نتایج جستجو برای: vhl

تعداد نتایج: 1947  

Journal: :Cancer research 1995
F Chen T Kishida F M Duh P Renbaum M L Orcutt L Schmidt B Zbar

Clear cell renal carcinomas are most frequently characterized by loss of function of both copies of the von Hippel-Lindau (VHL) disease gene, suggesting that the VHL gene product plays an important role in regulating renal cell proliferation. To directly assess the function of the VHL gene product, we transfected the wild-type VHL gene into two renal carcinoma cell lines that lacked normal expr...

Journal: :Cancer research 2002
Steven T Lott Dawn S Chandler Steven A Curley Carolyn J Foster Adel El-Naggar Marsha Frazier Louise C Strong Mercedes Lovell Ann McNeill Killary

Germ-line mutation of the von Hippel-Lindau (VHL) gene predisposes to the development of multifocal, benign lesions, including retinal and central nervous system hemangioblastomas, pheochromocytomas, and renal and pancreatic cysts. Progression to malignancy in VHL disease is associated primarily with the development of renal cell carcinoma (RCC) and pancreatic islet cell tumors (PICT). Although...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2008
Michael L Nickerson Erich Jaeger Yangu Shi Jeffrey A Durocher Sunil Mahurkar David Zaridze Vsevolod Matveev Vladimir Janout Hellena Kollarova Vladimir Bencko Marie Navratilova Neonilia Szeszenia-Dabrowska Dana Mates Anush Mukeria Ivana Holcatova Laura S Schmidt Jorge R Toro Sara Karami Rayjean Hung Gary F Gerard W Marston Linehan Maria Merino Berton Zbar Paolo Boffetta Paul Brennan Nathaniel Rothman Wong-Ho Chow Frederic M Waldman Lee E Moore

PURPOSE To provide a comprehensive, thorough analysis of somatic mutation and promoter hypermethylation of the von Hippel-Lindau (VHL) gene in the cancer genome, unique to clear cell renal cancer (ccRCC). Identify relationships between the prevalence of VHL gene alterations and alteration subtypes with patient and tumor characteristics. EXPERIMENTAL DESIGN As part of a large kidney cancer cas...

Journal: :Molecular and cellular biology 2005
Jiabin An Matthew B Rettig

Biallelic inactivating mutations of the von Hippel-Lindau tumor suppressor gene (VHL) are a hallmark of clear cell renal cell carcinoma (CCRCC), the most common histologic subtype of RCC. Biallelic VHL loss results in accumulation of hypoxia-inducible factor alpha (HIFalpha). Restoring expression of the wild-type protein encoded by VHL (pVHL) in tumors with biallelic VHL inactivation (VHL(-)(/)...

2017
Shunsaku TAKAYANAGI Akitake MUKASA Hirofumi NAKATOMI Hiroshi KANNO Jun-ichi KURATSU Ryo NISHIKAWA Kazuhiko MISHIMA Atushi NATSUME Toshihiko WAKABAYASHI Kiyohiro HOUKIN Shunsuke TERASAKA Masahiro YAO Nobuo SHINOHARA Taro SHUIN Nobuhito SAITO

von Hippel-Lindau (VHL) disease is a hereditary tumor disease in which tumors develop in multiple organs, not only as hemangioblastomas (HBs) in the central nervous system, but also as kidney tumors, pheochromocytomas, and so on. Much about the epidemiology of VHL disease remained unknown until fairly recently in Japan, leading to calls for the establishment of a VHL disease epidemiological dat...

Journal: :Head & neck 2016
Birke Bausch Ulrich Wellner Mathieu Peyre Carsten C Boedeker Frederik J Hes Mariagiulia Anglani Jose M de Campos Hiroshi Kanno Eamonn R Maher Tobias Krauss Gabriela Sansó Marta Barontini Claudio Letizia Claudia Hader Francesca Schiavi Elisabetta Zanoletti Carlos Suárez Christian Offergeld Angelica Malinoc Stefan Zschiedrich Sven Glasker Serge Bobin Olivier Sterkers Patrice Tran Ba Huy Sophie Giraud Thera Links Charis Eng Giuseppe Opocher Stephane Richard Hartmut P H Neumann

BACKGROUND Endolymphatic sac tumors (ELSTs) are, with a prevalence of up to 16%, a component of von Hippel-Lindau (VHL) disease. Data from international registries regarding heritable fraction and characteristics, germline VHL mutation frequency, and prevalence are lacking. METHODS Systematic registration of ELSTs from international centers of otorhinolaryngology and from multidisciplinary VH...

Journal: :Journal of medical genetics 1993
F M Richards E R Maher F Latif M E Phipps K Tory M Lush P A Crossey B Oostra P Enblad K H Gustavson

Von Hippel-Lindau (VHL) disease is an autosomal dominant inherited familial cancer syndrome characterised by a predisposition to the development of retinal, cerebellar, and spinal haemangioblastomas, renal cell carcinoma, and phaeochromocytoma. The gene for VHL disease has been mapped to chromosome 3p25-p26 and flanking markers identified. We report the detailed genetic mapping of the VHL disea...

Journal: :Journal of medical genetics 2000
F J Hes S McKee M J Taphoorn P Rehal R B van Der Luijt R McMahon J J van Der Smagt D Dow R A Zewald J Whittaker C J Lips F MacDonald P L Pearson E R Maher

OBJECTIVES Central nervous system haemangioblastoma (HAB) is a major feature of von Hippel-Lindau (VHL) disease, and it is estimated that about 30% of HAB patients have VHL disease. Consequently, it is widely recommended that sporadic HAB patients are screened for clinical and radiological features of VHL disease because of the risk of multiple tumours. We investigated the frequency of VHL germ...

2014
David Macías Mary Carmen Fernández-Agüera Victoria Bonilla-Henao José López-Barneo

Mutations of the von Hippel-Lindau (VHL) gene are associated with pheochromocytomas and paragangliomas, but the role of VHL in sympathoadrenal homeostasis is unknown. We generated mice lacking Vhl in catecholaminergic cells. They exhibited atrophy of the carotid body (CB), adrenal medulla, and sympathetic ganglia. Vhl-null animals had an increased number of adult CB stem cells, although the sur...

2017
Wenyi Zhu Saritha Krishna Cristina Garcia Chia-Ching John Lin Bartley D Mitchell Kenneth L Scott Carrie A Mohila Chad J Creighton Seung-Hee Yoo Hyun Kyoung Lee Benjamin Deneen

Von Hippel-Landau (VHL) protein is a potent tumor suppressor regulating numerous pathways that drive cancer, but mutations in VHL are restricted to limited subsets of malignancies. Here we identified a novel mechanism for VHL suppression in tumors that do not have inactivating mutations. Using developmental processes to uncover new pathways contributing to tumorigenesis, we found that Daam2 pro...

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