We present a calculation of soft-gluon corrections in FCNC top-quark production via anomalous t-q-g couplings. The soft anomalous dimension matrix is explicitly calculated at one-loop accuracy. This calculation allows threshold resummation at next-to-leadinglogarithm accuracy. We also derive expressions for the soft-gluon corrections at NLO and at NNLO.

BACKGROUND Modification of human airway smooth muscle (ASM) function by proinflammatory cytokines has been regarded as a potential mechanism underlying bronchial hyperresponsiveness in asthma. Human ASM cells express intercellular adhesion molecule (ICAM)-1 in response to cytokines. Synthetic ligands for peroxisome proliferator-activated receptor (PPAR)γ reportedly possess anti-inflammatory and...

In a population-based case-control study in California of 228 infants, we investigated 75 genetic variants in 20 genes and risk of gastroschisis with regard to maternal age, race/ethnicity, vitamin use, and smoking exposure. We hypothesized that genes related to vascular compromise may interact with environmental factors to affect the risk of gastroschisis. Haplotypes were constructed for 75 ge...

BACKGROUND Intra-articular injection of hyaluronan (HA) has been suggested to have a disease-modifying effect in osteoarthritis, but little is known about the possible mechanisms. OBJECTIVE To investigate the effects of HA species of different molecular mass, including 800 kDa (HA800) and 2700 kDa (HA2700), on the expression of aggrecanases (ie, ADAMTS species), which play a key role in aggre...

Repetitive transcranial magnetic stimulation (rTMS) is a form of non-invasive brain frequently used to induce neuroplasticity in the brain. Even at low intensities, rTMS has been shown modulate aspects neuronal plasticity such as motor learning and structural reorganisation neural tissue. However, impact intensity on glial cells astrocytes remains largely unknown. This study investigated change...

KCNK2 was previously shown to regulate immune-cell trafficking into the central nervous system (CNS). Kcnk2-/- mice demonstrated a more severe disease course in experimental autoimmune encephalomyelitis, an animal model of multiple sclerosis, due to an increased immune-cell migration into the CNS. An upregulation of the cellular adhesion molecules ICAM1 and VCAM1 on brain endothelial cells in K...