نتایج جستجو برای: glucuronosyltransferase gene

تعداد نتایج: 1142424  

2007
John P. Vanden Heuvel George C. Clark Michael C. Kohn

The purpose of the present experiments was to examine dose-response relationships for induction of hepatic mRNA following a single administration of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) to rats. The induction of cytochrome P450-1A1 (CYP1A1) mRNA is compared to other "dioxin-responsive" genes including UDP-glucuronosyltransferase I, plasminogen activator inhibitor 2, and transforming growt...

Journal: :Cureus 2023

Gilbert syndrome (GS) is an autosomal recessive inherited bilirubin metabolism disorder characterized by chronic unconjugated hyperbilirubinemia in the absence of hemolysis and liver disease. Primary Sjogren's (pSS), mainly occurring women, a common connective tissue disease (CTD) wherein levels are generally reduced. We report rare case pSS coexisting with GS. A 35-year-old female patient pres...

2014
Adiphol Dilokpimol Naomi Geshi

Arabinogalactan proteins are abundant cell-surface proteoglycans in plants and are involved in many cellular processes including somatic embryogenesis, cell-cell interactions, and cell elongation. We reported a glucuronosyltransferase encoded by Arabidopsis AtGlcAT14A, which catalyzes an addition of glucuronic acid residues to β-1,3- and β-1,6-linked galactans of arabinogalactan (Knoch et al. 2...

Journal: :Clinical chemistry 2008
Barbara Rantner Barbara Kollerits Marietta Anderwald-Stadler Peter Klein-Weigel Ingrid Gruber Anke Gehringer Markus Haak Mirjam Schnapka-Köpf Gustav Fraedrich Florian Kronenberg

BACKGROUND Bilirubin has antioxidative and cytoprotective properties. Low plasma concentrations of bilirubin are reportedly associated with the development of coronary and cerebrovascular disease, and bilirubin concentrations are strongly correlated with the enzyme activity of the hepatic uridine diphosphate glucuronosyltransferase (UGT1A1). The activity of UGT1A1 is influenced by a TA-repeat p...

Journal: :iranian red crescent medical journal 0
mohammad reza heydari department of pharmacology, medical school, shiraz university of medical sciences, shiraz, iran majid fardaei department of medical genetic, medical school, shiraz university of medical sciences, shiraz, iran; department of medical genetic, medical school, shiraz university of medical sciences, zand street, postal code: 71348-53185, shiraz, iran. tel: +98-7132349610, fax: +98-7132349610 mohammad rahim kadivar department of pediatric, namazi hospital, shiraz university of medical sciences, shiraz, iran abbas rezaianzadeh department of epidemiology, shiraz university of medical sciences, shiraz, iran mohammad reza panjehshahin department of pharmacology, medical school, shiraz university of medical sciences, shiraz, iran zeinab gholami bardeji department of radiology, medical imaging research center, namazi hospital, shiraz university of medical sciences, shiraz, iran

conclusions the present findings showed that the ta7/7 promoter of ugt1a1 gene accounted for a considerable number of gilbert’s syndrome cases (11.3%). the studied variations had a significant effect on creatine phosphokinase and serum total bilirubin levels. results about 78.9% of the studied subjects had normal homozygous genotypes, and 21.1% were heterozygous for the gly71arg variation. in t...

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