نتایج جستجو برای: بیماری lhon

تعداد نتایج: 44681  

2015
Cherise Meyerson Greg Van Stavern Collin McClelland

Leber hereditary optic neuropathy (LHON) is one of the most common inherited optic neuropathies causing bilateral central vision loss. The disorder results from point mutations in mitochondrial DNA and subsequent mitochondrial dysfunction. The primary cell type that is lost in LHON is the retinal ganglion cell, which is highly susceptible to disrupted ATP production and oxidative stress. Inheri...

2018
Leonardo Caporali Luisa Iommarini Chiara La Morgia Anna Olivieri Alessandro Achilli Alessandra Maresca Maria Lucia Valentino Mariantonietta Capristo Francesca Tagliavini Valentina Del Dotto Claudia Zanna Rocco Liguori Piero Barboni Michele Carbonelli Veronica Cocetta Monica Montopoli Andrea Martinuzzi Giovanna Cenacchi Giuseppe De Michele Francesco Testa Anna Nesti Francesca Simonelli Anna Maria Porcelli Antonio Torroni Valerio Carelli

We here report on the existence of Leber's hereditary optic neuropathy (LHON) associated with peculiar combinations of individually non-pathogenic missense mitochondrial DNA (mtDNA) variants, affecting the MT-ND4, MT-ND4L and MT-ND6 subunit genes of Complex I. The pathogenic potential of these mtDNA haplotypes is supported by multiple evidences: first, the LHON phenotype is strictly inherited a...

Journal: :Archivos de la Sociedad Espanola de Oftalmologia 2007
N Barnils E Mesa S Muñoz A Ferrer-Artola J Arruga

OBJECTIVE To ascertain the efficacy of idebenone and multivitamin treatment in Leber's hereditary optic neuropathy (LHON). METHOD Two patients diagnosed of unilateral LHON were treated with megadoses of idebenone, vitamin C and riboflavin for one year. They were examined clinically before, during and after treatment. RESULTS No improvement of visual function was observed. Despite the ideben...

Journal: :Investigative ophthalmology & visual science 2017
Nahid Akhtar Khan Periyasamy Govindaraj Nagasamy Soumittra Sonika Sharma Sundaramoorthy Srilekha Selvakumar Ambika Ayyasamy Vanniarajan Angamuthu Kanikannan Meena Megha S Uppin Challa Sundaram Parayil Sankaran Bindu Narayanappa Gayathri Arun B Taly Kumarasamy Thangaraj

Purpose Leber's hereditary optic neuropathy (LHON; OMIM 535000) is one of the most common maternally inherited mitochondrial disorders. Three mitochondrial DNA point mutations-m.3460G>A (MT-ND1), m.11778G>A (MT-ND4), and m.14484T>C (MT-ND6)-account for the majority of reported LHON cases. Only approximately 50% of males and approximately 10% of females carrying these mutations develop optic neu...

Journal: :Transactions of the American Ophthalmological Society 2006
Alfredo A Sadun Solange R Salomao Adriana Berezovsky Federico Sadun Anna Maria Denegri Peter A Quiros Filipe Chicani Dora Ventura Piero Barboni Jerome Sherman Erich Sutter Rubens Belfort Valerio Carelli

PURPOSE The authors previously presented the results of their 2001 field investigation to rural Brazil to investigate a 336-member pedigree of Leber hereditary optic neuropathy (LHON). The present work describes the yearly field investigations 2001 to 2005, utilizing a variety of highly sophisticated psychophysical and electrophysiologic procedures, in asymptomatic LHON carriers, some of whom c...

2014
Chiara La Morgia Leonardo Caporali Francesca Gandini Anna Olivieri Francesco Toni Stefania Nassetti Daniela Brunetto Carlotta Stipa Cristina Scaduto Antonia Parmeggiani Caterina Tonon Raffaele Lodi Antonio Torroni Valerio Carelli

BACKGROUND An increasing number of mitochondrial DNA (mtDNA) mutations, mainly in complex I genes, have been associated with variably overlapping phenotypes of Leber's hereditary optic neuropathy (LHON), mitochondrial encephalomyopathy with stroke-like episodes (MELAS) and Leigh syndrome (LS). We here describe the first case in which the m.4171C>A/MT-ND1 mutation, previously reported only in as...

2017
Wei Li Qiang Li Aiqin Nie Li Jiang

Mutations in mitochondrial genome are the important causes of Leber’s Hereditary Optic Neuropathy (LHON). To investigate the pathophysiology of LHON, we recently initiated a systematic mutational screening for the candidate pathogenic mutations in mitochondrial genome. In this study, we described a Chinese family with LHON. Four of nine matrilineal relatives exhibited variable degree of vision ...

2016
Sandy S.C. Hung Nicole J. Van Bergen Stacey Jackson Helena Liang David A. Mackey Damián Hernández Shiang Y. Lim Alex W. Hewitt Ian Trounce Alice Pébay Raymond C.B. Wong

Reprogramming of somatic cells into a pluripotent state is known to be accompanied by extensive restructuring of mitochondria and switch in metabolic requirements. Here we utilized Leber's hereditary optic neuropathy (LHON) as a mitochondrial disease model to study the effects of homoplasmic mtDNA mutations and subsequent oxidative phosphorylation (OXPHOS) defects in reprogramming. We obtained ...

Journal: :Journal of medical genetics 2004
P Yu-Wai-Man N Howell D A Mackey S Nørby T Rosenberg D M Turnbull P F Chinnery

L eber hereditary optic neuropathy (LHON; OMIM #535000) is a mitochondrial genetic disease that causes blindness in young adults, with an estimated minimum prevalence of 3.2 per 100 000 in the north east of England. It classically presents as bilateral subacute loss of central vision due to the focal neurodegeneration of the retinal ganglion cell layer. Over 95% of cases are principally due to ...

Journal: :Slovenian Medical Journal 2022

Leberjeva hereditarna optična nevropatija (LHON) je redka dedna mitohondrijska bolezen, ki povzroča slepoto najpogosteje pri mladih odraslih. Navadno se izrazi kot subakutna, neboleča izguba vida na eno oko, ji sledi poslabšanje drugega očesa v nekaj tednih do mesecih. Bolezen večinoma pušča trajne posledice, le nekaterih bolnikih lahko redkih primerih pride delnega spontanega izboljšanja vida....

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