نتایج جستجو برای: tay sachs disease

تعداد نتایج: 1492516  

Journal: :Human molecular genetics 2017
Alexander W M Hooper Javier F Alamilla Rosemarie E Venier Deda C Gillespie Suleiman A Igdoura

GM2 gangliosidoses are a group of lysosomal storage disorders which include Sandhoff disease and Tay-Sachs disease. Dysregulation of glutamate receptors has been recently postulated in the pathology of Sandhoff disease. Glutamate receptor association with neuronal pentraxins 1 and 2, and the neuronal pentraxin receptor facilitates receptor potentiation and synaptic shaping. In this study, we ha...

2010
Volkan Seyrantepe Pablo Lema Aurore Caqueret Larbi Dridi Samar Bel Hadj Stephane Carpentier Francine Boucher Thierry Levade Lionel Carmant Roy A. Gravel Edith Hamel Pascal Vachon Graziella Di Cristo Jacques L. Michaud Carlos R. Morales Alexey V. Pshezhetsky

Tay-Sachs disease is a severe lysosomal disorder caused by mutations in the HexA gene coding for the α-subunit of lysosomal β-hexosaminidase A, which converts G(M2) to G(M3) ganglioside. Hexa(-/-) mice, depleted of β-hexosaminidase A, remain asymptomatic to 1 year of age, because they catabolise G(M2) ganglioside via a lysosomal sialidase into glycolipid G(A2), which is further processed by β-h...

Journal: :The Application of Clinical Genetics 2015

Journal: :Biochemistry and Molecular Biology Education 2002

Journal: :Obstetrics and gynecology 2009

Certain autosomal recessive disease conditions are more prevalent in individuals of Eastern European Jewish (Ashkenazi) descent. Previously, the American College of Obstetricians and Gynecologists recommended that individuals of Eastern European Jewish ancestry be offered carrier screening for Tay-Sachs disease, Canavan disease, and cystic fibrosis as part of routine obstetric care. Based on th...

Journal: :Frontiers in Physiology 2018

2012
Gary Mockli

In 1887, Bernard Sachs reported on the clinical features diagnostic signs and symptoms and pathology of a patient with a condition that would someday bear his name(l). It was several decades later before it was discovered that these patients suffered from a recessive genetic trait resulting in the accumulation of a specific ganglioside, GM2, due to the deficiency of the lysosomal enzyme hexosam...

Behnam Kamalidehghan, Massoud Houshmand, Nasim Eskandari, Omid Aryani, Shadab Salehpour, Solmaz Jamali, Talieh Zaman,

Background: Tay-Sachs disease (TSD), or GM2 gangliosidosis, is a lethal autosomal recessive neurodegenerative disorder, which is caused by a deficiency of beta-hexosaminidase A (HEXA), resulting in lysosomal accumulation of GM2 ganglioside. The aim of this study was to identify the TSD-causing mutations in‌ an Iranian population. Methods: In this study, we examined 31 patients for TSD-causing m...

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