نتایج جستجو برای: cd52

تعداد نتایج: 350  

Journal: :BioMed Research International 2022

Several factors have been reported to affect graft survival following kidney transplantation. CD52 molecules may increase T cell proliferation and activation, which contribute acute rejection survival. In the current study, we studied possible value of preoperative levels in predicting renal Ninety-six patients with end-stage disease who had transplantation were included study from our prospect...

Journal: :Blood 2011
Marco Montillo Alessandra Tedeschi Valeria Belsito Petrizzi Francesca Ricci Monica Crugnola Mauro Spriano Pierangelo Spedini Fiorella Ilariucci Lilj Uziel Immacolata Attolico Eleonora Vismara Angelo De Blasio Alfonso Zaccaria Enrica Morra

Although combination regimens have improved outcomes over monotherapy in chronic lymphocytic leukemia (CLL), patients eventually relapse. Combined fludarabine, cyclophosphamide, and monoclonal anti-CD52 antibody alemtuzumab (FCC) provided synergistic cytotoxicity with effective clearing of minimal residual disease. This phase 2 study determined FCC efficacy and safety in relapsed/refractory CD5...

2017
Paolo Gallo Diego Centonze Maria Giovanna Marrosu

Alemtuzumab (Lemtrada®) is a humanized anti-CD52 IgG1 monoclonal antibody that depletes CD52-expressing cells from the circulation. Robust clinical and radiologic data, derived from clinical trials and long-term observational studies, indicate that alemtuzumab induces a marked immunosuppression related to the depletion of circulating T and B lymphocytes. However, recent advances suggest that th...

2013
Cécile Schiffer Mannioui Laëtitia Lemaire Laurent Poirot Agnes Gouble Sylvain Arnould Roman Galetto Julianne Smith Andrew Scharenberg

Encouraging data have emerged from adoptive T-cell therapies in advanced forms of cancer. Anti-tumor immunity is found in tumor infiltrating lymphocytes as well as engineered T cells where exogenous expression of a chimeric antigen receptor (CAR) confers cancer recognition on the cells. Present adoptive immunotherapy methods are restricted to the use of autologous patient T-cells due to the lim...

Journal: :Blood 1994
H Nakakuma S Nagakura T Kawaguchi N Iwamoto M Hidaka K Horikawa T Kagimoto R Tsuruzaki K Takatsuki

Long-term clinical remission of more than 10 years is rarely seen in paroxysmal nocturnal hemoglobinuria (PNH). Affected blood cells in PNH lack glycosylphosphatidylinositol (GPI)-anchored membrane proteins such as decay-accelerating factor (DAF) and CD59. We performed a flow cytometric analysis of circulating blood cells obtained from two patients with PNH who had been in clinical remission fo...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2009
Srdan Verstovsek Ayalew Tefferi Hagop Kantarjian Taghi Manshouri Raja Luthra Animesh Pardanani Alfonso Quintás-Cardama Farhad Ravandi Pat Ault Carlos Bueso-Ramos Jorge E Cortes

PURPOSE Patients with hypereosinophilic syndrome (HES) or chronic eosinophilic leukemia (CEL) that are refractory to standard therapies are difficult to manage and have significantly shortened life expectancy. EXPERIMENTAL DESIGN CD52 is a surface glycoprotein highly expressed on eosinophils. We treated 11 patients with advanced HES/CEL with alemtuzumab, a humanized anti-CD52 monoclonal antib...

2016
Catharina C. Gross Diana Ahmetspahic Tobias Ruck Andreas Schulte-Mecklenbeck Kathrin Schwarte Silke Jörgens Stefanie Scheu Susanne Windhagen Bettina Graefe Nico Melzer Luisa Klotz Volker Arolt Heinz Wiendl Sven G. Meuth Judith Alferink

OBJECTIVE To characterize changes in myeloid and lymphoid innate immune cells in patients with relapsing-remitting multiple sclerosis (MS) during a 6-month follow-up after alemtuzumab treatment. METHODS Circulating innate immune cells including myeloid cells and innate lymphoid cells (ILCs) were analyzed before and 6 and 12 months after onset of alemtuzumab treatment. Furthermore, a potential...

Journal: :Blood 2010
Ronjon Chakraverty Guillermo Orti Michael Roughton Jun Shen Adele Fielding Panagiotis Kottaridis Donald Milligan Matthew Collin Charles Crawley Peter Johnson Andrew Clark Anne Parker Adrian Bloor Ruth Pettengell John Snowden Andrew Pettitt Richard Clark Geoff Hale Karl Peggs Kirsty Thomson Emma Morris Stephen Mackinnon

In vivo alemtuzumab reduces the risk of graft-versus-host disease (GVHD) and nonrelapse mortality after reduced intensity allogeneic transplantation. However, it also delays immune reconstitution, leading to frequent infections and potential loss of graft-versus-tumor responses. Here, we tested the feasibility of alemtuzumab dose deescalation in the context of fludarabine-melphalan conditioning...

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