نتایج جستجو برای: juvenile hemochromatosis

تعداد نتایج: 48167  

Journal: :Journal of medical genetics 2004
K J H Robson A T Merryweather-Clarke E Cadet V Viprakasit M G Zaahl J J Pointon D J Weatherall J Rochette

Mutations in the hepcidin gene HAMP and the hemojuvelin gene HJV have recently been shown to result in juvenile haemochromatosis (JH). Hepcidin is an antimicrobial peptide that plays a key role in regulating intestinal iron absorption. Hepcidin levels are reduced in patients with haemochromatosis due to mutations in the HFE and HJV genes. Digenic inheritance of mutations in HFE and HAMP can res...

Journal: :Haematologica 1995
G Farina C Pedrotti P Cerani A Rovati E Strada G Bergamaschi L Montanari

Heart failure and hypogonadotropic hypogonadism are the most frequent clinical problems encountered in patients with juvenile idiopathic hemochromatosis (JIH). In this context, amenorrhea is one of the first symptoms in female patients, and hormone therapy must be added to phlebotomy to restore menstrual cycles. Here we report the case of a woman in childbearing age with hypogonadotropic hypogo...

Journal: :Blood 2004
Carmela Lanzara Antonella Roetto Filomena Daraio Silvain Rivard Romina Ficarella Hervey Simard Timothy M Cox Mario Cazzola Alberto Piperno Anne-Paule Gimenez-Roqueplo Paola Grammatico Stefano Volinia Paolo Gasparini Clara Camaschella

Juvenile or type 2 hemochromatosis (JH) is transmitted as a recessive trait that leads to severe iron overload and organ damage typically before age 30 years. Linkage to a locus on chromosome 1q has been found in most patients with JH. The recently identified causal gene encodes hemojuvelin, a protein with a proposed crucial role in iron metabolism. A second, rare type of JH, with clinical expr...

Journal: :Environmental Health Perspectives 2004
Robert O Wright Edwin K Silverman Joel Schwartz Shring-Wern Tsaih Jody Senter David Sparrow Scott T Weiss Antonio Aro Howard Hu

Because body iron burden is inversely associated with lead absorption, genes associated with hemochromatosis may modify body lead burden. Our objective was to determine whether the C282Y and/or H63D hemochromatosis gene (HFE) is associated with body lead burden. Patella and tibia lead levels were measured by K X-ray fluorescence in subjects from the Normative Aging Study. DNA samples were genot...

Journal: :Hematology. American Society of Hematology. Education Program 2003
Ernest Beutler A Victor Hoffbrand James D Cook

In the past seven years numerous genes that influence iron homeostasis have been discovered. Dr. Beutler provides a brief overview of these genes, genes that encode HFE, DMT-1, ferroportin, transferrin receptor 2, hephaestin, and hepcidin to lay the groundwork for a discussion of the various clinical forms of iron storage disease and how they differ from one another. In Section I, Dr. Beutler a...

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