نتایج جستجو برای: nefl

تعداد نتایج: 100  

Journal: :Neuro-oncology 2022

Abstract BACKGROUND Pediatric high-grade gliomas (pHGGs) are an aggressive CNS tumor which often characterized by mutations in H3F3A, the gene that encodes Histone H3.3 (H3.3). A substitution of Glycine at position 34 with either Arginine or Valine (H3.3G34R/V), was recently described a large cohort pHGG samples and has been as occurring anywhere between 5-20% pHGGs. Attempts to study mechanism...

2016
Joakim Bergman Ann Dring Henrik Zetterberg Kaj Blennow Niklas Norgren Jonathan Gilthorpe Tommy Bergenheim Anders Svenningsson

OBJECTIVE In an ongoing, open-label, phase 1b study on the intrathecal administration of rituximab for progressive multiple sclerosis, an intraventricular catheter was inserted for drug delivery. The objective of this study was to characterize the limited white matter axonal injury evoked by catheter insertion by analyzing a panel of markers for tissue damage in CSF and serum. METHODS Lumbar ...

Journal: :Journal of neuropathology and experimental neurology 2004
Lars Tatenhorst Volker Senner Sylvia Püttmann Werner Paulus

Diffuse brain invasion is a major reason for poor prognosis of glioma patients. The molecular mechanisms underlying infiltration are different from those of other cancer types. To detect genes associated with glioma invasion, highly migratory clones were selected from U373MG glioma cells and from primary glioblastoma cells, and the gene expression pattern of these "fast" cells was compared with...

2016
Friederike Werheid Hamid Azzedine Eva Zwerenz Ahmet Bozkurt Marcus J. Moeller Lilian Lin Michael Mull Martin Häusler Jörg B. Schulz Joachim Weis Kristl G. Claeys

INTRODUCTION Charcot-Marie-Tooth neuropathy (CMT) is a genetically heterogeneous group of peripheral neuropathies. In addition to the classical clinical phenotype, additional features can occur. METHODS We studied a wide range of additional features in a cohort of 49 genetically confirmed CMT patients and performed a systematic literature revision. RESULTS Patients harbored a PMP22 gene alt...

2014
Christina DiVincenzo Christopher D Elzinga Adam C Medeiros Izabela Karbassi Jeremiah R Jones Matthew C Evans Corey D Braastad Crystal M Bishop Malgorzata Jaremko Zhenyuan Wang Khalida Liaquat Carol A Hoffman Michelle D York Sat D Batish James R Lupski Joseph J Higgins

We report the frequency, positive rate, and type of mutations in 14 genes (PMP22, GJB1, MPZ, MFN2, SH3TC2, GDAP1, NEFL, LITAF, GARS, HSPB1, FIG4, EGR2, PRX, and RAB7A) associated with Charcot-Marie-Tooth disease (CMT) in a cohort of 17,880 individuals referred to a commercial genetic testing laboratory. Deidentified results from sequencing assays and multiplex ligation-dependent probe amplifica...

2016
Morten Stilund Mikkel Carstensen Gjelstrup Tove Christensen Holger Jon Møller Thor Petersen

OBJECTIVES This study aimed to examine the levels of the macrophage marker sCD163 and other biomarkers at the time of diagnosis of patients with either clinically isolated syndrome (CIS) or relapsing-remitting multiple sclerosis (RRMS), and assess relation to clinical indicators of prognosis, disease activity (DA), and changes in the levels of these biomarkers at follow-up. MATERIALS AND METH...

Journal: :Human molecular genetics 2015
Bethany L Johnson-Kerner Faizzan S Ahmad Alejandro Garcia Diaz John Palmer Greene Steven J Gray Richard Jude Samulski Wendy K Chung Rudy Van Coster Paul Maertens Scott A Noggle Christopher E Henderson Hynek Wichterle

Giant axonal neuropathy (GAN) is a progressive neurodegenerative disease caused by autosomal recessive mutations in the GAN gene resulting in a loss of a ubiquitously expressed protein, gigaxonin. Gene replacement therapy is a promising strategy for treatment of the disease; however, the effectiveness and safety of gigaxonin reintroduction have not been tested in human GAN nerve cells. Here we ...

2018
Tina Likar Mensuda Hasanhodžić Nataša Teran Aleš Maver Borut Peterlin Karin Writzl

Hereditary hearing loss (HL) is a common sensory disorder, with an incidence of 1-2 per 1000 newborns, and has a genetic etiology in over 50% of cases. It occurs either as part of a syndrome or in isolation and is genetically very heterogeneous which poses a challenge for clinical and molecular diagnosis. We used exome sequencing to seek a genetic cause in a group of 56 subjects (49 probands) w...

2015
Johann Gross Heidi Olze Birgit Mazurek

We used organotypic cultures of the stria vascularis (SV), the organ of Corti (OC) and the modiolus (MOD) of newborn rats to analyze the differential expression levels and responses of cytoskeletal, myelinand neural growth factor-associated genes following preparatory injury and hypoxia. The transcript mRNA levels of the neurofilaments Nefl, Nefm, the microtubule-associated proteins Mapt, Map1a...

Journal: :Archives of neurology 2010
Sara Benedetti Stefano Carlo Previtali Silvia Coviello Marina Scarlato Federica Cerri Emanuela Di Pierri Lara Piantoni Ivana Spiga Raffaella Fazio Nilo Riva Maria Grazia Natali Sora Patrizia Dacci Maria Chiara Malaguti Elisabetta Munerati Luigi Maria Edoardo Grimaldi Maria Giovanna Marrosu Maurizio De Pellegrin Maurizio Ferrari Giancarlo Comi Angelo Quattrini Alessandra Bolino

BACKGROUND Charcot-Marie-Tooth (CMT) neuropathies are very heterogeneous disorders from both a clinical and genetic point of view. The CMT genes identified so far encode different proteins that are variably involved in regulating Schwann cells and/or axonal functions. However, the function of most of these proteins still remains to be elucidated. OBJECTIVE To characterize a large cohort of pa...

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