نتایج جستجو برای: atenolol

تعداد نتایج: 2057  

Journal: :Clinical science and molecular medicine. Supplement 1976
R Fagard A Amery J F Deplaen P Lijnen A Missotten

1. The anti-hypertensive effect of atenolol was greater than that of bendrofluazide. 2. The change in systolic blood pressure caused by bendrofluazide showed a significant inverse correlation iwth the plasma renin concentration (r=-0-47; P less than 0-01). 3. The correlation between the change in systolic blood pressure caused by atenolol and the plasma renin concentration was not significant (...

Journal: :Circulation 2000
R Kuperstein Z Sasson

BACKGROUND Glucose and insulin levels are associated with left ventricular mass (LVM) in insulin-resistant individuals. Antihypertensive drugs have different effects on glucose and insulin metabolism (GIM) and on LVM. To evaluate whether the effects of antihypertensive therapy on LVM are associated with its effects on GIM, we compared the effects of atenolol and perindopril on these parameters ...

2015
Yoshihisa Mimura Tomoya Yasujima Kinya Ohta Katsuhisa Inoue Hiroaki Yuasa

Atenolol, a β1-adrenergic receptor blocker, is administered orally and its intestinal absorption has recently been indicated to be mediated by carrier protein and reduced markedly by ingestion of some fruit juices, such as apple and orange juices. This could be postulated to be a problem arising from the interaction of some components of fruit juices with atenolol at a transporter involved in i...

2005
GEZA SIMON

Early systemic hemodynamic adjustments to antihypertensire therapy with the cardioselective beta inhibitor, atenolol, were investigated in 12 hospitalized men, mean age 52 years, with uncomplicated mildto-moderate essential hypertension. Twice daily measurements of cardiac output (CO) by CO3 rebreathing, blood pressure by cuff, and heart rate were performed in all subjects for 3 days before and...

2015
Jia Yin Haichuan Duan Yoshiyuki Shirasaka Bhagwat Prasad Joanne Wang

Atenolol is a b-blocker widely used in the treatment of hypertension. Atenolol is cleared predominantly by the kidney by both glomerular filtration and active secretion, but the molecular mechanisms involved in its renal secretion are unclear. Using a panel of human embryonic kidney cell lines stably expressing human organic cation transporter (hOCT) 1–3, human organic anion transporter (hOAT) ...

Journal: :Hypertension 1981
G Simon J A Franciosa H J Gimenez J N Cohn

Early systemic hemodynamic adjustments to antihypertensive therapy with the cardioselective beta inhibitor, atenolol, were investigated in 12 hospitalized men, mean age 52 years, with uncomplicated mild-to-moderate essential hypertension. Twice daily measurements of cardiac output (CO) by CO2 rebreathing, blood pressure by cuff, and heart rate were performed in all subjects for 3 days before an...

Journal: :European heart journal 2007
Laurence Lucats Bijan Ghaleh Xavier Monnet Patrice Colin Alain Bizé Alain Berdeaux

AIMS Post-systolic wall thickening (PSWT) occurs after aortic valve closure. This waste of thickening does not participate in ejection. PSWT increases with myocardial ischaemia and stunning but the effects of anti-anginal drugs on PSWT during myocardial dysfunction remain unknown. The effects of two heart rate reducing agents, i.e. the beta-blocker atenolol and the selective I(f) current inhibi...

2016
Giovanna Talarico Stefania Orecchioni Katiuscia Dallaglio Francesca Reggiani Patrizia Mancuso Angelica Calleri Giuliana Gregato Valentina Labanca Teresa Rossi Douglas M. Noonan Adriana Albini Francesco Bertolini

Metformin can induce breast cancer (BC) cell apoptosis and reduce BC local and metastatic growth in preclinical models. Since Metformin is frequently used along with Aspirin or beta-blockers, we investigated the effect of Metformin, Aspirin and the beta-blocker Atenolol in several BC models. In vitro, Aspirin synergized with Metformin in inducing apoptosis of triple negative and endocrine-sensi...

Journal: :Journal of hypertension 2015
Yan Gong Caitrin W McDonough Amber L Beitelshees Nihal El Rouby Timo P Hiltunen Jeffrey R O'Connell Sandosh Padmanabhan Taimour Y Langaee Karen Hall Siegfried O F Schmidt Robert W Curry John G Gums Kati M Donner Kimmo K Kontula Kent R Bailey Eric Boerwinkle Atsushi Takahashi Toshihiro Tanaka Michiaki Kubo Arlene B Chapman Stephen T Turner Carl J Pepine Rhonda M Cooper-DeHoff Julie A Johnson

OBJECTIVE The aim of this study is to identify single-nucleotide polymorphisms (SNPs) influencing blood pressure (BP) response to the β-blocker atenolol. METHODS Genome-wide association analysis of BP response to atenolol monotherapy was performed in 233 white participants with uncomplicated hypertension in the pharmacogenomic evaluation of antihypertensive responses study. Forty-two polymorp...

Journal: :Hypertension 2004
Joanna Burns David A S G Mary Alan F Mackintosh Stephen G Ball John P Greenwood

Although the beta1-adrenergic blocking agent atenolol is an established antihypertensive therapy, its effect on peripheral sympathetic vasoconstrictor drive has remained controversial. In patients with hypertension, atenolol therapy has been reported to either increase or have no effect on peripheral vascular resistance, despite other reports showing no change or a decrease in peripheral sympat...

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