نتایج جستجو برای: ifnβ

تعداد نتایج: 404  

2017
Jenny Link Ryan Ramanujam Michael Auer Malin Ryner Signe Hässler Delphine Bachelet Cyprien Mbogning Clemens Warnke Dorothea Buck Poul Erik Hyldgaard Jensen Claudia Sievers Kathleen Ingenhoven Nicolas Fissolo Raija Lindberg Verena Grummel Naoimh Donnellan Manuel Comabella Xavier Montalban Bernd Kieseier Per Soelberg Sørensen Hans-Peter Hartung Tobias Derfuss Andy Lawton Dan Sikkema Marc Pallardy Bernhard Hemmer Florian Deisenhammer Philippe Broët Pierre Dönnes Julie Davidson Anna Fogdell-Hahn

Antibodies against biopharmaceuticals (anti-drug antibodies, ADA) have been a well-integrated part of the clinical care of multiple sclerosis (MS) in several European countries. ADA data generated in Europe during the more than 10 years of ADA monitoring in MS patients treated with interferon beta (IFNβ) and natalizumab have been pooled and characterized through collaboration within a European ...

2017
Jana Libertinova Eva Meluzinova Vaclav Matoska Miroslav Zajac Ivana Kovarova Eva Havrdova Dana Horakova Ales Tomek Petr Marusic Martin Bojar

BACKGROUND Multiple sclerosis (MS) patients treated with interferon beta (IFNβ) are at risk of a declining response to treatment because of the production of IFNβ-neutralizing antibodies (NAbs). The expression of Myxovirus resistance protein A (MxA) mRNA is regarded as a marker of IFNβ bioactivity. AIMS The aim of this study was to analyze the kinetics of MxA mRNA expression during long-term ...

2012
Megan L. Mittelstadt Rekha C. Patel

Matrix metalloproteinase-9 (MMP-9) is a 92 kDa zinc-dependant endopeptidase that degrades components of the extracellular matrix. Increased expression of MMP-9 is implicated in many pathological conditions including metastatic cancer, multiple sclerosis, and atherosclerosis. Although it has been widely noted that interferon-β (IFNβ) downregulates both the basal and phorbol 12-myristate 13-aceta...

2011
Richard A. Rudick M. R. Sandhya Rani Yaomin Xu Jar-Chi Lee Jie Na Jennifer Shrock Anupama Josyula Elizabeth Fisher Richard M. Ransohoff

BACKGROUND Interferon-beta (IFNβ) is used to inhibit disease activity in multiple sclerosis (MS), but its mechanisms of action are incompletely understood, individual treatment response varies, and biological markers predicting response to treatment have yet to be identified. METHODS The relationship between the molecular response to IFNβ and treatment response was determined in 85 patients u...

2014
Edward Fox Barbara Green Clyde Markowitz Ronald Murray Andrew D Goodman Stephen J Glenski Pippa Loupe Jo Nita Cogburn

BACKGROUND Many patients with relapsing-remitting multiple sclerosis (MS) treated with high-dose interferon-β (IFNβ) develop serum binding antibodies (BAb) and neutralizing antibodies (NAb). NAb reduces the biological activity of IFNβ, which contributes to clinical failure in these patients. We investigated whether access to antibody (Ab) test results would alter usual care of (IFNβ)-treated pa...

2015
Afsaneh Shirani Yinshan Zhao John Petkau Paul Gustafson Mohammad Ehsanul Karim Charity Evans Elaine Kingwell Mia L van der Kop Joel Oger Helen Tremlett

BACKGROUND We examined (1) patient characteristics and disease-modifying drug (DMD) exposure in late-onset (LOMS, ≥50 years at symptom onset) versus adult-onset (AOMS, 18-<50 years) MS and (2) the association between interferon-beta (IFNβ) and disability progression in older relapsing-onset MS adults (≥50 years). METHODS This retrospective study (1980-2004, British Columbia, Canada) included ...

2013
Maryam Kay Zohreh Hojati Fariba Dehghanian

Multiple sclerosis (MS) is one of the most important autoimmune diseases recognized by demyelination and axonal lesion. It is the most common cause of disability in the young population. Various immunomodulatory and immunosuppressive therapies, including different formulations of interferon beta (IFNβ), glatiramer acetate (GA), mitoxantrone, and natalizumab are available for this disease. Howev...

2016
Delphine Bachelet Signe Hässler Cyprien Mbogning Jenny Link Malin Ryner Ryan Ramanujam Michael Auer Poul Erik Hyldgaard Jensen Nils Koch-Henriksen Clemens Warnke Kathleen Ingenhoven Dorothea Buck Verena Grummel Andy Lawton Naoimh Donnellan Agnès Hincelin-Mery Dan Sikkema Marc Pallardy Bernd Kieseier Bernard Hemmer Hans Peter Hartung Per Soelberg Sorensen Florian Deisenhammer Pierre Dönnes Julie Davidson Anna Fogdell-Hahn Philippe Broët

Immunogenicity of biopharmaceutical products in multiple sclerosis is a frequent side effect which has a multifactorial etiology. Here we study associations between anti-drug antibody (ADA) occurrence and demographic and clinical factors. Retrospective data from routine ADA test laboratories in Sweden, Denmark, Austria and Germany (Dusseldorf group) and from one research study in Germany (Munic...

Journal: :Neurology 2012
Florian Deisenhammer Harald Hegen

OBJECTIVE To report the long-term safety and efficacy results from CAMMS223 comparing alemtuzumab with interferon β-1a in early, active relapsing-remitting multiple sclerosis (RRMS). What are the long-term effects of alemtuzumab treatment, received 36 to 48 months previously, on relapse and disability in early, active RRMS? This study provides evidence of the effectiveness of alemtuzumab in red...

2013
Paul I. Creeke Rachel A. Farrell

Biopharmaceuticals are drugs which are based on naturally occurring proteins (antibodies, receptors, cytokines, enzymes, toxins), nucleic acids (DNA, RNA) or attenuated microorganisms. Immunogenicity of these agents has been commonly described and refers to a specific antidrug antibody response. Such immunogenicity represents a major factor impairing the efficacy of biopharmaceuticals due to bi...

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