Inter-process communication (ipc) has to be fast and eeective, otherwise programmers will not use remote procedure calls (RPC), multithreading and multitasking adequately. Thus ipc performance is vital for modern operating systems, especially-kernel based ones. Surprisingly , most-kernels exhibit poor ipc performance, typically requiring 100 s for a short message transfer on a modern processor,...

Interprocess communication (IPC) is a valuable mechanism for structuring complex systems, as it allows systems to be decomposed into separate protection domains. In addition to being a good structuring mechanism, IPC is a fundamental primitive in distributed and parallel computing; the performance of distributed systems and parallel programs is often determined by the performance of IPC primiti...

BACKGROUND Several studies have shown that both early and late effects of ischemic preconditioning (IPC) protect against myocardial injury after ischemic reperfusion. METHODS AND RESULTS The purpose of this study was to evaluate the late effects of IPC on endothelial function in humans. Late phase of IPC was induced by upper limb ischemia (cuff inflation of over 200 mm Hg for 5 minutes) 6 tim...

Background/Rationale: Recent legislative amendments to Ontario’s health professional regulatory system require regulated health professionals, including speech-language pathologists and audiologists (S-LP&As), to collaborate interprofessionally where they share controlled acts. These changes have implications on the interprofessional collaboration (IPC) of regulated health professionals and the...

Renal ischemic reperfusion (IR) injury is a significant clinical problem in anesthesia and surgery. Recently, it was demonstrated that both renal ischemic preconditioning (IPC) and systemic adenosine pretreatment protect against renal IR injury. In cardiac IPC, pertussis toxin-sensitive G-proteins (i.e., G(i/o)), protein kinase C (PKC), and ATP-sensitive potassium (K+(ATP)) channels are implica...

BACKGROUND Cerebral ischemic preconditioning (IPC) protects brain against ischemic injury. Activation of Toll-like receptor 3 (TLR3) signaling can induce neuroprotective mediators, but whether astrocytic TLR3 signaling is involved in IPC-induced ischemic tolerance is not known. METHODS IPC was modeled in mice with three brief episodes of bilateral carotid occlusion. In vitro, IPC was modeled ...