نتایج جستجو برای: ciliopathy

تعداد نتایج: 423  

Journal: :Journal of the American Society of Nephrology : JASN 2009
Friedhelm Hildebrandt Massimo Attanasio Edgar Otto

Nephronophthisis (NPHP), a recessive cystic kidney disease, is the most frequent genetic cause of end-stage kidney disease in children and young adults. Positional cloning of nine genes (NPHP1 through 9) and functional characterization of their encoded proteins (nephrocystins) have contributed to a unifying theory that defines cystic kidney diseases as "ciliopathies." The theory is based on the...

Journal: :The Journal of clinical investigation 2014
Yangfan P Liu I-Chun Tsai Manuela Morleo Edwin C Oh Carmen C Leitch Filomena Massa Byung-Hoon Lee David S Parker Daniel Finley Norann A Zaghloul Brunella Franco Nicholas Katsanis

Cilia are critical mediators of paracrine signaling; however, it is unknown whether proteins that contribute to ciliopathies converge on multiple paracrine pathways through a common mechanism. Here, we show that loss of cilopathy-associated proteins Bardet-Biedl syndrome 4 (BBS4) or oral-facial-digital syndrome 1 (OFD1) results in the accumulation of signaling mediators normally targeted for pr...

Journal: :American journal of physiology. Renal physiology 2012
Amiya K Ghosh Toby Hurd Friedhelm Hildebrandt

Ciliopathies are a heterogeneous group of diseases that exhibit broad clinical phenotypes, including renal cysts, retinal degeneration, and cerebellar vermis aplasia. Nephronophthisis (NPHP) is a renal ciliopathy that causes chronic kidney disease and is characterized by kidney cysts at the cortico-medullary border. Among the 10 different disease-causing genes (NPHP1-NPHP10), mutations in NPHP3...

2016
Qing Zhang Yan Li Yuxia Zhang Vicente E. Torres Peter C. Harris Kun Ling Jinghua Hu

Primary cilia are sensory organelles indispensable for organogenesis and tissue pattern formation. Ciliopathy small GTPase ARLs are proposed as prominent ciliary switches, which when disrupted result in dysfunctional cilia, yet how ARLs are activated remain elusive. Here, we discover a novel small GTPase functional module, which contains ARL-3, ARL-13, and UNC-119, localizes near the poorly und...

Journal: :Developmental biology 2014
Xue Mei Trudi A Westfall Qihong Zhang Val C Sheffield Alexander G Bassuk Diane C Slusarski

Ciliopathies are genetic disorders that are caused by dysfunctional cilia and affect multiple organs. One type of ciliopathy, Bardet-Biedl syndrome, is a rare disorder characterized by obesity, retinitis pigmentosa, polydactyly, mental retardation and susceptibility to cardiovascular diseases. The Wnt/Planar cell polarity (PCP) has been associated with cilia function and ciliogenesis in directi...

Journal: :American journal of human genetics 2015
Eugenia Migliavacca Christelle Golzio Katrin Männik Ian Blumenthal Edwin C Oh Louise Harewood Jack A Kosmicki Maria Nicla Loviglio Giuliana Giannuzzi Loyse Hippolyte Anne M Maillard Ali Abdullah Alfaiz Mieke M van Haelst Joris Andrieux James F Gusella Mark J Daly Jacques S Beckmann Sébastien Jacquemont Michael E Talkowski Nicholas Katsanis Alexandre Reymond

The 16p11.2 600 kb copy-number variants (CNVs) are associated with mirror phenotypes on BMI, head circumference, and brain volume and represent frequent genetic lesions in autism spectrum disorders (ASDs) and schizophrenia. Here we interrogated the transcriptome of individuals carrying reciprocal 16p11.2 CNVs. Transcript perturbations correlated with clinical endophenotypes and were enriched fo...

Journal: :The Journal of clinical investigation 2015
Gisela G Slaats Joshua C Saldivar Julien Bacal Michelle K Zeman Andrew C Kile Ann Marie Hynes Shalabh Srivastava Jekaterina Nazmutdinova Krista den Ouden Miriam S Zagers Veronica Foletto Marianne C Verhaar Colin Miles John A Sayer Karlene A Cimprich Rachel H Giles

Juvenile ciliopathy syndromes that are associated with renal cysts and premature renal failure are commonly the result of mutations in the gene encoding centrosomal protein CEP290. In addition to centrosomes and the transition zone at the base of the primary cilium, CEP290 also localizes to the nucleus; however, the nuclear function of CEP290 is unknown. Here, we demonstrate that reduction of c...

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