نتایج جستجو برای: sunitinib

تعداد نتایج: 3081  

2016
Yongqian Gao Jinyi Li Na Qiao Qingsong Meng Ming Zhang Xin Wang Jianghua Jia Shuwen Yang Changbao Qu Wei Li Dongbin Wang

PURPOSE To evaluate the mechanisms underlying sunitinib resistance in RCC and to identify targets that may be used to overcome this resistance. RESULTS Reanalysis of transcriptome microarray datasets (GSE64052 and GSE76068) showed that adrenomedullin expression was increased in sunitinib-resistant tumors. And adrenomedullin expression was increased in sunitinib-resistant tumor xenografts, acc...

2014
Ibrahim Yildiz Fatma Sen Leyla Kilic Rumeysa Ciftci Mert Basaran

Sunitinib is a multiple tyrosine kinase receptor inhibitor that is approved for the treatment of metastatic renal cell carcinoma (RCC). However, neither an appropriate dose nor dosing schedule of sunitinib has yet been established for patients with metastatic RCC who are on hemodialysis. Here, we report on two hemodialysis patients who received sunitinib to treat metastatic RCC. Sunitinib was p...

Journal: :Cancer research 2010
Dan Huang Yan Ding Yan Li Wang-Mei Luo Zhong-Fa Zhang John Snider Kristin Vandenbeldt Chao-Nan Qian Bin Tean Teh

Sunitinib is a broad-spectrum small-molecule inhibitor of receptor tyrosine kinases (RTK) that serves as the present standard of care for first-line therapy of advanced clear cell renal cell carcinoma (ccRCC). A full understanding of the targets and mechanism of action of sunitinib in ccRCC treatment remains incomplete. In this study, we evaluated several tumor cell and endothelial targets of s...

Journal: :Cancer research 2009
Hong Xin Chunyan Zhang Andreas Herrmann Yan Du Robert Figlin Hua Yu

The novel multitargeted tyrosine kinase inhibitor sunitinib is used as an antiangiogenic agent for the treatment of several types of cancer, including metastatic renal cell carcinoma (RCC). Sunitinib was shown to positively change the immunosuppressive phenotype in RCC patients. To improve its antitumor efficacy, and offer strategies for its combination with other approaches, it is critical to ...

2015
P Nowak-Sliwinska A Weiss J R van Beijnum T J Wong W W Kilarski G Szewczyk H M W Verheul T Sarna H van den Bergh A W Griffioen

The angiogenesis inhibitor sunitinib is a tyrosine kinase inhibitor that acts mainly on the VEGF and PDGF pathways. We have previously shown that sunitinib is sequestered in the lysosomes of exposed tumor and endothelial cells. This phenomenon is part of the drug-induced resistance observed in the clinic. Here, we demonstrate that when exposed to light, sequestered sunitinib causes immediate de...

2017
Tingyu Wen Hai Xiao Chao Luo Li Huang Meimei Xiong

The most efficient sequence of targeted agents for metastatic renal cell carcinoma patients has yet to be identified. Whether the sequence of sorafenib and sunitinib really matters is controversial and not answered clearly until now. This meta-analysis aims to estimate the efficacy of receptor tyrosine kinase inhibitors sorafenib-sunitinib and sunitinib-sorafenib for metastatic renal cell carci...

Journal: :The Journal of clinical investigation 2017
Mohamed Elgendy Amal Kamal Abdel-Aziz Salvatore Lorenzo Renne Viviana Bornaghi Giuseppe Procopio Maurizio Colecchia Ravindran Kanesvaran Chee Keong Toh Daniela Bossi Isabella Pallavicini Jose Luis Perez-Gracia Maria Dolores Lozano Valeria Giandomenico Ciro Mercurio Luisa Lanfrancone Nicola Fazio Franco Nole Bin Tean Teh Giuseppe Renne Saverio Minucci

Most patients who initially respond to treatment with the multi-tyrosine kinase inhibitor sunitinib eventually relapse. Therefore, developing a deeper understanding of the contribution of sunitinib's numerous targets to the clinical response or to resistance is crucial. Here, we have shown that cancer cells respond to clinically relevant doses of sunitinib by enhancing the stability of the anti...

2017
Le Qu Wen-Quan Zhou Zheng-Yu Zhang

© Translational Andrology and Urology. All rights reserved. Transl Androl Urol, 2017;6(S3) tau.amegroups.com sunitinib resistance, but the picture remains unclear. On the other hand, few prognostic factors have been validated as predictive biomarkers of sunitinib response. Thus, it is urgent to elucidate the underlying mechanisms of sunitinib resistance and discover reliable biomarkers that can...

Journal: :Molecular cancer therapeutics 2012
Peter B Makhov Konstantin Golovine Alexander Kutikov Ervin Teper Daniel J Canter Jay Simhan Robert G Uzzo Vladimir M Kolenko

Tyrosine kinase inhibitors exhibit impressive activity against advanced renal cell carcinoma. However, recent clinical studies have shown an equivocal response to sunitinib in patients with castration-resistant prostate cancer. The tumor suppressor PTEN acts as a gatekeeper of the phosphoinositide 3-kinase (PI3K)/Akt/mTOR cell-survival pathway. Our experiments showed that PTEN expression invers...

Journal: :Sarcoma 2009
Changhoon Yoo Jeong-Eun Kim Shin-Kyo Yoon Song Cheol Kim Jin-Hee Ahn Tae Won Kim Cheolwon Suh Jae-Lyun Lee

A 52 year-old woman presented with an incidentally detected retroperitoneal angiosarcoma and multiple hepatic metastases. After chemotherapy with weekly paclitaxel and doxorubicin, angiosarcoma had progressed rapidly. Because few chemotherapeutic options were available for her, sunitinib (37.5 mg/day, daily) as a salvage regimen was administered. Although sunitinib was interrupted after two wee...

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