نتایج جستجو برای: allergic airway inflammation

تعداد نتایج: 272736  

Journal: :Biochimica et Biophysica Acta (BBA) - General Subjects 2011

2017
Christopher Skappak Ramses Ilarraza Ying-Qi Wu Matthew G Drake Darryl J Adamko

Asthma exacerbation can be a life-threatening condition, and is most often triggered by common respiratory viruses. Poor asthma control and worsening of respiratory function is associated with increased airway inflammation, including eosinophilia. Prevention of asthma exacerbation relies on treatment with corticosteroids, which preferentially inhibit allergic inflammation like eosinophils. Huma...

2016
Elizabeth R. Davies Joanne F.C. Kelly Peter H. Howarth David I. Wilson Stephen T. Holgate Donna E. Davies Jeffrey A. Whitsett Hans Michael Haitchi

Asthma is a chronic inflammatory airways disease that usually begins in early life and involves gene-environment interactions. Although most asthma exhibits allergic inflammation, many allergic individuals do not have asthma. Here, we report how the asthma gene a disintegrin and metalloprotease 33 (ADAM33) acts as local tissue susceptibility gene that promotes allergic asthma. We show that enzy...

2011
Christian Taube Christine Tertilt Gabor Gyülveszi Nina Dehzad Katharina Kreymborg Kristin Schneeweiss Erich Michel Sebastian Reuter Jean-Christophe Renauld Danielle Arnold-Schild Hansjörg Schild Roland Buhl Burkhard Becher

Interleukin (IL)-22 is an effector cytokine, which acts primarily on epithelial cells in the skin, gut, liver and lung. Both pro- and anti-inflammatory properties have been reported for IL-22 depending on the tissue and disease model. In a murine model of allergic airway inflammation, we found that IL-22 is predominantly produced by innate lymphoid cells in the inflamed lungs, rather than TH ce...

Journal: :JCI insight 2018
Jan Beute Melanie Lukkes Ewout P Koekoek Hedwika Nastiti Keerthana Ganesh Marjolein Jw de Bruijn Steve Hockman Menno van Nimwegen Gert-Jan Braunstahl Louis Boon Bart N Lambrecht Vince C Manganiello Rudi W Hendriks Alex KleinJan

Phosphodiesterase 3 (PDE3) and PDE4 regulate levels of cyclic AMP, which are critical in various cell types involved in allergic airway inflammation. Although PDE4 inhibition attenuates allergic airway inflammation, reported side effects preclude its application as an antiasthma drug in humans. Case reports showed that enoximone, which is a smooth muscle relaxant that inhibits PDE3, is benefici...

2015
Jiaxing Xie Yin Xi Qingling Zhang Guoqin Chen Luo Wei Kefang Lai Nanshan Zhong

PURPOSE Extensive data support the influence of the upper airway on lower airway inflammation and pathophysiology in allergic disease. However, few studies have focused on allergic inflammation in the nose after an isolated lower airway allergen challenge, a situation that can exist clinically when human subjects breathe primarily through the mouth, as occurs when nasally congested. This study ...

Journal: :iranian journal of allergy, asthma and immunology 0
majid kianmeher neurogenic inflammation research centre, mashhad university of medical sciences, mashhad, iran and deptartment of physiology, school of medicine, mashhad university of medical sciences, mashhad, iran vahideh ghorani pharmaceutical research centre and department of physiology, school of medicine, mashhad university of medical sciences, mashhad, iran mohammad hosein boskabady neurogenic inflammation research centre, mashhad university of medical sciences, mashhad, iran and deptartment of physiology, school of medicine, mashhad university of medical sciences, mashhad, iran

asthma is a chronic inflammatory disease of the airway with extensive airway remodeling. the ethical issues associated with the studies in asthmatic patients, required development of animal model of asthma. animal models of asthma can provide valuable information on several features of asthma pathogenesis and treatment. although these models cannot carry out all clinical features, they are valu...

Journal: :Journal of immunology 2013
Nicolas J Dorsey Svetlana P Chapoval Elizabeth P Smith Jonathan Skupsky David W Scott Achsah D Keegan

STAT6 plays a central role in IL-4-mediated allergic responses. Several studies indicate that regulatory T cells (Tregs) can be modulated by IL-4 in vitro. We previously showed that STAT6(-/-) mice are highly resistant to allergic lung inflammation even when wild-type Th2 effectors were provided and that they have increased numbers of Tregs. However, the role of STAT6 in modulating Tregs in viv...

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