نتایج جستجو برای: als gene

تعداد نتایج: 1166211  

2016
Florie Borel Gwladys Gernoux Brynn Cardozo Jake P. Metterville Gabriela C. Toro Cabreja Lina Song Qin Su Guang Ping Gao Mai K. Elmallah Robert H. Brown Christian Mueller

Amyotrophic lateral sclerosis (ALS) is a fatal neurodegenerative disease; survival in ALS is typically 3-5 years. No treatment extends patient survival by more than three months. Approximately 20% of familial ALS and 1-3% of sporadic ALS patients carry a mutation in the gene encoding superoxide dismutase 1 (SOD1). In a transgenic ALS mouse model expressing the mutant SOD1(G93A) protein, silenci...

Amyotrophic lateral sclerosis (ALS) is a progressive neurodegenerative disease characterized by death of motor neurons leading to devastating muscle weakness and wasting and weight loss. It causes mixed picture of lower motor neuron (LMN) and upper motor neuron (UMN) dysfunction. The wide spectrums of atypical presentations can frequently lead to expensive work-up and undue delay in diagnosis o...

2013
Katsunobu Sugihara Hirofumi Maruyama Masaki Kamada Hiroyuki Morino Hideshi Kawakami

Mutations in the optineurin (OPTN) gene cause amyotrophic lateral sclerosis (ALS). We previously reported three types of OPTN mutation in Japanese ALS subjects. Here, to identify the OPTN mutations in individuals of different ethnicity, we screened 563 sporadic ALS (SALS) subjects and 124 familial ALS (FALS) subjects who were mainly Caucasian. We found a c.964T>C synonymous variation in exon 8....

2013
Yusuke Fujioka Shinsuke Ishigaki Akio Masuda Yohei Iguchi Tsuyoshi Udagawa Hirohisa Watanabe Masahisa Katsuno Kinji Ohno Gen Sobue

FUS is genetically and pathologically linked to amyotrophic lateral sclerosis (ALS) and frontotemporal lobar degeneration (FTLD). To clarify the RNA metabolism cascade regulated by FUS in ALS/FTLD, we compared the FUS-regulated transcriptome profiles in different lineages of primary cells from the central nervous system. The profiles of FUS-mediated gene expression and alternative splicing in m...

Journal: :The Journal of biological chemistry 2004
François Gros-Louis Roxanne Larivière Geneviève Gowing Sandra Laurent William Camu Jean-Pierre Bouchard Vincent Meininger Guy A Rouleau Jean-Pierre Julien

Peripherin is a neuronal intermediate filament associated with inclusion bodies in motor neurons of patients with amyotrophic lateral sclerosis (ALS). A possible peripherin involvement in ALS pathogenesis has been suggested based on studies with transgenic mouse overexpressors and with a toxic splicing variant of the mouse peripherin gene. However, the existence of peripherin gene mutations in ...

2016
David G. Brohawn Laura C. O’Brien James P. Bennett

ALS is a rapidly progressive, devastating neurodegenerative illness of adults that produces disabling weakness and spasticity arising from death of lower and upper motor neurons. No meaningful therapies exist to slow ALS progression, and molecular insights into pathogenesis and progression are sorely needed. In that context, we used high-depth, next generation RNA sequencing (RNAseq, Illumina) ...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2009
Francois Gros-Louis Peter M Andersen Nicolas Dupre Makoto Urushitani Patrick Dion Frederique Souchon Monique D'Amour William Camu Vincent Meininger Jean-Pierre Bouchard Guy A Rouleau Jean-Pierre Julien

Recently, chromogranins were reported to interact specifically with mutant forms of superoxide dismutase that are linked to amyotrophic lateral sclerosis (ALS). This interaction led us to analyze the frequencies of sequence variants of the CHGB gene in ALS patients and matched controls from three different countries. Of particular interest was the finding of the P413L CHGB variant present in 10...

Journal: :JAMA neurology 2013
Isabelle Le Ber Agnès Camuzat Rita Guerreiro Kawtar Bouya-Ahmed Jose Bras Gael Nicolas Audrey Gabelle Mira Didic Anne De Septenville Stéphanie Millecamps Timothée Lenglet Morwena Latouche Edor Kabashi Dominique Campion Didier Hannequin John Hardy Alexis Brice

IMPORTANCE Mutations in the SQSTM1 gene, coding for p62, are a cause of Paget disease of bone and amyotrophic lateral sclerosis (ALS). Recently, SQSTM1 mutations were confirmed in ALS, and mutations were also identified in 3 patients with frontotemporal dementia (FTD), suggesting a role for SQSTM1 in FTD. OBJECTIVE To evaluate the exact contribution of SQSTM1 to FTD and FTD with ALS (FTD-ALS)...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2013
Hemali P Phatnani Paolo Guarnieri Brad A Friedman Monica A Carrasco Michael Muratet Sean O'Keeffe Chiamaka Nwakeze Florencia Pauli-Behn Kimberly M Newberry Sarah K Meadows Juan Carlos Tapia Richard M Myers Tom Maniatis

ALS results from the selective and progressive degeneration of motor neurons. Although the underlying disease mechanisms remain unknown, glial cells have been implicated in ALS disease progression. Here, we examine the effects of glial cell/motor neuron interactions on gene expression using the hSOD1(G93A) (the G93A allele of the human superoxide dismutase gene) mouse model of ALS. We detect st...

2002
Barbara J. Mazur

Acetolactate synthase (ALS) catalyzes the first common step in the biosynthesis of isoleucine, leucine, and valine. The previous cloning of two tobacco (Nicotiana tabacum) ALS genes (SurA and SurB) has allowed transcript accumulation from these genes to be monitored. mRNA blot analysis of ALS transcripts showed a message size of 2.2 kb. Quantitation of the levels of ALS messages in tobacco orga...

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