نتایج جستجو برای: tlr4 agonist

تعداد نتایج: 61583  

Journal: :Human molecular genetics 2015
Christian Giordano Kamalika Mojumdar Feng Liang Christian Lemaire Tong Li John Richardson Maziar Divangahi Salman Qureshi Basil J Petrof

Toll-like receptor 4 (TLR4) recognizes specific structural motifs associated with microbial pathogens and also responds to certain endogenous host molecules associated with tissue damage. In Duchenne muscular dystrophy (DMD), inflammation plays an important role in determining the ultimate fate of dystrophic muscle fibers. In this study, we used TLR4-deficient dystrophic mdx mice to assess the ...

Journal: :Colloids and Surfaces B: Biointerfaces 2014

Journal: :Journal of immunology 2003
Rudolf Haase Carsten J Kirschning Andreas Sing Percy Schröttner Koichi Fukase Shoichi Kusumoto Hermann Wagner Jürgen Heesemann Klaus Ruckdeschel

Conserved bacterial components potently activate host immune cells through transmembrane Toll-like receptors (TLRs), which trigger a protective immune response but also may signal apoptosis. In this study, we investigated the roles of TLR2 and TLR4 as inducers of apoptosis in Yersinia enterocolitica-infected macrophages. Yersiniae suppress activation of the antiapoptotic NF-kappaB signaling pat...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2012
Umeharu Ohto Koichi Fukase Kensuke Miyake Toshiyuki Shimizu

Lipopolysaccharide (LPS), also known as endotoxin, activates the innate immune response through toll-like receptor 4 (TLR4) and its coreceptor, MD-2. MD-2 has a unique hydrophobic cavity that directly binds to lipid A, the active center of LPS. Tetraacylated lipid IVa, a synthetic lipid A precursor, acts as a weak agonist to mouse TLR4/MD-2, but as an antagonist to human TLR4/MD-2. However, it ...

Journal: :Journal of immunology 2011
Vladimir Y Toshchakov Henryk Szmacinski Leah A Couture Joseph R Lakowicz Stefanie N Vogel

Agonist-induced dimerization of TLR4 Toll/IL-1R (TIR) domains initiates intracellular signaling. Therefore, identification of the TLR4-TIR dimerization interface is one key to the rational design of therapeutics that block TLR4 signaling. A library of cell-permeating decoy peptides, each of which represents a nonfragmented patch of the TLR4 TIR surface, was designed such that the peptides entir...

2012
Jinghua Sun Yanchun Ding

OBJECTIVE To investigate the expression of NOD2 in human VSMCs, its role in the production of inflammatory cytokines in VSMC and the possible interaction of NOD2-mediated signaling pathway with those mediated by TLR2 and TLR4. METHODS Human coronary artery smooth muscle cells were stimulated with NOD2 agonist MDP alone or in combination with either TLR2 agonist PAM3 or TLR4 agonist LPSs. The ...

Journal: :Infection and immunity 2005
Hiroyuki Tada Setsuya Aiba Ken-Ichiro Shibata Toshiaki Ohteki Haruhiko Takada

A synthetic Nod2 agonist, muramyldipeptide (MDP), and two Nod1 agonists, FK565 and FK156, mimic the bacterial peptidoglycan moiety and are powerful adjuvants that induce cell-mediated immunity, especially delayed-type hypersensitivity. In this study, we used human dendritic cell (DC) cultures to examine possible T helper type 1 (Th1) responses induced by MDP and FK565/156 in combination with va...

Journal: :Journal of immunology 2009
Mark H Wenink Kim C M Santegoets Mieke F Roelofs Richard Huijbens Hans J P M Koenen Ronald van Beek Irma Joosten Friederike Meyer-Wentrup Linda Mathsson Johan Ronnelid Gosse J Adema Ezio Bonvini Scott Koenig Wim B van den Berg Piet L C M van Riel Timothy R D J Radstake

Rheumatoid arthritis (RA) is a common autoimmune disease leading to profound disability and premature death. Although a role for FcgammaRs and TLRs is accepted, their precise involvement remains to be elucidated. FcgammaRIIb is an inhibitory FcR important in the maintenance of tolerance. We hypothesized that the inhibitory FcgammaRIIb inhibits TLR responses on monocyte-derived dendritic cells (...

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