نتایج جستجو برای: prasugrel

تعداد نتایج: 1097  

2014
Matthew W. Sherwood Stephen D. Wiviott S. Andrew Peng Matthew T. Roe James DeLemos Eric D. Peterson Tracy Y. Wang

BACKGROUND P2Y12 antagonist therapy improves outcomes in acute myocardial infarction (MI) patients. Novel agents in this class are now available in the US. We studied the introduction of prasugrel into contemporary MI practice to understand the appropriateness of its use and assess for changes in antiplatelet management practices. METHODS AND RESULTS Using ACTION Registry-GWTG (Get-with-the-G...

Journal: :Revista espanola de cardiologia 2011
Carlos Felipe Barrera-Ramírez Luis Ramón Pineda-Pompa Carlos E Guzmán-Rodríguez

We read with interest the editorial by Drs. Freedman and Iafrati recently published in Revista Española de Cardiologı́a. The editorial mentions that prasugrel is an inhibitor of the P2Y12 receptor, that it is not metabolized in the liver, and that it does not appear to be affected by the variability of isozyme P450. We believe that it might be a good idea here to indicate the pharmacokinetic cha...

Journal: :The Journal of pharmacology and experimental therapeutics 2013
Elisabetta Liverani Mario C Rico Analia E Garcia Laurie E Kilpatrick Satya P Kunapuli

Clopidogrel and prasugrel belong to a thienopyridine class of oral antiplatelet drugs that, after having been metabolized in the liver, can inhibit platelet function by irreversibly antagonizing the P2Y(12) receptor. Furthermore, thienopyridines influence numerous inflammatory conditions, but their effects on neutrophils have not been evaluated, despite the important role of these cells in infl...

2018
Matt S. Anderson Teddy Kosoglou Paul Statkevich Jing Li Jennifer Rotonda Alan G. Meehan David L. Cutler

Vorapaxar is a first-in-class antagonist of the protease-activated receptor-1, the primary thrombin receptor on human platelets, which mediates the downstream effects of thrombin in hemostasis and thrombosis. Prasugrel is a platelet inhibitor that acts as a P2Y12 receptor antagonist through an active metabolite, R-138727. This study investigated the interaction of these 2 platelet antagonists w...

Journal: :Circulation 2008
Stephen D Wiviott Eugene Braunwald Dominick J Angiolillo Simha Meisel Anthony J Dalby Freek W A Verheugt Shaun G Goodman Ramon Corbalan Drew A Purdy Sabina A Murphy Carolyn H McCabe Elliott M Antman

BACKGROUND Patients with diabetes mellitus (DM) are at high risk for recurrent cardiovascular events after acute coronary syndromes, in part because of increased platelet reactivity. The Trial to Assess Improvement in Therapeutic Outcomes by Optimizing Platelet Inhibition With Prasugrel-Thrombolysis in Myocardial Infarction 38 (TRITON-TIMI 38) showed an overall reduction in ischemic events with...

Journal: :Archives of cardiovascular diseases 2016
Raoul Bacquelin Emmanuel Oger Emmanuelle Filippi Jean-Philippe Hacot Vincent Auffret Marielle Le Guellec Isabelle Coudert Philippe Castellant Benoît Moquet Philippe Druelles Antoine Rialan Gilles Rouault Bertrand Boulanger Josiane Treuil Guillaume Leurent Marc Bedossa Dominique Boulmier Bertrand Avez Martine Gilard Hervé Le Breton

BACKGROUND Antiplatelet therapies, including prasugrel, are a cornerstone in the treatment of ST-segment elevation myocardial infarction (STEMI), but are associated with a bleeding risk. This risk has been evaluated in randomized trials, but few data on real-world patients are available. AIM To evaluate prasugrel safety in real-world patients with STEMI. METHODS Consecutive patients with ST...

2013
Dimitrios Alexopoulos Ioanna Xanthopoulou Eleni Mavronasiou Katerina Stavrou Argyro Siapika Evropi Tsoni Periklis Davlouros

OBJECTIVE It has been postulated that prasugrel might be the preferred treatment option in diabetes mellitus (DM) patients with acute coronary syndrome (ACS) undergoing percutaneous coronary intervention (PCI). We aimed to compare the pharmacodynamic action of ticagrelor versus prasugrel. RESEARCH DESIGN AND METHODS In a prospective, single-center, single-blind, crossover study, 30 consecutiv...

Journal: :Circulation journal : official journal of the Japanese Circulation Society 2015
Young Seok Lee Cai De Jin Moo Hyun Kim Long Zhe Guo Young-Rak Cho Kyungil Park Jong Sung Park Tae-Ho Park Young Dae Kim

BACKGROUND There is insufficient data on the efficacy of prasugrel and ticagrelor in Korean patients with ST-segment elevation myocardial infarction (STEMI). METHODS AND RESULTS I n the current double-blind, prospective pilot study, 39 patients with STEMI undergoing primary percutaneous coronary intervention were randomized to receive prasugrel 60 mg loading dose (LD) followed by 10 mg daily ...

Journal: :Drug metabolism and disposition: the biological fate of chemicals 2016
Takaya Kurokawa Tatsuki Fukami Tomohiro Yoshida Miki Nakajima

Prasugrel, a thienopyridine anti-platelet agent, is pharmacologically activated by hydrolysis and hydroxylation. It is efficiently hydrolyzed in the intestine after oral administration, and the enzyme responsible for the hydrolysis in humans was demonstrated to be carboxylesterase (CES)2. Prasugrel hydrolase activity is detected in dog intestines, where CES enzymes are absent; therefore, this p...

Journal: :Haematologica 2015
Constance C F M J Baaten Leo F Veenstra Rick Wetzels Johanna P van Geffen Frauke Swieringa Susanne M de Witt Yvonne M C Henskens Harry Crijns Sven Nylander J J J van Giezen Johan W M Heemskerk Paola E J van der Meijden

In patients with acute coronary syndrome, dual antiplatelet therapy with aspirin and a P2Y12 inhibitor like prasugrel is prescribed for one year. Here, we investigated how the hemostatic function of platelets recovers after discontinuation of prasugrel treatment. Therefore, 16 patients who suffered from ST-elevation myocardial infarction were investigated. Patients were treated with aspirin (10...

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