نتایج جستجو برای: tnfr

تعداد نتایج: 1060  

Journal: :Journal of immunology 2000
B Balasa K Van Gunst N Jung D Balakrishna P Santamaria T Hanafusa N Itoh N Sarvetnick

Several death-signaling or death-inducing molecules have been implicated in beta cell destruction, including Fas, perforin, and TNFR-1. In this study, we examined the role of each death-signaling molecule in the IL-10-accelerated diabetes of nonobese diabetic (NOD) mice. Groups of IL-10-NOD mice, each deficient in either Fas, perforin, or TNFR-1 molecules, readily developed insulitis, and subse...

2012
Navita Kaushal Vijay Ramesh David Gozal

TNF-α plays critical roles in host-defense, sleep-wake regulation, and the pathogenesis of various disorders. Increases in the concentration of circulating TNF-α after either sleep deprivation or sleep fragmentation (SF) appear to underlie excessive daytime sleepiness in patients with sleep apnea (OSA). Following baseline recordings, mice were subjected to 15 days of SF (daily for 12 h/day from...

Journal: :Journal of immunology 2010
Laura M Snell Ann J McPherson Gloria H Y Lin Shimon Sakaguchi Pier Paolo Pandolfi Carlo Riccardi Tania H Watts

The regulation of T cell expansion by TNFR family members plays an important role in determining the magnitude of the immune response to pathogens. As several members of the TNFR family, including glucocorticoid-induced TNFR-related protein (GITR), are found on both regulatory and effector T cells, there is much interest in understanding how their effects on these opposing arms of the immune sy...

Journal: :Frontiers in Cell and Developmental Biology 2021

2014
Juan M. Zapata Sophie Lefebvre John C. Reed

TNF-Receptors Associated Factors (TRAFs) are the molecules that upon engagement of the TNF-receptor (TNFR) by a TNF-family ligand come first in contact with the activated TNFR, initially acting as docking molecules for kinases and other effector proteins that are recruited to the activated receptor. TRAFs later regulate the subcellular relocalization of the receptor-ligand complex and finally t...

Journal: :Asian Pacific journal of cancer prevention : APJCP 2014
Jun-Qin Li Hui Xue Lan Zhou Li-Hua Dong Da-Peng Wei Hua Li

OBJECTIVE The mechanism of action of fatty acid synthase (FASN) in drug tolerance of breast cancer cells with epithelial-mesenchymal transition (EMT) features was investigated. METHODS The breast cancer cell line MCF-7-MEK5 with stably occurring EMT and tumour necrosis factor-α (TNF-α) tolerance was used as the experimental model, whereas MCF-7 acted as the control. Tumour cells were implante...

Journal: :BMC Infectious Diseases 2008
Isaac Zulu Ghaniah Hassan Lungowe Njobvu RN Winnie Dhaliwal Sandie Sianongo Paul Kelly

BACKGROUND Mortality in Zambian AIDS patients is high, especially in patients with diarrhoea, and there is still unacceptably high mortality in Zambian patients just starting anti-retroviral therapy. We set out to determine if high concentrations of serum cytokines correlate with mortality. METHODS Serum samples from 30 healthy controls (HIV seropositive and seronegative) and 50 patients with...

Journal: :The Journal of clinical investigation 2012
Xueqiang Zhao Lijie Rong Xiaopu Zhao Xiao Li Xiaoman Liu Jingjing Deng Hao Wu Xia Xu Ulrike Erben Peihua Wu Uta Syrbe Joachim Sieper Zhihai Qin

TNF, an inflammatory cytokine that is enriched in the tumor microenvironment, promotes tumor growth and subverts innate immune responses to cancer cells. We previously reported that tumors implanted in TNF receptor-deficient (Tnfr-/-) mice are spontaneously rejected; however, the molecular mechanisms underlying this rejection are unclear. Here we report that TNF signaling drives the peripheral ...

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