نتایج جستجو برای: myelodysplastic syndrome

تعداد نتایج: 625034  

Journal: :Russian Journal of Children Hematology and Oncology 2018

Journal: :Nihon Naika Gakkai Zasshi 2012

Journal: :International journal of hematology 2003
Ghulam Mufti Alan F List Steven D Gore Aloysius Y L Ho

The last decade has witnessed a multistep evolution in the understanding of the natural history, clinical manifestations, and some of the molecular mechanisms that underlie the ineffective hematopoiesis and leukemic transformation in the myelodysplastic syndrome (MDS). The international prognostic scoring system, FAB, and WHO classifications have helped define specific subgroups with their char...

1998
Jeanne E. Anderson Theo M. de Witte

The hallmark of neoplastic disease, including myelodysplastic syndrome (MDS), is clonal proliferation of cells. Clonal proliferation is the consequence of acquired somatic mutation that confers a proliferative advantage to cells. For the myelodysplastic syndrome, the ability to identify clonally derived cells has provided valuable information about the molecular pathogenesis of the disease, the...

Journal: :Blood 2011
Kelly A McGowan Wendy W Pang Rashmi Bhardwaj Marcelina G Perez John V Pluvinage Bertil E Glader Reem Malek Susan M Mendrysa Irving L Weissman Christopher Y Park Gregory S Barsh

Reduced gene dosage of ribosomal protein subunits has been implicated in 5q- myelodysplastic syndrome and Diamond Blackfan anemia, but the cellular and pathophysiologic defects associated with these conditions are enigmatic. Using conditional inactivation of the ribosomal protein S6 gene in laboratory mice, we found that reduced ribosomal protein gene dosage recapitulates cardinal features of t...

Journal: :Haematologica 2012
Csaba Bödör Aline Renneville Matthew Smith Aurélie Charazac Sameena Iqbal Pascaline Etancelin Jamie Cavenagh Michael J Barnett Karolina Kramarzová Biju Krishnan András Matolcsy Claude Preudhomme Jude Fitzgibbon Carolyn Owen

While most myelodysplastic syndrome/acute myeloid leukemia cases are sporadic, rare familial cases occur and provide some insight into leukemogenesis. The most clearly defined familial cases result from inherited mutations in RUNX1 or CEBPA. Recently, novel germline mutations in GATA2 have been reported. We, therefore, investigated individuals from families with one or more first-degree relativ...

Journal: :Annual review of medicine 2005
Wolf-K Hofmann H Phillip Koeffler

During the past 15 years, important progress has been made in the understanding of the biology and prognosis of myelodysplastic syndrome (MDS). MDS is a clonal disorder characterized by ineffective hematopoiesis, which can lead to either fatal cytopenias or acute myelogenous leukemia (AML). Risk-adapted treatment strategies were established because of the high median age (60-75 years) of the MD...

2011
Olivier Theisen Jean-Luc Lai Olivier Nibourel Catherine Roche-Lestienne

Disease Myelodysplastic syndrome (21 cases), Acute Myeloid Leukemia (5 cases), Chronic Myelomonocytic Leukemia (1 case). Phenotype/cell stem origin Thrombopenia (90%) with anemia (60%). Dysplastic changes in bone marrow: dyserythropoeisis associated with dysgranulopoieisis and/or dysmegakaryocytopoeisis. Epidemiology The frequency of ider(20q) is estimated at 0.49% in myelodysplastic syndrome a...

2014
Mehmet Hilmi Dogu Ismail Sari Sibel Hacioglu Ali Keskin

5q-syndrome is a special subgroup of myelodysplastic syndrome in terms of follow-up and treatment. Lenalidomide is an immunomodulatory drug that is frequently used in the treatment of multiple myeloma. Some clinical studies have shown that lenalidomide treatment is effective in 5q syndrome and significantly decreases the transfusion dependency in these patients. In this paper, we would like to ...

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