نتایج جستجو برای: cpg island

تعداد نتایج: 66989  

Journal: :Molecular pharmacology 2018
Toshihiro Tanaka Takeshi Hirota Ichiro Ieiri

Multidrug and toxin extrusion protein 1 (MATE1), which is encoded by solute carrier 47A1 (SLC47A1), mediates the excretion of organic cations into bile and urine. Some genetic variants in human MATE1 altered its transport function in in vitro experiments; however, differences in the pharmacokinetics of substrate drugs cannot be explained by genetic variations in humans. In this study, we invest...

2013
Yongmei Zhang Chenxiao Li Yijun Zhang Haoxiang Zhu Yaoyue Kang Hongyan Liu Jinyu Wang Yanli Qin Richeng Mao Yi Xie Yuxian Huang Jiming Zhang

DNA methylation is being increasingly recognized to play a role in regulation of hepatitis B virus (HBV) gene expression. The aim of this study was to compare the CpG island distribution among different HBV genotypes. We analyzed 176 full-length HBV genomic sequences obtained from the GenBank database, belonging to genotypes A through J, to identify the CpG islands in the HBV genomes. Our resul...

Journal: :Cancer research 2001
X Lin K Asgari M J Putzi W R Gage X Yu B S Cornblatt A Kumar S Piantadosi T L DeWeese A M De Marzo W G Nelson

Among the many somatic genome alterations present in cancer cells, changes in DNA methylation may represent reversible "epigenetic" lesions, rather than irreversible "genetic" alterations. Cancer cell DNA is typically characterized by increases in the methylation of CpG dinucleotides clustered into CpG islands, near the transcriptional regulatory regions of critical genes, and by an overall red...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2002
Sheng Zhong Mandy W Tang Winnie Yeo Cuiling Liu Y M Dennis Lo Philip J Johnson

PURPOSE AND EXPERIMENTAL DESIGN Glutathione S-transferases, enzymes that defend cells against damage mediated by oxidant and electrophilic carcinogens, may be critical determinants of cancer pathogenesis. In this report, we assess the role of epigenetic silencing of the GSTP1 gene, a gene encoding the pi-class glutathione S-transferase, in the pathogenesis of hepatitis B virus (HBV)-associated ...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 1996
R G Lapidus A T Ferguson Y L Ottaviano F F Parl H S Smith S A Weitzman S B Baylin J P Issa N E Davidson

Hormonal factors have a profound influence on the development, treatment, and outcome of breast cancer. The absence of steroid hormone receptors is highly correlated with resistance to antihormonal treatments. Work in cultured human breast cancer cell lines has shown that the absence of estrogen receptor (ER) gene expression in ER- cells is associated with extensive methylation of the ER gene 5...

Journal: :Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology 2004
Nigel J Belshaw Giles O Elliott Elizabeth A Williams David M Bradburn Sarah J Mills John C Mathers Ian T Johnson

Hypermethylation of cytosine residues in the CpG islands of tumor suppressor genes is a key mechanism of colorectal carcinogenesis. Detection and quantification of CpG island methylation in human DNA isolated from stools might provide a novel strategy for the detection and investigation of colorectal neoplasia. To explore the feasibility of this approach, colorectal biopsies and fecal samples w...

Journal: :Human molecular genetics 2001
M M Pao M Tsutsumi G Liang E Uzvolgyi F A Gonzales P A Jones

The 5' region for the endothelin receptor B (EDNRB) gene is a complex CpG island giving rise to four individual transcripts initiating within the island. Here, for the first time, we analyze the relationship between methylation and gene expression in a CpG island located in the 5' region of a gene with multiple transcription start sites. The CpG island was unmethylated in normal prostate and bl...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2012
Seung-Mo Hong Noriyuki Omura Audrey Vincent Ang Li Spencer Knight Jun Yu Ralph H Hruban Michael Goggins

PURPOSE Intraductal papillary mucinous neoplasms (IPMN) are precursors to infiltrating pancreatic ductal adenocarcinomas. Widespread epigenetic alterations are characteristic of many cancers, yet few studies have systematically analyzed epigenetic alterations of neoplastic precursors. Our goal was to conduct genome-wide CpG island methylation profiling to identify aberrantly methylated loci in ...

Journal: :Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology 2009
Shauna N Vasilatos Gloria Broadwater William T Barry Joseph C Baker Siya Lem Eric C Dietze Gregory R Bean Andrew D Bryson Patrick G Pilie Vanessa Goldenberg David Skaar Carolyn Paisie Alejandro Torres-Hernandez Tracey L Grant Lee G Wilke Catherine Ibarra-Drendall Julie H Ostrander Nicholas C D'Amato Carola Zalles Randy Jirtle Valerie M Weaver Victoria L Seewaldt

BACKGROUND Only 5% of all breast cancers are the result of BRCA1/2 mutations. Methylation silencing of tumor suppressor genes is well described in sporadic breast cancer; however, its role in familial breast cancer is not known. METHODS CpG island promoter methylation was tested in the initial random periareolar fine-needle aspiration sample from 109 asymptomatic women at high risk for breast...

2010
Neil P. Blackledge Jin C. Zhou Michael Y. Tolstorukov Anca M. Farcas Peter J. Park Robert J. Klose

In higher eukaryotes, up to 70% of genes have high levels of nonmethylated cytosine/guanine base pairs (CpGs) surrounding promoters and gene regulatory units. These features, called CpG islands, were identified over 20 years ago, but there remains little mechanistic evidence to suggest how these enigmatic elements contribute to promoter function, except that they are refractory to epigenetic si...

نمودار تعداد نتایج جستجو در هر سال

با کلیک روی نمودار نتایج را به سال انتشار فیلتر کنید