نتایج جستجو برای: glanzmann thrombasthenia

تعداد نتایج: 568  

Journal: :Journal of Indian Society of Pedodontics and Preventive Dentistry 2014

Journal: :international journal of preventive medicine 0
rakesh kumar manne kannan natarajan rajendra patil venkata sarath prathi beeraka swapna suneel kumar k.v

glanzmann’s thrombasthenia (gt) is an autosomal recessive inherited platelet function defect characterized by normal platelet count, prolonged bleeding time and abnormal clot retraction. this disease typically presents in infancy or early childhood and has proven to have very good prognosis. in this case study, a 22‑year‑old gt patient who also developed human immunodeficiency virus (hiv) infec...

Journal: :iranian journal of pediatric hematology and oncology 0
shima kazemzadeh department of laboratory hematology and blood banking, faculty of allied medicine, kerman university of medical sciences rezvan mohammadi pathology and stem cell research center, kerman university of medical sciences, kerman, iran.سازمان اصلی تایید شده: دانشگاه علوم پزشکی کرمان (kerman university of medical sciences) fatemeh shadkam farokhi pathology and stem cell research center, kerman university of medical sciences, kerman, iranسازمان اصلی تایید شده: دانشگاه علوم پزشکی کرمان (kerman university of medical sciences) alireza shafiian school of veterinary medicine, shahid bahonar university of kerman, kerman, iran.سازمان اصلی تایید شده: دانشگاه علوم پزشکی کرمان (kerman university of medical sciences) mohammad faranoush pediatric growth and development research center, endocrinology institute, iran university of medical science, tehran,سازمان اصلی تایید شده: دانشگاه علوم پزشکی کرمان (kerman university of medical sciences) alireza farsinejad pathology and stem cell research center, kerman university of medical sciences, kerman, iranسازمان اصلی تایید شده: دانشگاه علوم پزشکی ایران (iran university of medical sciences)

background: the most common polymorphisms identified in the methylenetetrahydrofolate reductase (mthfr) gene, c677t and a1298c lead to defective activity of this enzyme and increase the risk of venous and arterial thrombosis. there are limited investigations regarding the effects of thrombogenic polymorphisms on the clinical phenotypes of rare hereditary hemorrhagic disorders like glanzmann's t...

Journal: :Haematologica 2008
Manasi Vijapurkar Kanjaksha Ghosh Shrimati Shetty Mary Ann McLane Ana Maria Moura da Silva Diego Butera

Glanzmann thrombasthenia (GT) is an inherited bleeding disorder due to either absence or dysfunction of fibrinogen binding receptors, i.e either GPIIb (GPIIβ) or GPIIIa (GPIIIα) on platelet membrane. The complete fibrinogen receptor, i.e. GPIIβGPIII which binds fibrinogen on activated platelets involves association of these two glycoproteins. Absence of either or both of these receptors can, th...

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