نتایج جستجو برای: fragile histidine triad

تعداد نتایج: 35876  

2001
Qifeng Yang Goro Yoshimura Takaomi Suzuma Takeshi Tamaki Teiji Umemura Misa Nakamura Yasushi Nakamura Xiaojuan Wang Ichiro Mori Takeo Sakurai Kennichi Kakudo

The fragile histidine triad (Fhit) gene, which is frequently lost in many cancers, was identified as a candidate tumor suppressor gene at chromosome 3p locus 14.2. Loss of Fhit expression is an important step in tumor progression from premalignancy, to in situ, to invasive breast carcinoma. To determine whether the absence of Fhit protein correlates with other established pathological-clinical ...

Journal: :European journal of cancer 2000
C Huiping J G Jonasson B A Agnarsson B I Sigbjornsdottir K Huebner S Ingvarsson

The fragile histidine triad (FHIT) gene is a candidate tumour suppressor gene in breast and other cancers. We investigated deletions within the FHIT gene in lobular breast cancer and found that 16% of cases showed loss of heterozygosity (LOH) within the gene. We compared LOH within FHIT in lobular and ductal breast tumours and found a significant association between LOH at FHIT and the ductal h...

2012
Maria Strazzullo Annunziata Corteggio Gennaro Altamura Romina Francioso Franco Roperto Maurizio D'Esposito Giuseppe Borzacchiello

BACKGROUND Sarcoids are peculiar equine benign tumours. Their onset is associated with Bovine Papillomavirus type -1 or -2 (BPV-1/2) infection. Little is known about the molecular interplay between viral infection and neoplastic transformation. The data regarding papillomavirus infections in human species show the inactivation of a number of tumour suppressor genes as basic mechanism of transfo...

2006
Adhemar Longatto-Filho Daniela Etlinger Sônia Maria Miranda Pereira Cristina Takami Kanamura Gilda da Cunha Sérgio Makabe José A. Marques

Journal: :Zhonghua bing li xue za zhi = Chinese journal of pathology 2000
X P Hao J E Willis T G Pretlow J S Rao G T MacLennan I C Talbot T P Pretlow

Abnormal expression of the fragile histidine triad (FHIT) candidate tumor suppressor gene has been observed in a variety of human tumors, but little is known about its expression during colorectal tumorigenesis. Sections of 70 aberrant crypt foci (ACF), 55 adenomas, 84 primary colorectal carcinomas, and 13 metastatic lesions were evaluated immunohistochemically for Fhit expression. All normal c...

Journal: :Cancer research 2001
K Uematsu A Yoshimura A Gemma H Mochimaru Y Hosoya S Kunugi K Matsuda M Seike F Kurimoto K Takenaka K Koizumi Y Fukuda S Tanaka K Chin D M Jablons S Kudoh

Idiopathic pulmonary fibrosis (IPF) seems to be closely associated with lung carcinogenesis. To identify the genetic characteristics of precancerous IPF lesions in the peripheral lung, we performed PCR-based microsatellite analysis with DNA extracted from microdissected tissues; fluorescent in situ hybridization (FISH) analysis of the fragile histidine triad (FHIT) gene and immunohistochemical ...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2000
T Segawa T Sasagawa K Saijoh M Inoue

Abnormalities in structure and expression of the fragile histidine triad transcription (FHIT) gene have been reported in a variety of cancers, including endometrial cancers. A good correlation between FHIT gene alteration and loss of Fhit expression was observed in endometrial cancers, although those are the selected cases. Therefore, we investigated the association of Fhit expression with clin...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2006
Omar Kujan Richard Oliver Luca Roz Gabriella Sozzi Noel Ribeiro Robert Woodwards Nalin Thakker Philip Sloan

PURPOSE Fragile histidine triad (FHIT) expression in precursor oral lesions (POL) and oral squamous cell carcinomas (OSCC) was studied with regard to (a) the frequency of loss of FHIT expression, (b) whether loss of FHIT expression correlates with degree of dysplasia in POLs, (c) whether FHIT loss predicts high-risk POLs that are more likely to transform, and (d) whether FHIT loss in OSCCs corr...

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