نتایج جستجو برای: procainamide

تعداد نتایج: 706  

2012
Soraya Shahrzad Samira Taban Amir Aslani Zahra Emkanjoo

Objectives-The Brugada syndrome is a heterogeneous genetic disease that predisposes one to lifethreatening ventricular tachyarrhythmia and sudden cardiac death (SCD). In this study, we sought to compare the efficacy of intravenous Procainamide versus oral Flecainide to unmask the typical electrocardiographic changes of this syndrome. Methods-From October 2001 to December 2010, we evaluated pati...

Journal: :The American journal of cardiology 2007
George E Kochiadakis Nikos E Igoumenidis Michail E Hamilos Maria E Marketou Gregory I Chlouverakis Panos E Vardas

The appropriate treatment for the restoration of sinus rhythm in patients with atrial fibrillation (AF) of recent onset is still the subject of controversy. In this prospective, randomized, single-blind, placebo-controlled clinical study, we investigated the effectiveness and safety of procainamide, propafenone, and amiodarone, administered intravenously, for the conversion of recent-onset AF. ...

Journal: :Circulation 1987
K M Wu B F Hoffman

We studied the effects of procainamide and N-acetylprocainamide (NAPA) in a conscious dog preparation of atrial flutter resulting from circus movement around the tricuspid orifice. We also recorded transmembrane potentials of atrial tissues from the circus path in vitro. In 12 instrumented dogs, average flutter cycle length was 157 msec, the duration of the excitable gap was 73 msec, and conduc...

Journal: :Circulation research 1989
T Nakajima Y Kurachi H Ito R Takikawa T Sugimoto

Effects of quinidine, disopyramide, and procainamide on the acetylcholine (ACh)-induced K+ channel current were examined in single atrial cells, using the tight-seal, whole-cell clamp technique. The pipette solution contained guanosine-5'-triphosphate (GTP) or guanosine-5'-O-(3-thiotriphosphate) (GTP-gamma S, a nonhydrolysable GTP analogue). In GTP-loaded cells, not only ACh but also adenosine ...

Journal: :British heart journal 1970
P Szekely N A Wynne D T Pearson G A Batson D A Sideris

A review is given of 457 episodes of atrial fibrillation that occurred in 318 patients and were treated by DC shock. Antidysrhythmic drugs, such as quinidine, procainamide, and propranolol, given singly or in combination, were used concomitantly in 389 instances, and DC shock alone was given in 68 instances. The combined effects of quinidine and DC shock, and of procainamide and DC shock were s...

Journal: :Chest 1993
R W Peters H J Kim G A Buser M R Gold

We describe a patient with ischemically mediated sustained monomorphic ventricular tachycardia occurring at rest and resistant to treatment with intravenously administered procainamide. Percutaneous transluminal coronary angioplasty was initially successful, but rest angina and ventricular tachycardia, resistant to procainamide therapy, recurred 2 weeks later and responded to aggressive anti-is...

Journal: :Annals of the rheumatic diseases 1989
R A Asherson J Zulman G R Hughes

Procainamide is the commonest cause of a drug induced lupus syndrome. Long term administration of this compound may induce a variety of immunological abnormalities, including antinuclear antibodies. Uncommonly, 'lupus anticoagulants' have been demonstrated in the absence of other evidence of drug induced lupus. Details of a 67 year old man who developed not only drug induced lupus but also anti...

2005
FRANCIS E. MARCHLINSKI

Induction of rapid ventricular tachycardia or fibrillation during therapy with amiodarone is associated with an increased risk of sudden death. To determine whether the addition of a type IA antiarrhythmic agent to therapy would improve outcome, 37 patients in whom ventricular tachyarrhythmia of a cycle length less than 350 msec was induced after 14 + 2 days of amiodarone were randomly assigned...

Journal: :Circulation 1976
R Levites J I Haft J Calderon Venkatachalapathy

In 14 mongrel dogs, refractory periods were determined in nonischemic and acutely ischemic zones of myocardium during control conditions, 15 minutes after coronary ligation, and 10 and 20 minutes after a procainamide infusion. Following coronary ligation, refractory periods in the nonischemic area remained unchanged (100.8% of control) while in the ischemia area they decreased to 88.6% of contr...

Journal: :British Journal of Clinical Pharmacology 1977

نمودار تعداد نتایج جستجو در هر سال

با کلیک روی نمودار نتایج را به سال انتشار فیلتر کنید