نتایج جستجو برای: insolubility

تعداد نتایج: 737  

Journal: :Annals of the rheumatic diseases 1971
J K van der Korst A G Lansink A E van Hooft-Aarnoutse

The solubility of human costal cartilage during exhaustive digestion with proteases (van der Korst, Sokoloff, and Miller, 1968) or 5 M guanidine-HCI (Miller, van der Korst, and Sokoloff, 1969) decreases with ageing. These observations stimulated us to investigate the quantitative effect of glycosaminoglycan and protein-degrading enzymes on costal cartilage in regard to age. The first enzyme stu...

2016
Mathias Lang Alexandra Schade Stefan Bräse

Herein we report the syntheses of two porous hyper-crosslinked polymers (HCPs) via thiol-yne reaction with rigid tetrahedral and pseudo-octahedral core structures. Sorption measurements with nitrogen gas at 77 K revealed BET-surface areas up to 650 m²/g. Those networks also showed a high thermal stability as well as insolubility in common organic solvents.

Journal: :Cell 2005
Joseph Lin Andrey S. Shaw

The role of plasma-membrane microdomains in the organization of signaling proteins has been a controversial topic in T cell signaling. In this issue of Cell, use of single-molecule fluorescence suggests that protein-protein interactions, not detergent insolubility, regulate the assembly of signaling complexes in the plasma membrane.

2012
Ewa Damrath Melanie V. Heck Suzana Gispert Mekhman Azizov Joachim Nowock Carola Seifried Udo Rüb Michael Walter Georg Auburger

Spinocerebellar Ataxia Type 2 (SCA2) is caused by expansion of a polyglutamine encoding triplet repeat in the human ATXN2 gene beyond (CAG)(31). This is thought to mediate toxic gain-of-function by protein aggregation and to affect RNA processing, resulting in degenerative processes affecting preferentially cerebellar neurons. As a faithful animal model, we generated a knock-in mouse replacing ...

2014
Ida M. Klang Birgit Schilling Dylan J. Sorensen Alexandria K. Sahu Pankaj Kapahi Julie K. Andersen Peter Swoboda David W. Killilea Bradford W. Gibson Gordon J. Lithgow

Many late-onset proteotoxic diseases are accompanied by a disruption in homeostasis of metals (metallostasis) including iron, copper and zinc. Although aging is the most prominent risk factor for these disorders, the impact of aging on metallostasis and its role in proteotoxic disease remain poorly understood. Moreover, it is not clear whether a loss of metallostasis influences normal aging. We...

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