نتایج جستجو برای: macrophage colony stimulating factor m

تعداد نتایج: 1443433  

Journal: :Clinical and Developmental Immunology 2008
B. Rumore-Maton J. Elf N. Belkin B. Stutevoss F. Seydel E. Garrigan S. A. Litherland

Defects in macrophage colony-stimulating factor (M-CSF) signaling disrupt myeloid cell differentiation in nonobese diabetic (NOD) mice, blocking myeloid maturation into tolerogenic antigen-presenting cells (APCs). In the absence of M-CSF signaling, NOD myeloid cells have abnormally high granulocyte macrophage colony-stimulating factor (GM-CSF) expression, and as a result, persistent activation ...

Journal: :Cancer research 1986
E Naparstek T J FitzGerald M A Sakakeeny V Klassen J H Pierce B A Woda J Falco S Fitzgerald P Nizin J S Greenberger

X-irradiation of purified primary cultures of mouse bone marrow stroma or permanent cloned marrow stromal cell lines in plateau phase decreases production of macrophage progenitor cell-specific colony-stimulating factor to a plateau minimum of 40% of control levels after doses of 50 to 500 Gy delivered at 2 Gy/min. After 50 Gy there is increased bioavailability of another growth factor(s) that ...

Journal: :Oncology reports 2006
Yuming Zhang Yasushi Adachi Masayoshi Iwasaki Keizo Minamino Yasuhiro Suzuki Keiji Nakano Yasushi Koike Hiromi Mukaide Akio Shigematsu Naoko Kiriyama Chunfu Li Susumu Ikehara

It has been reported that granulocyte-colony stimulating factor (G-CSF) and granulocyte-macrophage-colony stimulating factor (GM-CSF) can mobilize endothelial progenitor cells (EPCs) in bone marrow cells (BMCs) into peripheral blood (PB) in vivo. Previously, we also reported that macrophage-colony stimulating factor (M-CSF) can mobilize EPCs into PB, which results in the rapid recovery of blood...

Journal: :Stroke 2011
Yukio Sugiyama Yoshiki Yagita Naoki Oyama Yasukazu Terasaki Emi Omura-Matsuoka Tsutomu Sasaki Kazuo Kitagawa

BACKGROUND AND PURPOSE Enhancing collateral artery growth is a potent therapeutic approach to treat cardiovascular ischemic disease from occlusive artery. Granulocyte-macrophage colony-stimulating factor (GM-CSF) has gained attention for its ability to promote arteriogenesis, ameliorating brain damage, by the mechanisms involving monocyte upregulation. However, the recent clinical study testing...

Journal: :Endocrinology 2001
K Itoh N Udagawa T Katagiri S Iemura N Ueno H Yasuda K Higashio J M Quinn M T Gillespie T J Martin T Suda N Takahashi

Bone is a major storage site for TGFbeta superfamily members, including TGFbeta and bone morphogenetic proteins. It is believed that these cytokines are released from bone during bone resorption. Recent studies have shown that both RANKL and macrophage colony-stimulating factor are two essential factors produced by osteoblasts for inducing osteoclast differentiation. In the present study we exa...

Journal: :Cancer research 1984
K Motoyoshi Y Ishizaka K Ikeda S Kajigaya K Hatake S Shionoya M Saito Y Miura

The effects of bone marrow-conditioned medium obtained from colony-stimulating factor-producing tumor-bearing nude mice (G-BM-CM) on mouse and human granulocyte-macrophage colony formation and mouse erythroid colony and burst formation were studied. Addition of G-BM-CM into the mouse granulocyte-macrophage colony-forming system containing colony-stimulating activity more strongly inhibited gran...

Journal: :Blood 1995
T Tanigawa N Nicola G A McArthur A Strasser C G Begley

The physiologic program of macrophage differentiation normally proceeds in a coordinated manner in response to several different growth factors. Although the utilization of common receptor subunits may explain in part overlapping biologic functions, mechanisms by which unique actions are mediated remain obscure. We examined growth factor-induced macrophage differentiation in M1 leukemia cells t...

2005
Takashi Okamura Michio Sata Mine Harada Koji Kato Ken Takase Akihiko Numata Koji Nagafuji Kenjiro Kamezaki Fumihiko Ishikawa Hideho Henzan Rie Imamura Toshihiro Miyamoto Goichi Yoshimoto Shoichi Inaba

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