021

Journal: :Circulation Research 2004

2017
Tong Liu Deli Song Jianzeng Dong Pinghui Zhu Jie Liu Wei Liu Xiaohai Ma Lei Zhao Shukuan Ling

Myocardial fibrosis is an important part of cardiac remodeling that leads to heart failure and death. Myocardial fibrosis results from increased myofibroblast activity and excessive extracellular matrix deposition. Various cells and molecules are involved in this process, providing targets for potential drug therapies. Currently, the main detection methods of myocardial fibrosis rely on serum m...

Journal: :JACC: Basic to Translational Science 2018

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