نتایج جستجو برای: rho

تعداد نتایج: 18986  

Journal: :Physical review 2021

Recently, the BESIII Collaboration reported a resonance $Y(2040)$ with $M=2034\pm13\pm9$ MeV and $\Gamma=234\pm30\pm25$ in process of $e^+e^-\rightarrow\omega\pi^0$. In addition, new measurements much higher precision for $\rho(1900)$ $\rho(2150)$ states are obtained by BABAR Collaborations. this work, we perform systematic study on mass spectrum excited $\rho$ resonances using modified Godfrey...

Journal: :American journal of physiology. Gastrointestinal and liver physiology 2003
Hitoshi Ikeda Kayo Nagashima Mikio Yanase Tomoaki Tomiya Masahiro Arai Yukiko Inoue Kazuaki Tejima Takako Nishikawa Masao Omata Satoshi Kimura Kenji Fujiwara

Hepatic stellate cells (HSCs) play a central role in the development of hepatic fibrosis. Recent evidence has revealed that HSCs also play a role in its resolution, where HSC apoptosis was determined. Moreover, induction of HSC apoptosis caused a reduction of experimental hepatic fibrosis in rats. Thus knowing the mechanism of HSC apoptosis might be important to clarify the pathophysiology and ...

Journal: :Infection and immunity 2002
Rebecca Krall Jianjun Sun Kristin J Pederson Joseph T Barbieri

ExoS is a bifunctional type III cytotoxin secreted by Pseudomonas aeruginosa, which comprises a C-terminal ADP ribosyltransferase domain and an N-terminal Rho GTPase-activating protein (GAP) domain. In vitro, ExoS is a Rho GAP for Rho, Rac, and Cdc42; however, the in vivo modulation of Rho GTPases has not been addressed. Using a transient transfection system and delivery by P. aeruginosa, inter...

Journal: :International Journal of Pure and Apllied Mathematics 2016

Journal: :The Journal of biological chemistry 1997
K Takahashi T Sasaki A Mammoto K Takaishi T Kameyama S Tsukita Y Takai

The Rho GDP dissociation inhibitor (GDI) forms a complex with the GDP-bound form of the Rho family small G proteins and inhibits their activation. The GDP-bound form complexed with Rho GDI is not activated by the GDP/GTP exchange factor for the Rho family members, suggesting the presence of another factor necessary for this activation. We have reported that the Rho subfamily members regulate th...

2005
Russell J. Ferland Xiaoyu Li Janet E. Buhlmann Xia Bu Christopher A. Walsh Bing Lim

Rho-GDIs are a family of Rho GDP-dissociation inhibitors that are critical in modulating the activity of the small GTPases, Cdc42 and RhoA. Two Rho-GDI isoforms are expressed in the brain, Rho-GDIg and Rho-GDIa. Here, we describe the expression of both of these isoforms in the developing and mature brain. The mRNA expression patterns of Rho-GDIg and Rho-GDIa were almost identical in the brain w...

Journal: :Simon Stevin 2021

Let $D$ be the open disk $|x|< R$ of a complete ultrametric algebraically closed field $\mathbb K$. We define growth order $\rho(f)$, type $\sigma(f)$ cotype $\psi(f)$ and another expression $\theta(f)$ an analytic function in we show relations between number zeros $f$ ``maximum modulus'' involving $\rho,\sigma,\psi$. Then $\rho(f)$ lies $[\theta(f)-1, \theta(f)]$. Moreover, if $0< \rho(f)< +\i...

Journal: :Physical review letters 2007
R Kawai J M R Parrondo C Van den Broeck

We show, through a refinement of the work theorem, that the average dissipation, upon perturbing a Hamiltonian system arbitrarily far out of equilibrium in a transition between two canonical equilibrium states, is exactly given by =W-DeltaF=kTD(rho||rho[over ])=kT, where rho and rho[over ] are the phase-space density of the system measured at the same intermediate ...

2014
Sérgio de Matos Simões Avantika Mainieri Jennifer A. Zallen

Actomyosin contraction generates mechanical forces that influence cell and tissue structure. During convergent extension in Drosophila melanogaster, the spatially regulated activity of the myosin activator Rho-kinase promotes actomyosin contraction at specific planar cell boundaries to produce polarized cell rearrangement. The mechanisms that direct localized Rho-kinase activity are not well un...

Background: Growth retardation is a long-term complication in pediatric transfusion-dependent thalassemias (TDTs), presented as short-stature and upper body segment shortening. The cause of this condition was chronic hypoxia, iron overload, endocrinopathy, inadequate transfusion, and iron chelation. We analyze the relationship between ferritin level and growth status of pediatric TDTs. Methods...

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