نتایج جستجو برای: type ii tgfβ receptor

تعداد نتایج: 2207007  

2003
A Wimmel B Wiedenmann S Rosewicz

Background and aim: The role of transforming growth factor β-1 (TGFβ-1) in neuroendocrine tumour biology is currently unknown. We therefore examined the expression and biological significance of TGFβ signalling components in neuroendocrine tumours (NETs) of the gastroenteropancreatic (GEP) tract. Methods: Expression of TGFβ-1 and its receptors, Smads and Smad regulated proteins, was examined in...

Journal: :The Journal of neuroscience : the official journal of the Society for Neuroscience 2014
Javier Palazuelos Michael Klingener Adan Aguirre

Research on myelination has focused on identifying molecules capable of inducing oligodendrocyte (OL) differentiation in an effort to develop strategies that promote functional myelin regeneration in demyelinating disorders. Here, we show that transforming growth factor β (TGFβ) signaling is crucial for allowing oligodendrocyte progenitor (OP) cell cycle withdrawal, and therefore, for oligodend...

2013
Xiangwei Xiao John Wiersch Yousef El-Gohary Ping Guo Krishna Prasadan Jose Paredes Carey Welsh Chiyo Shiota George K. Gittes

Protection and restoration of a functional β-cell mass are fundamental strategies for prevention and treatment of diabetes. Consequently, knowledge of signals that determine the functional β-cell mass is of immense clinical relevance. Transforming growth factor β (TGFβ) superfamily signaling pathways play a critical role in development and tissue specification. Nevertheless, the role of these p...

2017
Ryan J Gleason Mehul Vora Ying Li Nanci S Kane Kelvin Liao Richard W Padgett

Signal transduction of the conserved transforming growth factor-β (TGFβ) family signaling pathway functions through two distinct serine/threonine transmembrane receptors, the type I and type II receptors. Endocytosis orchestrates the assembly of signaling complexes by coordinating the entry of receptors with their downstream signaling mediators. Recently, we showed that the C. elegans type I bo...

Journal: :Human molecular genetics 2014
Junichi Iwata Akiko Suzuki Richard C Pelikan Thach-Vu Ho Pedro A Sanchez-Lara Yang Chai

Mutations in transforming growth factor beta (TGFβ) receptor type II (TGFBR2) cause Loeys-Dietz syndrome, characterized by craniofacial and cardiovascular abnormalities. Mice with a deletion of Tgfbr2 in cranial neural crest cells (Tgfbr2(fl/fl);Wnt1-Cre mice) develop cleft palate as the result of abnormal TGFβ signaling activation. However, little is known about metabolic processes downstream ...

Journal: :مجله غدد درون ریز و متابولیسم ایران 0
آزیتا پروانه تفرشی a. parvaneh tafreshi پژوهشگاه ملی مهندسی ژنتیک و زیست فناوری تهران، صندوق پستی 161-14965، دکتر آزیتا پروانه تفرشی راضیه جلال r jalal شمیلا درویش علی پور s darvishalipour حوری سپهری h sepehri خسرو عادلی k adeli

abstract introduction: there is limited knowledge available on the metabolism of glucose in the brain, an insulin insensitive organ. insulin receptors hybridize with insulin like growth factor receptor (igf-i) to transduce the signals in different areas of the brain. in this article we aimed at investigating whether the expression of igf-i receptor and igf-i binding proteins (igfbp1) is changed...

Journal: :Circulation research 2016
Aaron Kunamalla Jason Ng Vamsi Parini Shin Yoo Kate A McGee Todd T Tomson David Gordon Edward B Thorp Jon Lomasney Qiang Zhang Sanjiv Shah Suzanne Browne Bradley P Knight Rod Passman Jeffrey J Goldberger Gary Aistrup Rishi Arora

RATIONALE Fibrosis is an important structural contributor to formation of atrial fibrillation (AF) substrate in heart failure. Transforming growth factor-β (TGF-β) signaling is thought to be intricately involved in creation of atrial fibrosis. OBJECTIVE We hypothesized that gene-based expression of dominant-negative type II TGF-β receptor (TGF-β-RII-DN) in the posterior left atrium in a canin...

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