نتایج جستجو برای: amegakaryocytic thrombocytopenia

تعداد نتایج: 24775  

Journal: :Blood 1986
T Nagasawa T Sakurai H Kashiwagi T Abe

We studied a patient with a rare complication of amegakaryocytic thrombocytopenia (AMT) associated with systemic lupus erythematosus (SLE). To investigate the underlying pathogenesis of AMT, the effects of peripheral blood T cells and serum on human megakaryocyte progenitor cells were studied using in vitro coculture techniques. Mononuclear bone marrow cells (2 X 10(5) from normal donors produc...

2018
Alessandro Pecci Iman Ragab Valeria Bozzi Daniela De Rocco Serena Barozzi Tania Giangregorio Heba Ali Federica Melazzini Mohamed Sallam Caterina Alfano Annalisa Pastore Carlo L Balduini Anna Savoia

Congenital amegakaryocytic thrombocytopenia (CAMT) is an inherited disorder characterized at birth by thrombocytopenia with reduced megakaryocytes, which evolves into generalized bone marrow aplasia during childhood. Although CAMT is genetically heterogeneous, mutations of MPL, the gene encoding for the receptor of thrombopoietin (THPO), are the only known disease-causing alterations. We identi...

2010
Fatma S. Al-Qahtani

Congenital amegakaryocytic thrombocytopenia (CAMT) is a rare inherited autosomal recessive disorder that presents with thrombocytopenia and absence of megakaryocytes. It presents with bleeding recognized on day 1 of life or at least within the first month. The cause for this disorder appears to be a mutation in the gene for the thrombopoeitin (TPO) receptor, c-Mpl, despite high levels of serum ...

Journal: :Blood 2001
M Ballmaier M Germeshausen H Schulze K Cherkaoui S Lang A Gaudig S Krukemeier M Eilers G Strauss K Welte

Congenital amegakaryocytic thrombocytopenia (CAMT) is a rare disease presenting with isolated thrombocytopenia in infancy and developing into a pancytopenia in later childhood. Thrombopoietin (TPO) is the main regulator of thrombocytopoiesis and has also been demonstrated to be an important factor in early hematopoiesis. We analyzed 9 patients with CAMT for defects in TPO production and reactiv...

2017
Christine Bellanné-Chantelot Matthieu Mosca Caroline Marty Rémi Favier William Vainchenker Isabelle Plo

The molecular basis of hereditary thrombocytosis is germline mutations affecting the thrombopoietin (TPO)/TPO receptor (MPL)/JAK2 signaling axis. Here, we report one family presenting two cases with a mild thrombocytosis. By sequencing JAK2 and MPL coding exons, we identified a germline MPL R102P heterozygous mutation in the proband and his daughter. Concomitantly, we detected high TPO levels i...

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