نتایج جستجو برای: ifnl3

تعداد نتایج: 233  

2014
Alessandra Mangia Giovanni Cenderello Alessandra Orlandini Valeria Piazzolla Antonio Picciotto Massimo Zuin Alessia Ciancio Giuseppina Brancaccio Paolo Forte Vito Carretta Anna Linda Zignego Nicola Minerva Gaetano Brindicci Massimo Marignani Gianluca Svegliati Baroni Gaetano Bertino Giuseppe Cuccorese Leonardo Mottola Maria Ripoli Mario Pirisi Giovanni Li Volti

BACKGROUND Triple therapy including Telaprevir or Boceprevir still represents in many European countries the standard of care for patients with Hepatitis C Virus genotype 1 infection. The number of patients who received this treatment resulted generally lower than expected. We investigated, among naïve patients, number and characteristics of treatment candidates who were started on triple or du...

2017
Bogna Świątek-Kościelna Ewelina Kałużna Ewa Strauss Jerzy Nowak Iwona Bereszyńska Ewelina Gowin Jacek Wysocki Jolanta Rembowska Dominika Barcińska Iwona Mozer-Lisewska Danuta Januszkiewicz-Lewandowska

AIM To evaluate the association of IFNL3 (IL28B) SNP rs4803217 with severity of disease and treatment outcome in chronic hepatitis C (CHC). METHODS The study enrolled 196 CHC Polish patients (82 women and 114 men in age 20-64) infected with hepatitis C virus (HCV) genotype 1. They were treatment naïve and qualified to pegylated interferon alpha (PEG-IFN-α) and ribavirin (RBV) therapy. The ana...

2016
Camila Fernanda da Silveira Alves Camila Schultz Grott Vagner Ricardo Lunge Jorge Umberto Béria Daniela Cardoso Tietzmann Airton Tetelbom Stein Daniel Simon

Approximately one-third of the individuals infected with human immunodeficiency virus type 1 (HIV-1) are co-infected with hepatitis C virus (HCV). Co-infected patients have an increased risk for developing end-stage liver diseases. Variants upstream of the IFNL3 gene have been associated with spontaneous and treatment-induced clearance of HCV infection. Recently, a novel polymorphism was discov...

2015
Barbara Rehermann Antonio Bertoletti

Hepatitis B virus (HBV) and hepatitis C virus (HCV) cause a large proportion of acute and chronic liver disease worldwide. Over the past decades many immunological studies defined host immune responses that mediate spontaneous clearance of acute HBV and HCV infection. However, host immune responses are also relevant in the context of treatment-induced clearance of chronic HBV and HCV infection....

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