نتایج جستجو برای: agpat2 mutation

تعداد نتایج: 291444  

E Rezaei , H Dastsooz , H Faraji , J Manoochehri , Kh Sadeghi , M Fardaei , R Masoumi Dehshiri , S Mohammadi , T Moradi ,

Background Wilson disease (WD) is a rare autosomal recessive disorder, which leads to copper metabolism, due to mutations in ATP7B gene. The gene responsible for WD consists of 21 exons that span a genomic region of about 80 kb and encodes a copper transporting P-type ATPase (ATP7B), a protein consisting of 1465 amino acids. Identifying mutation in ATP7B gene is important to find carrier i...

بهار, بابک, توگه, غلام‌رضا, درگاهی, حسین, علی‌مقدم, کامران, عین‌الهی, ناهید, غفاری, سیدحمید‌اله, قوام‌زاده, اردشیر, موسوی, اسداله, نادعلی, فاطمه, چهاردولی, بهرام, کریم‌زاده, پریسا, ‌فردوسی, شیرین,

Background: JAK2 is a nonreceptor tyrosine kinase that plays a major role in myeloid disorders. This mutation is characterized by a G to T transverse at nucleotide 1849 in exon 12 of the JAK2 gene, located on the chromosome 9p, leading to a substitution of valine to phenylalanine at amino acid position 617 in the JAK2 protein. In this study we compared the amplification refractory mutation (ARM...

ذاکر کیش, مهرنوش, سمرباف زاده, علیرضا, مروج آل‌علی, ارمغان, مولا, کریم,

Background: Behcet’s disease (BD) is a multisystemic inflammatory disease with unknown origin characterized by recurrent oral aphtous ulcers, genital, ocular and skin lesions. A single point mutation 1691G to A in the factor V gene increases the risk of venous thrombosis. This study designed to determine factor V Leiden mutation in Behcet’s disease, and to find out it's relationship with the cl...

A. Karami, A. Khalilpoor F. Biramijamal M. Eshraghi M. Ghanei S. Arjmand

Objective Mustard gas (MG) is a poisoning chemical, mutagenic and carcinogenic alkylating agent. It is used during World War I and also Iran-Iraq conflict. The p53 tumor suppressor gene is involved in the pathogenesis of malignant disease. The aim of this study is to determine possible mutation in p53 gene of lung sample from mustard gas exposed patients. Material and Methods Twelve lung bio...

Jalali, Hossein, Mahdavi, Mohammad Reza , Shekarriz, Ramin,

Background and purpose: 5-Flourouracil (5-FU) is one of the most common chemical drugs used in chemotherapy of patients with cancers. Dihydropyrimidine dehydrogenase (DPD) is a critical enzyme in the catabolism of 5-FU. More than 80% of the administered 5-FU is catabolized by DPD. c.1905+1G>A mutation on DPD gene is the most important mutation associated with DPD enzymatic deficiency which lead...

abdi rad, isa, bagheri, morteza, Kavosi, Negin, khadem vatani, kamal, Mohammad Zad, Mir Hossein Seyed, rahimi, Behzad, Rostamzadeh, Alireza,

Background & Aims: Recent studies have shown that some of the MEFV gene mutations are common in patients with coronary artery disease. The present study was designed to investigate the presence or absence of E148Q mutation in exon 2 of MEFV gene in patients with premature coronary artery disease. Materials & Methods: In this study, 90 patients with coronary artery disease were voluntarily sele...

Fatemeh Fahmi Mohsen Musaviun Saeid Reza Khatami Seyed Reza Kazemi Nezhad,

Glucose-6-phosphate dehydrogenase (G6PD) deficiency is one of the most common hereditary enzymatic disorders in human, increases the vulnerability of erythrocytes to oxidative stress. It is also characterized by remarkable molecular and biochemical heterogeneity. According to previous investigations, G6PD Cosenza (G1376C) is a common G6PD mutation in some parts of Iran. Therefore in the present...

Pezhman Fard-Esfahani, Shohreh Khatami

  Background and Objective: Familial hypercholesterolemia (FH) is an autosomal trait, which is caused by mutations in Low Density Lipoprotein Receptor (LDLR) gene. FH penetrance is about 100% and worldwide prevalence for heterozygous subjects is almost 1 in 500 and for homozygous 1 in 1,000,000. The patients are at risk of premature coronary heart disease (CHD) due to defective LDLR a...

Journal: :hepatitis monthly 0
hassan dastsooz department of medical genetics, shiraz university of medical sciences, shiraz, ir iran; department of molecular medicine, shiraz university of medical sciences, shiraz, ir iran mohammad hadi imanieh shiraz transplant research center, gastroenterohepatology research center, namazi teaching hospital, shiraz university of medical sciences, shiraz, ir iran seyed mohsen dehghani shiraz transplant research center, gastroenterohepatology research center, namazi teaching hospital, shiraz university of medical sciences, shiraz, ir iran mahmood haghighat shiraz transplant research center, gastroenterohepatology research center, namazi teaching hospital, shiraz university of medical sciences, shiraz, ir iran maryam moini department of internal medicine, gastroenterology and hepatology research center, shiraz university of medical sciences, shiraz, ir iran majid fardaei department of medical genetics, shiraz university of medical sciences, shiraz, ir iran; department of molecular medicine, shiraz university of medical sciences, shiraz, ir iran; stem cell and transgenic technology research center, shiraz university of medical sciences, shiraz, ir iran; department of medical genetics, shiraz university of medical sciences, 7134853185, shiraz, ir iran. tel: +98-7112349610, fax: +98-7112349610

results using these two sets, we identified h1069q mutation in four patients, c.2335t > g mutation in three, c.3061-1g > a splice site mutation in five, c.3305t > c mutation in one, and c.3809a > g mutation in two patients. conclusions the multiplex arms assay used in this study can be an efficient, reliable, and cost effective method as a primary screen for patients with wilson disease. patien...

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