نتایج جستجو برای: myo15a

تعداد نتایج: 60  

2012
Oscar Diaz-Horta Duygu Duman Joseph Foster Aslı Sırmacı Michael Gonzalez Nejat Mahdieh Nikou Fotouhi Mortaza Bonyadi Filiz Başak Cengiz Ibis Menendez Rick H. Ulloa Yvonne J. K. Edwards Stephan Züchner Susan Blanton Mustafa Tekin

Identification of the pathogenic mutations underlying autosomal recessive nonsyndromic hearing loss (ARNSHL) is difficult, since causative mutations in 39 different genes have so far been reported. After excluding mutations in the most common ARNSHL gene, GJB2, via Sanger sequencing, we performed whole-exome sequencing (WES) in 30 individuals from 20 unrelated multiplex consanguineous families ...

2014
María J Aparisi Elena Aller Carla Fuster-García Gema García-García Regina Rodrigo Rafael P Vázquez-Manrique Fiona Blanco-Kelly Carmen Ayuso Anne-Françoise Roux Teresa Jaijo José M Millán

BACKGROUND Usher syndrome is an autosomal recessive disease that associates sensorineural hearing loss, retinitis pigmentosa and, in some cases, vestibular dysfunction. It is clinically and genetically heterogeneous. To date, 10 genes have been associated with the disease, making its molecular diagnosis based on Sanger sequencing, expensive and time-consuming. Consequently, the aim of the prese...

2013
Maiko Miyagawa Takehiko Naito Shin-ya Nishio Naoyuki Kamatani Shin-ichi Usami

Target exon resequencing using Massively Parallel DNA Sequencing (MPS) is a new powerful strategy to discover causative genes in rare Mendelian disorders such as deafness. We attempted to identify genomic variations responsible for deafness by massive sequencing of the exons of 112 target candidate genes. By the analysis of 216randomly selected Japanese deafness patients (120 early-onset and 96...

Journal: :In vivo 2012
Michiko Watanabe Nobutake Akiyama Yoshinobu Manome Noriko Hasegawa

BACKGROUND We have established a mouse model of spontaneous deafness by sib-inbreeding over 10 years. The mouse was designated as kuru(2) and has been previously reported in this Journal. MATERIALS AND METHODS In order to identify the genetic abnormality, the mouse was back-crossed to Mus musculus castaneus (CAST), and myosine 15 or myoXV on chromosome 11 was assumed to be the responsive gene...

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