نتایج جستجو برای: p16ink4a

تعداد نتایج: 1424  

Journal: :International journal of oncology 2010
Kazuyuki Sato Yusuke Kusama Moe Tategu Kenichi Yoshida

Two crucial cell cycle regulators, p16INK4A and p14ARF, are produced from the cyclin-dependent kinase inhibitor 2A (CDKN2A) gene locus by alternative reading frames; these regulators act as tumor suppressors during tumorigenesis. However, the molecular events incidental to the acute functional loss of CDKN2A remain a critical issue. Two pivotal regulatory pathways of cell fate determination inv...

Journal: :Biochimie 2008
R Rajasekaran C George Priya Doss C Sudandiradoss K Ramanathan Rao Sethumadhavan

In this study, we identified the most deleterious nsSNP in CDKN2A gene through structural and functional properties of its protein (p16INK4A) and investigated its binding affinity with cdk6. Out of 118 SNPs, 14 are nsSNPs in the coding region and 17 SNPs were found in the untranslated region (UTR). FastSNP suggested that 7 SNPs in the 5' UTR might change the protein expression levels. Sixty-fou...

2014
JINGBO WU XIAO-JING LI WEI ZHU XIU-PING LIU

The aim of the present study was to investigate human papillomavirus (HPV) infection and p16INK4A and p53 protein expression, to evaluate their roles in the pathological diagnosis and grading for cervical intraepithelial neoplasia (CIN). The detection of HPV DNA and p16INK4A and p53 protein expression were examined in a panel of clinical tissue samples using polymerase chain reaction or immunoh...

2015
Hao Shi Xiong Chen Cheng Lu Changmei Gu Hongwei Jiang RuiWei Meng Xun Niu Yangxin Huang Meixia Lu

BACKGROUND The p16INK4a is an important tumor suppressor gene (TSG) and aberrant methylation of promoter is known to be a major inactivation mechanism of the tumor suppressor and tumor-related genes. Aberrant TSG methylation was considered an important epigenetic silencing mechanism in the progression of head and neck squamous cell carcinoma (HNSCC). However, some studies have reported differen...

2017
Zhi-Hui He Yan Chen Ping Chen Sheng-Dong He Hui-Hui Zeng Ji-Ru Ye Da Liu Jun Cao

Background There is a growing realization that COPD, or at least emphysema, involves several processes presenting in aging and cellular senescence. Endothelial progenitor cells (EPCs) contribute to neovascularization and play an important role in the development of COPD. The gene for p16Ink4a is a major dominant senescence one. The aim of the present study was to observe changes in lung functio...

2013
Magdalena Sawicka Jeffrey Pawlikowski Stephen Wilson Dudley Ferdinando Hong Wu Peter David Adams David Andrew Gunn William Parish

The validity of the identification and classification of human cancer using antibodies to detect biomarker proteins depends upon antibody specificity. Antibodies that bind to the tumour-suppressor protein p16INK4a are widely used for cancer diagnosis and research. In this study we examined the specificity of four commercially available anti-p16INK4a antibodies in four immunological applications...

Journal: :Carcinogenesis 2003
Terence Kin-Wah Lee Kwan Man Ming-Tat Ling Xiang-Hong Wang Yong-Chuan Wong Chung-Mau Lo Ronnie Tung-Ping Poon Irene Oi-Lin Ng Sheung-Tat Fan

Inhibitors of differentiation and DNA binding-1 (Id-1) have been demonstrated to oppose Ets-mediated activation of p16INK4a. As p16INK4a protein is inactivated in hepatocellular carcinoma (HCC), we aimed to investigate the role of Id-1 in regulating p16INK4a expression during the development of HCC in HCC patients and direct ectopic Id-1 introduction into the PLC/PRF/5 HCC cell line. Sixty-two ...

Journal: :Biochemical and biophysical research communications 2008
Yanhua Zhao Yan Zhang Zhen Yang Albert Li Jianli Dong

Abnormal BRAF and p16INK4A co-exist in 60% of melanomas. BRAF mutation also occurs in 80% of benign nevi where it turns-on p16INK4A resulting in proliferative senescence; loss of p16INK4A removes the inhibitory block leading to melanoma development. Since only melanomas with wild-type BRAF have amplified CDK4 and cyclin D1 genes, p16INK4A-CDK4/6-cyclin D pathway is viewed as linearly downstream...

Journal: :International journal of oncology 2014
Takuya Kawaguchi Kazuhiro Yoshikawa Keiji Kawamoto Kunikazu Yoshimura Hideyuki Oshige Akio Asai

Potential molecular targets in neuroblastoma include ALK mutations, p16 deletion and CDK2A mutations; however, targeted therapeutics have not been developed for these factors. We developed Wr-T, a new system for intracellular peptide and protein delivery with a 30-residue sequence that mediates molecule entrapment and intracellular permeability. Wr-T was used to introduce the p16INK4a functiona...

Journal: :International journal of oncology 2009
Mohamed Alaa Makoto Suzuki Mitsuru Yoshino Lei Tian Hidemi Suzuki Kaoru Nagato Taiki Fujiwara Hironobu Wada Yasumitsu Moriya Hidehisa Hoshino Shinichiro Motohashi Shigetoshi Yoshida Kiyoshi Shibuya Kenzo Hiroshima Yukio Nakatani Aliae Abdrabou Maha Elkholy Tarek Mahfouz Ichiro Yoshino

Several studies have described p16INK4A and prostaglandin E2 (PGE2) co-alterations in various solid tumors, including non-small cell lung cancer (NSCLC). In this study, we examined the correlation between PGE2 receptor 2 (EP2) expression and p16INK4A methylation in NSCLC, and the association with clinicopathological features and prognostic significance. We retrospectively reviewed 88 NSCLC pati...

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