نتایج جستجو برای: t cell immunodeficiency primary

تعداد نتایج: 2753082  

Introduction: Primary immunodeficiencies (PID) are rare heterogeneous disorders with defects in which one or more components of the immune system are malfunctioning. Clinical presentations of the patients according to type of immunodeficiency are variable. The majority of these patients are susceptible to infections depending on the type of disorder. In these patients, one of the most important...

2018
Lauren P. McLaughlin Catherine M. Bollard Michael D. Keller

Patients with primary immunodeficiency disorders (PID) have an increased risk from acute and chronic Epstein-Barr Virus (EBV) viral infections and EBV-associated malignancies. Hematopoietic stem cell transplantation (HSCT) is a curative strategy for many patients with PID, but EBV-related complications are common in the immediate post-transplant period due to delayed reconstitution of T cell im...

Journal: :Science 2004
Masayuki Shimojima Takayuki Miyazawa Yasuhiro Ikeda Elizabeth L McMonagle Hayley Haining Hiroomi Akashi Yasuhiro Takeuchi Margaret J Hosie Brian J Willett

Feline immunodeficiency virus (FIV) induces a disease similar to acquired immunodeficiency syndrome (AIDS) in cats, yet in contrast to human immunodeficiency virus (HIV), CD4 is not the viral receptor. We identified a primary receptor for FIV as CD134 (OX40), a T cell activation antigen and costimulatory molecule. CD134 expression promotes viral binding and renders cells permissive for viral en...

Journal: :Blood 2004
JiangFang Wang Susanne Marschner Terri H Finkel

The chemokine receptor, CXCR4, serves as the primary coreceptor for entry of T-cell tropic human immunodeficiency virus (HIV). Binding of either the CXC-chemokine, stromal-derived factor 1 alpha (SDF-1 alpha), or a CXCR4 antagonist, AMD3100, to CXCR4 inhibits infection of CD4(+) T cells by T-tropic HIV-1, although only SDF-1 alpha triggers T-cell signaling cascades. We have previously demonstra...

Journal: :The Journal of Experimental Medicine 2007
Afam Okoye Martin Meier-Schellersheim Jason M. Brenchley Shoko I. Hagen Joshua M. Walker Mukta Rohankhedkar Richard Lum John B. Edgar Shannon L. Planer Alfred Legasse Andrew W. Sylwester Michael Piatak Jeffrey D. Lifson Vernon C. Maino Donald L. Sodora Daniel C. Douek Michael K. Axthelm Zvi Grossman Louis J. Picker

Primary simian immunodeficiency virus (SIV) infections of rhesus macaques result in the dramatic depletion of CD4(+) CCR5(+) effector-memory T (T(EM)) cells from extra-lymphoid effector sites, but in most infections, an increased rate of CD4(+) memory T cell proliferation appears to prevent collapse of effector site CD4(+) T(EM) cell populations and acute-phase AIDS. Eventually, persistent SIV ...

Journal: :Current Opinion in Allergy & Clinical Immunology 2014

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2013
Benjamin J Chen Bjoern Chapuy Jing Ouyang Heather H Sun Margaretha G M Roemer Mina L Xu Hongbo Yu Christopher D M Fletcher Gordon J Freeman Margaret A Shipp Scott J Rodig

PURPOSE Programmed cell death ligand 1 (PD-L1) is an immunomodulatory molecule expressed by antigen-presenting cells and select tumors that engages receptors on T cells to inhibit T-cell immunity. Immunotherapies targeting the PD-1/PD-L1 pathway have shown durable antitumor effects in a subset of patients with solid tumors. PD-L1 can be expressed by Reed-Sternberg cells comprising classical Hod...

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