نتایج جستجو برای: scid

تعداد نتایج: 7562  

2017
Erez Rechavi Atar Lev Talia Saraf-Levy Amos Etzioni Shlomo Almashanu Raz Somech

Newborn screening (NBS) programs for severe combined immunodeficiency (SCID), the most severe type of primary immunodeficiency, are being implemented in more and more countries with every passing year. Since October 2015, SCID screening via T cell receptor excision circle (TREC) quantification in dried blood spots (DBS) has been part of the Israeli NBS program. As an NBS program in its infancy,...

Journal: :Haematologica 2006
Mirjam van der Burg Corry M R Weemaes Frank Preijers Paul Brons Barbara H Barendregt Maarten J D van Tol Peter Hoogerbrugge Jacques J M van Dongen

Severe combined immunodeficiencies (SCID) are commonly fatal early in life. Adequate diagnosis and rapid institution of treatment, such as allogeneic stem cell transplantation (SCT), is essential. Several studies demonstrated that reconstitution of B-cell function after SCT is better in B-positive SCID than in B-negative SCID. We demonstrate that B-cell reconstitution in a B-negative SCID patie...

2014
Amel Hassan Pamela Lee Paraskevi Maggina Jin Hua Xu Diana Moreira Mary Slatter Zohreh Nademi Austen Worth Stuart Adams Alison Jones Catherine Cale Zoe Allwood Kanchan Rao Robert Chiesa Persis Amrolia Hubert Gaspar E. Graham Davies Paul Veys Andrew Gennery Waseem Qasim

BACKGROUND Severe combined immunodeficiency (SCID) can be cured by using allogeneic hematopoietic stem cell transplantation, and the absence of host immunity often obviates the need for preconditioning. Depending on the underlying genetic defect and when blocks in differentiation occur during lymphocyte ontogeny, infants with SCID have absent or greatly reduced numbers of functional T cells. Na...

Journal: :The Journal of Experimental Medicine 1991
G H Sunshine B L Jimmo C Ianelli L Jarvis

We have examined the requirements for activating unprimed T cells in vivo by transferring T cells into scid mice, which lack mature B and T cells. Purified adult thymocytes and a protein antigen, keyhole limpet hemocyanin (KLH), were injected into scid mice. scid mice injected with T cells and KLH developed cellular lymph nodes containing CD4+ and CD8+ T cells. Cells recovered from the lymph no...

Journal: :The Journal of Experimental Medicine 1987
W J Murphy V Kumar M Bennett

C.B-17 scid (H-2d) mice are homozygous for the gene that causes severe combined immune deficiency (SCID). These mice have no T or B cell function, yet display normal natural killer (NK) activity. Irradiated SCID mice were challenged with marrow grafts to determine if antibodies are necessary for marrow allograft rejection. SCID mice rejected H-2/Hh-1 allogeneic marrow grafts. Moreover, this rej...

Journal: :Infection and immunity 1998
S C Cartner J R Lindsey J Gibbs-Erwin G H Cassell J W Simecka

Current evidence suggests that host defense in respiratory mycoplasmosis is dependent on both innate and humoral immunity. To further delineate the roles of innate and adaptive immunity in antimycoplasmal defenses, we intranasally infected C3H/HeSnJ-scid/scid (C3H-SCID), C3H/HeSnJ (C3H), C57BL/6J-scid/scid (C57-SCID), and C57BL/6N (C57BL) mice with Mycoplasma pulmonis and at 14 and 21 days post...

Journal: :Journal of immunology 1998
B M Frey S Rafii M Teterson D Eaton R G Crystal M A Moore

Thrombopoietin (TPO) cDNA can be effectively delivered in vivo by adenovectors. Immune normal mice (BALB/c) and syngeneic mice with variable degrees of immune dysfunction nu, SCID, and NOD-SCID) were treated with an adenovirus vector expressing the human TPO cDNA (AdTPO). Platelet peaks were significantly higher in SCID and NOD-SCID mice compared with BALB/c and nu mice. Human plasma TPO concen...

2002
Mamoru Ito Hidefumi Hiramatsu Kimio Kobayashi Kazutomo Suzue Mariko Kawahata Kyoji Hioki Yoshito Ueyama Yoshio Koyanagi Kazuo Sugamura Kohichiro Tsuji Toshio Heike

c null mice and NOD/Shi-scid mice. When human CD34 cells from umbilical cord blood were transplanted into this strain, the engraftment rate in the peripheral circulation, spleen, and bone marrow were significantly higher than that in NOD/Shiscid mice treated with anti-asialo GM1 antibody or in the 2-microglobulin– deficient NOD/LtSz-scid (NOD/SCID/ 2mnull) mice, which were as completely defecti...

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