نتایج جستجو برای: rare mutations

تعداد نتایج: 402573  

Journal: :hepatitis monthly 0
hassan dastsooz department of medical genetics, shiraz university of medical sciences, shiraz, ir iran; department of molecular medicine, shiraz university of medical sciences, shiraz, ir iran mohammad hadi imanieh shiraz transplant research center, gastroenterohepatology research center, namazi teaching hospital, shiraz university of medical sciences, shiraz, ir iran seyed mohsen dehghani shiraz transplant research center, gastroenterohepatology research center, namazi teaching hospital, shiraz university of medical sciences, shiraz, ir iran mahmood haghighat shiraz transplant research center, gastroenterohepatology research center, namazi teaching hospital, shiraz university of medical sciences, shiraz, ir iran maryam moini department of internal medicine, gastroenterology and hepatology research center, shiraz university of medical sciences, shiraz, ir iran majid fardaei department of medical genetics, shiraz university of medical sciences, shiraz, ir iran; department of molecular medicine, shiraz university of medical sciences, shiraz, ir iran; stem cell and transgenic technology research center, shiraz university of medical sciences, shiraz, ir iran; department of medical genetics, shiraz university of medical sciences, 7134853185, shiraz, ir iran. tel: +98-7112349610, fax: +98-7112349610

results using these two sets, we identified h1069q mutation in four patients, c.2335t > g mutation in three, c.3061-1g > a splice site mutation in five, c.3305t > c mutation in one, and c.3809a > g mutation in two patients. conclusions the multiplex arms assay used in this study can be an efficient, reliable, and cost effective method as a primary screen for patients with wilson disease. patien...

Congenital recessive myotonia is a rare genetic disorder caused by mutations in CLCN1, which codes for the main skeletal muscle chloride channel ClC-1. More than 120 mutations have been found in this gene. The main feature of this disorder is muscle membrane hyperexcitability. Here, we report a 59-year male patient suffering from congenital myotonia. He had transient generalized myotonia, which...

A Ghasemi, A Ghotaslou, B Chahardouli, F Nadali, S Abbasian, S Rostami,

Background Myeloproliferative disorders are a group of diseases characterized by increased proliferation of myeloid lineage. In addition to JAK2V617F mutation, several mutations in the c-MPL gene were described in patients with Philadelphia-negative chronic myeloproliferative disorders that could be important in the pathogenesis of diseases. The aim of present study was to investigate the fre...

Backgrounds: Hypohidrotic ectodermal dysplasia (HED) is a rare genetic disorder, distinguished by hypotrichosis, hypohidrosis, and hypodontia. HDE can be inherited in X-linked recessive manner as a result of mutations in the ectodysplasin A (EDA) gene as well as autosomal dominant and autosomal recessive manners both of them caused by mutations in EDA receptor (EDAR) and EDAR-associated death d...

Journal: :Journal of Thoracic Oncology 2015

پایان نامه :وزارت علوم، تحقیقات و فناوری - دانشگاه شهید بهشتی - دانشکده علوم 1372

a simple, sensitive and selective method for solvent extraction and spectrophotemetric determination of lanthanum (ii) europium (iii), and cerium (iii) is described , the rare earth metals are extracted into chloroform solution of n-phenylbenzohydroxamic acid (pbha) at ph 9-10 various parameters are studied to optimize the extraction conditions. the molar absorptivity is found to increase from ...

تقوی, سیداسداله, رستگار لاری, قاسم, رسول زادگان, مریم, علا, فریدون, کاظمی, احمد,

    Background & Aim: Bernard-Soulier syndrome (B.S.S) is a rare hereditary bleeding disorder due to molecular defects of platelet GPIb–IX–V. The GPIb-IX-V complex is composed of four chains of GPIbα, GPIbβ, GPIX and GPV.  The largest chain of this complex is GPIbα and is responsible for binding to ligand and most of identified mutations belong to this glycoprotein.  The aim of  this  study was...

Journal: :Diagnostic Molecular Pathology 2010

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