نتایج جستجو برای: npm1 mutation

تعداد نتایج: 292091  

Journal: :Archives of medical science : AMS 2016
Dorota Koczkodaj Szymon Zmorzyński Małgorzata Michalak-Wojnowska Ewa Wąsik-Szczepanek Agata A Filip

INTRODUCTION Acute myeloid leukemia (AML) is a genetically heterogeneous disease at both the cytogenetic and molecular levels. In AML cells many chromosomal aberrations are observed, some of them being characteristic of a particular subtype of patients, and others being less significant. Besides chromosomal abnormalities, the leukemic cells can have a variety of mutations involving individual g...

2014
Min Wang Na He Tian Tian Lu Liu Shuang Yu Daoxin Ma

Since the discovery of JAK2V617F tyrosine kinase-activating mutation, several genes have been found mutated in myeloproliferative neoplasms (MPNs). FLT3-ITD, NPM1, and DNMT3A mutations frequently occurred in AML patients and have been found conferred with myeloproliferative neoplasms in mouse model. Therefore, we sought to search for mutations in JAK2V617F, FLT3-ITD, NPM1, and DNMT3A in 129 cas...

Journal: :Haematologica 2008
Arcangelo Liso Filippo Castiglione Antonio Cappuccio Fabrizio Stracci Richard F Schlenk Sergio Amadori Christian Thiede Susanne Schnittger Peter J M Valk Konstanze Döhner Massimo F Martelli Markus Schaich Jürgen Krauter Arnold Ganser Maria P Martelli Niccolò Bolli Bob Löwenberg Torsten Haferlach Gerhard Ehninger Franco Mandelli Hartmut Döhner Franziska Michor Brunangelo Falini

Acute myeloid leukemia with mutated NPM1 gene and aberrant cytoplasmic expression of nucleophosmin (NPMc(+) acute myeloid leukemia) shows distinctive biological and clinical features. Experimental evidence of the oncogenic potential of the nucleophosmin mutant is, however, still lacking, and it is unclear whether other genetic lesion(s), e.g. FLT3 internal tandem duplication, cooperate with NPM...

2008
Arcangelo Liso Filippo Castiglione Antonio Cappuccio Fabrizio Stracci Richard F. Schlenk Sergio Amadori Christian Thiede Susanne Schnittger Peter J.M. Valk Konstanze Döhner Massimo F. Martelli Markus Schaich Jürgen Krauter Arnold Ganser Maria P. Martelli Niccolò Bolli Bob Löwenberg Torsten Haferlach Gerhard Ehninger Franco Mandelli Hartmut Döhner Franziska Michor Brunangelo Falini

Acute myeloid leukemia with mutated NPM1 gene and aberrant cytoplasmic expression of nucleophosmin (NPMc acute myeloid leukemia) shows distinctive biological and clinical features. Experimental evidence of the oncogenic potential of the nucleophosmin mutant is, however, still lacking, and it is unclear whether other genetic lesion(s), e.g. FLT3 internal tandem duplication, cooperate with NPM1 m...

Journal: :Journal of the Egyptian National Cancer Institute 2011
Neemat Kassem Alaa Abel Hamid Tarek Attia Sherif Baathallah Somaya Mahmoud Eman Moemen Ezzat Safwat Mohamed Khalaf Olfat Shaker

UNLABELLED Mutations of the nucleophosmin (NPM-1) gene have been reported in 50-60% of acute myeloid leukemia (AML) patients with normal karyotype. This work was designed to study the prevalence and nature of NPM1 gene mutations in a group of Egyptian patients with AML to get an idea about the profile of NPM1 gene mutations in our society. In 45 previously untreated patients with de novo AML, p...

2014
Omer Ziv Amit Zeisel Nataly Mirlas-Neisberg Umakanta Swain Reinat Nevo Nir Ben-Chetrit Maria Paola Martelli Roberta Rossi Stefan Schiesser Christine E. Canman Thomas Carell Nicholas E. Geacintov Brunangelo Falini Eytan Domany Zvi Livneh

Cells cope with replication-blocking lesions via translesion DNA synthesis (TLS). TLS is carried out by low-fidelity DNA polymerases that replicate across lesions, thereby preventing genome instability at the cost of increased point mutations. Here we perform a two-stage siRNA-based functional screen for mammalian TLS genes and identify 17 validated TLS genes. One of the genes, NPM1, is frequen...

Journal: :Annals of Oncology 2022

Molecular diagnostics for the detection and characterization of genetic variants is based in majority cases on PCR technology. Since primers probes are designed according to nucleotide sequence DNA region specific mutation, technology can only be applied if mutation well known. In where mutated a gene may have variable sequences, different between patients, set each variant necessary, which inv...

Journal: :Blood 2005
Giovanni Cazzaniga Maria Grazia Dell'Oro Cristina Mecucci Emanuela Giarin Riccardo Masetti Vincenzo Rossi Franco Locatelli Massimo F Martelli Giuseppe Basso Andrea Pession Andrea Biondi Brunangelo Falini

Nucleophosmin (NPM) is a nucleocytoplasmic shuttling protein involved in leukemia-associated chromosomal translocations, and it regulates the alternate reading frame (ARF)-p53 tumor-suppressor pathway. Recently, it has been demonstrated that mutations of the NPM1 gene alter the protein at its C-terminal, causing its cytoplasmic localization. Cytoplasmic NPM was detected in 35% of adult patients...

Journal: :Journal of clinical oncology : official journal of the American Society of Clinical Oncology 2015
Rosemary E Gale Katarina Lamb Christopher Allen Dima El-Sharkawi Cassandra Stowe Sarah Jenkinson Steven Tinsley Glenda Dickson Alan K Burnett Robert K Hills David C Linch

PURPOSE To evaluate the impact of DNMT3A mutations on outcome in younger patients with cytogenetic intermediate-risk acute myeloid leukemia. PATIENTS AND METHODS Diagnostic samples from 914 patients (97% < 60 years old) were screened for mutations in DNMT3A exons 13 to 23. Clinical outcome was evaluated according to presence or absence of a mutation and stratified according to type of mutatio...

2016
Nasrin Alizad Ghandforoush Bahram Chahardouli Shahrbano Rostami Habibeh Ghadimi Ali Ghasemi Kamran Alimoghaddam Ardeshir Ghavamzadeh Fatemeh Nadali

BACKGROUND Minimal residual disease (MRD) tests provide early identification of hematologic relapse and timely management of acute myeloid leukemia (AML) patients. Approximately, 50% of AML patients do not have clonal chromosomal aberrations and categorize as a cytogenetically normal acute myeloid leukemia (CN-AML). About 60% of adult CN-AML has a mutation in exon 12 of NPM1 gene. This mutation...

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