نتایج جستجو برای: tyramine

تعداد نتایج: 1374  

Journal: :Circulation 1964
F M ABBOUD J W ECKSTEIN

IN ANIMALS, reserpine depletes tissues of catecholamines, -3 reduces the response of smooth muscle to tyramine,4' 5 and potentiates the action of norepinephrine.4 6, The decreased response to tyramine probably results from depletion, since the action of this sympathomimetic amine appears to be mediated through the release of norepinephrine.4-6 The cause of hypersensitivity to exogenous norepine...

2005
JOHN W. ECKSTEIN

IN ANIMALS, reserpine depletes tissues of catecholamines, -3 reduces the response of smooth muscle to tyramine,4' 5 and potentiates the action of norepinephrine.4 6, The decreased response to tyramine probably results from depletion, since the action of this sympathomimetic amine appears to be mediated through the release of norepinephrine.4-6 The cause of hypersensitivity to exogenous norepine...

Journal: :Gut 1986
K E Murray R F Adams J W Earl K J Shaw

The free primary amines present in the faeces of 44 infants (1-18 months) with gastroenteritis have been examined by field desorption mass spectrometry of the lactone form of their fluorescamine derivatives without their prior separation. p-Tyramine, 2-phenylethylamine, the diamines, putrescine and cadaverine and several of their acyl derivatives were common constituents, but a number of other ...

2005
JOHN W. ECKSTEIN

IN ANIMALS, reserpine depletes tissues of catecholamines, -3 reduces the response of smooth muscle to tyramine,4' 5 and potentiates the action of norepinephrine.4 6, The decreased response to tyramine probably results from depletion, since the action of this sympathomimetic amine appears to be mediated through the release of norepinephrine.4-6 The cause of hypersensitivity to exogenous norepine...

2005
JOHN W. ECKSTEIN

IN ANIMALS, reserpine depletes tissues of catecholamines, -3 reduces the response of smooth muscle to tyramine,4' 5 and potentiates the action of norepinephrine.4 6, The decreased response to tyramine probably results from depletion, since the action of this sympathomimetic amine appears to be mediated through the release of norepinephrine.4-6 The cause of hypersensitivity to exogenous norepine...

Journal: :Japanese journal of pharmacology 1965
A Sakuma Y Nonomura H Kanamura M Otsuka

cium was omitted from the medium, and markedly inhibited by the addition of cocaine. 3. Addition of reserpine or tyramine stimulated the release of catecholamine from the slices. The effect of reserpine was diminished in calcium free medium, but that of tyramine was not affected in calcium free medium. Cocaine had a weak inhibitory effect on the release of catecholamine caused by reserpine but ...

2016
Jolanta Kochana Karolina Wapiennik Paweł Knihnicki Aleksandra Pollap Paula Janus Marcin Oszajca Piotr Kuśtrowski

A voltammetric biosensor based on tyrosinase (TYR) was developed for determination of tyramine. Carbon material (multi-walled carbon nanotubes or mesoporous carbon CMK-3-type), polycationic polymer-i.e., poly(diallyldimethylammonium chloride) (PDDA), and Nafion were incorporated into titania dioxide sol (TiO2) to create an immobilization matrix. The features of the formed matrix were studied by...

Journal: :The Journal of neuroscience : the official journal of the Society for Neuroscience 2007
Björn Brembs Frauke Christiansen Hans Joachim Pflüger Carsten Duch

Insect flight is one of the fastest, most intense and most energy-demanding motor behaviors. It is modulated on multiple levels by the biogenic amine octopamine. Within the CNS, octopamine acts directly on the flight central pattern generator, and it affects motivational states. In the periphery, octopamine sensitizes sensory receptors, alters muscle contraction kinetics, and enhances flight mu...

Journal: :Circulation research 1993
J D Thornton J F Daly M V Cohen X M Yang J M Downey

The role of catecholamines in ischemic preconditioning is unclear. Accordingly, the effects of tyramine-induced norepinephrine release and alpha 1-receptor blockade were examined. Ischemic preconditioning with a 5-minute coronary occlusion 10 minutes before a 30-minute ischemic interval resulted in only 7.7 +/- 3.1% infarction of the risk area, significantly less than that in control rabbits wi...

Journal: :Japanese Journal of Pharmacology 1966

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