نتایج جستجو برای: kit

تعداد نتایج: 29127  

Journal: :Proceedings of the National Academy of Sciences of the United States of America 1998
K Akashi M Kondo I L Weissman

Most mouse thymocytes undergoing positive selection are found on one of two pathways; the c-Kit+ and the c-Kit- pathways. Here, we show that c-Kit and interleukin-7 receptor (IL-7R)-mediated signals support positive selection during the transition from the subpopulation that first expresses cell surface T cell receptor (TCR)-the TCRalpha/betaloCD4(int)/CD8(int) (DPint) c-Kit+ cells to TCRalpha/...

Journal: :Molecular cancer therapeutics 2015
Marianne Le Gall Ronan Crépin Madeline Neiveyans Christian Auclair Yongfeng Fan Yu Zhou James D Marks André Pèlegrin Marie-Alix Poul

KIT is a cell surface tyrosine kinase receptor whose ligand stem cell factor (SCF) triggers homodimerization and activation of downstream effector pathways involved in cell survival, proliferation, homing, or differentiation. KIT-activating mutations are major oncogenic drivers in subsets of acute myeloid leukemia (AML), in mast cell leukemia, and in gastrointestinal stromal tumors (GIST). The ...

Journal: :Slagmark - Tidsskrift for idéhistorie 1970

Journal: :Behavior Analysis in Practice 2013

Journal: :The Journal of Architecture 2020

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2007
Tianhua Guo Narasimhan P Agaram Grace C Wong Glory Hom David D'Adamo Robert G Maki Gary K Schwartz Darren Veach Bayard D Clarkson Samuel Singer Ronald P DeMatteo Peter Besmer Cristina R Antonescu

PURPOSE Resistance is commonly acquired in patients with metastatic gastrointestinal stromal tumor who are treated with imatinib mesylate, often due to the development of secondary mutations in the KIT kinase domain. We sought to investigate the efficacy of second-line tyrosine kinase inhibitors, such as sorafenib, dasatinib, and nilotinib, against the commonly observed imatinib-resistant KIT m...

2015
Xiaoning Gao Ji Lin Li Gao Ailing Deng Xiaolin Lu Yonghui Li Lili Wang Li Yu

The reason that a certain subgroup of acute myeloid leukemia (AML) patients with t(8;21) translocation (generating the AML1/ETO fusion gene) displays a poor survival remains elusive. The proto-oncogene c-kit is expressed in approximately 80% of AML cases. The kinase domain mutation of the c-kit gene, one of the most common gain-of-function mutations associated with t(8;21) AML, predicts higher ...

Journal: :International journal of oncology 2009
Xiaomin Zheng Claudia Oancea Reinhard Henschler Martin Ruthardt

Acute myeloid leukemia (AML) is caused by the cooperation between class I, mostly mutated receptor tyrosine kinases (RTK), and class II oncoproteins, chimeric transcription factors derived from chromosomal translocations. The blasts of 80-90% of AML-patients are positive for the RTK c-Kit. In about 50% of the 'core binding factor' (CBF)-AMLs, c-Kit harbors additional gain-of-function mutations,...

Journal: :Development 1996
F Bernex P De Sepulveda C Kress C Elbaz C Delouis J J Panthier

In the mouse, the Kit receptor and its ligand, the stem cell factor (SCF), are encoded at the W/Kit and Steel loci, respectively. The Kit/SCF transduction pathway is involved in promoting cellular migration, proliferation and/or survival of melanoblasts, hematopoietic progenitors and primordial germ cells. Furthermore, a functional Kit/SCF pathway is required for the development of interstitial...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2011
Yan Kong Lu Si Yanyan Zhu Xiaowei Xu Christopher L Corless Keith T Flaherty Li Li Haifu Li Xinan Sheng Chuanliang Cui Zhihong Chi Siming Li Mei Han Lili Mao Aiping Lu Jun Guo

PURPOSE KIT aberrations were described in acral and mucosal melanomas in largely Caucasian populations. Asian populations are more prone to develop acral and mucosal than cutaneous melanomas, and may harbor a high frequency of KIT aberrations. EXPERIMENTAL DESIGN Melanoma subtypes (n = 502) were analyzed histologically to determine melanoma subtype. Tissue samples were analyzed for mutations ...

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