نتایج جستجو برای: amyloid deposition

تعداد نتایج: 126596  

Journal: :Neurobiology of aging 2012
Timo Grimmer Maximilian Faust Florian Auer Panagiotis Alexopoulos Hans Förstl Gjermund Henriksen Robert Perneczky Christian Sorg Behrooz H Yousefi Alexander Drzezga Alexander Kurz

Impaired amyloid clearance probably contributes to increased amyloid deposition in sporadic Alzheimer's disease (AD). Diminished perivascular drainage due to cerebral small-vessel disease (CSVD) has been proposed as a cause of reduced amyloid clearance. White matter hyperintensities (WMHs) are considered to reflect CSVD and can be measured using fluid-attenuated inversion recovery (FLAIR) magne...

2014
Steffen Burgold Severin Filser Mario M Dorostkar Boris Schmidt Jochen Herms

A major neuropathological hallmark of Alzheimer's disease is the deposition of amyloid plaques in the brains of affected individuals. Amyloid plaques mainly consist of fibrillar β-amyloid, which is a cleavage product of the amyloid precursor protein. The amyloid-cascade-hypothesis postulates Aβ accumulation as the central event in initiating a toxic cascade leading to Alzheimer's disease pathol...

Journal: :American journal of nuclear medicine and molecular imaging 2013
Ryogo Minamimoto Kazuo Kubota Kenji Ishii Miyako Morooka Momoko Okasaki Yoko Miyata Kazuhiko Nakajima Takashi Sato Toru Igari Risen Hirai Osamu Okazaki

The definitive diagnosis of amyloidosis is made histologically with Congo red stain. Noninvasive imaging techniques for amyloidosis are beneficial for early and definite diagnosis of amyloid deposition in the body. (99m)Tc-aprotinin has the benefit of detecting amyloid deposits mainly in the heart, but it can also detect a wide range of lesions in other locations. The usefulness and limitations...

Journal: :Archives of neurology 2012
Yafei Huang Rachel Potter Wendy Sigurdson Tom Kasten Rose Connors John C Morris Tammie Benzinger Mark Mintun Tim Ashwood Mats Ferm Samantha L Budd Randall J Bateman

OBJECTIVES To investigate dynamic changes in human plasma β-amyloid (Aβ) concentrations, evaluate the effects of aging and amyloidosis on these dynamics, and determine their correlation with cerebrospinal fluid (CSF) Aβ concentrations. DESIGN A repeated plasma and CSF sampling study. SETTING The Washington University School of Medicine in St Louis, Missouri. PARTICIPANTS Older adults with...

Journal: :International journal of clinical and experimental pathology 2014
Jinping Liu Jie Lan Peng Zhao Fang Zheng Jingjing Song Peilan Zhang Xuguo Sun

Transthyretin (TTR) is a major amyloid fibril protein found in patients with familial amyloidotic polynuropathy (FAP) and senile systemic amyloidosis (SSA). Mainly synthesized in the live, TTR is transferred in the form of tetramer bound with thyroxine, retinol-binding protein (RBP) and lipoprotein in the blood. The aim of this study was to demonstrate the presence of amyloid substances in the ...

2011
Hisako Muramatsu1 Katsunori Yokoi Lan Chen Keiko Ichihara-Tanaka Terutoshi Kimura Takashi Muramatsu

BACKGROUND Midkine is a heparin-binding cytokine involved in cell survival and various inflammatory processes. Midkine accumulates in senile plaques of patients with Alzheimer's disease, while it counteracts the cytotoxic effects of amyloid β-peptide and inhibits its oligomerization. The present study was conducted to understand the role of midkine upon plaque formation of amyloid β-peptide. ...

Journal: :Brain : a journal of neurology 2014
Sigan L Hartley Benjamin L Handen Darlynne A Devenny Regina Hardison Iulia Mihaila Julie C Price Annie D Cohen William E Klunk Marsha R Mailick Sterling C Johnson Bradley T Christian

Nearly all adults with Down syndrome show neuropathology of Alzheimer's disease, including amyloid-β deposition, by their fifth decade of life. In the current study, we examined the association between brain amyloid-β deposition, assessed via in vivo assessments of neocortical Pittsburgh compound B, and scores on an extensive neuropsychological battery of measures of cognitive functioning in 63...

2014
Timo Grimmer Oliver Goldhardt Liang-Hao Guo Behrooz H. Yousefi Stefan Förster Alexander Drzezga Christian Sorg Panagiotis Alexopoulos Hans Förstl Alexander Kurz Robert Perneczky

OBJECTIVE Impaired amyloid clearance has been proposed to contribute to β-amyloid deposition in sporadic late-onset Alzheimer's disease (AD). Low density lipoprotein receptor-related protein 1 (LRP-1) is involved in the active outward transport of β-amyloid across the blood-brain barrier (BBB). The C667T polymorphism (rs1799986) of the LRP-1 gene has been inconsistently associated with AD in ge...

Journal: :Alzheimer's & dementia : the journal of the Alzheimer's Association 2016
Patrick J Lao Tobey J Betthauser Ansel T Hillmer Julie C Price William E Klunk Iulia Mihaila Andrew T Higgins Peter D Bulova Sigan L Hartley Regina Hardison Rameshwari V Tumuluru Dhanabalan Murali Chester A Mathis Annie D Cohen Todd E Barnhart Darlynne A Devenny Marsha R Mailick Sterling C Johnson Benjamin L Handen Bradley T Christian

INTRODUCTION In Down syndrome (DS), the overproduction of amyloid precursor protein is hypothesized to predispose young adults to early expression of Alzheimer-like neuropathology. METHODS PET imaging with carbon 11-labeled Pittsburgh compound B examined the pattern of amyloid-β deposition in 68 nondemented adults with DS (30-53 years) to determine the relationship between deposition and norm...

Journal: :Brain : a journal of neurology 2016
Hwamee Oh Cindee Madison Suzanne Baker Gil Rabinovici William Jagust

SEE HANSSON AND GOURAS DOI101093/AWW146 FOR A SCIENTIFIC COMMENTARY ON THIS ARTICLE: Although some brain regions such as precuneus and lateral temporo-parietal cortex have been shown to be more vulnerable to Alzheimer's disease than other areas, a mechanism underlying the differential regional vulnerability to Alzheimer's disease remains to be elucidated. Using fluorodeoxyglucose and Pittsburgh...

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