نتایج جستجو برای: alveolar epithelial

تعداد نتایج: 195783  

Journal: :Acta medica Okayama 1977
K Suwa

The effect of an intravenous injection of squid-ink (sepia-melanin) solution on adult mouse spheroid alveolar epithelial cells was observed by the electron microscope. Sepia-melanin particles were seen in all alveolar wall cells examined that seems to suggest the entrance of sepia-melanin particles into the spheroid alveolar epithlial cells from the alveolar blood capillary. In cases of large p...

Journal: :American Journal of Respiratory Cell and Molecular Biology 2017

Journal: :Thorax 2008
G D Perkins F Gao D R Thickett

BACKGROUND Acute lung injury is an important cause of respiratory failure in the critically ill patient. It is caused by damage to the alveolar barrier with subsequent alveolar flooding leading to the development of refractory hypoxaemia. beta Agonists stimulate alveolar fluid clearance in animal models of lung injury. In a clinical trial (BALTI-1), intravenous beta agonists reduced extravascul...

Journal: :Journal of applied physiology 2011
Anthony D Dorr Michael R Wilson Kenji Wakabayashi Alicia C Waite Brijesh V Patel Nico van Rooijen Kieran P O'Dea Masao Takata

Elevated soluble tumor necrosis factor-α receptor (sTNFR) levels in bronchoalveolar lavage fluid (BALF) are associated with poor patient outcome in acute lung injury (ALI). The mechanisms underlying these increases are unknown, but it is possible that pulmonary inflammation and increased alveolar epithelial permeability may individually contribute. We investigated mechanisms of elevated BALF sT...

Journal: :Infection and immunity 2012
Marybeth Langer Elizabeth Stewart Duggan John Leland Booth Vineet Indrajit Patel Ryan A Zander Robert Silasi-Mansat Vijay Ramani Tibor Zoltan Veres Frauke Prenzler Katherina Sewald Daniel M Williams Kenneth Mark Coggeshall Shanjana Awasthi Florea Lupu Dennis Burian Jimmy Dale Ballard Armin Braun Jordan Patrick Metcalf

The lung is the site of entry for Bacillus anthracis in inhalation anthrax, the deadliest form of the disease. Bacillus anthracis produces virulence toxins required for disease. Alveolar macrophages were considered the primary target of the Bacillus anthracis virulence factor lethal toxin because lethal toxin inhibits mouse macrophages through cleavage of MEK signaling pathway components, but w...

Journal: :American journal of physiology. Lung cellular and molecular physiology 2003
Scott M O'Grady So Yeong Lee

Electrolyte transport across the adult alveolar epithelium plays an important role in maintaining a thin fluid layer along the apical surface of the alveolus that facilitates gas exchange across the epithelium. Most of the work published on the transport properties of alveolar epithelial cells has focused on the mechanisms and regulation of Na(+) transport and, in particular, the role of amilor...

Journal: :American journal of respiratory cell and molecular biology 2003
Fushan Wang Brandy Daugherty Lisa L Keise Zhangyong Wei Joseph P Foley Rashmin C Savani Michael Koval

Claudins are proteins that participate in epithelial barrier function and regulate paracellular permeability. By immunohistochemistry of adult rat lung sections, claudin-3, claudin-4, and claudin-5 were found to be co-expressed by type II alveolar epithelial cells. Claudin-3 and claudin-4 were also co-expressed by some alveolar epithelial cells adjacent to type II cells. In contrast, claudin-5 ...

Journal: :European medical journal 2021

This article relates the current pathophysiologic and radiologic findings to fundamental idea of acute lung epithelial infection, alveolar inflammation causing leak into interstitial space, subsequent secondary or concurrent endothelial infection dysfunction. Understanding mechanisms timings damage can better inform types ventilatory support required timing targeted pharmacotherapies.

Journal: :The Journal of clinical investigation 1986
R H Simon P D DeHart R F Todd

The damage to pulmonary alveolar epithelial cells that occurs in many inflammatory conditions is thought to be caused in part by phagocytic neutrophils. To investigate this process, we exposed monolayers of purified rat alveolar epithelial cells to stimulated human neutrophils and measured cytotoxicity using a 51Cr-release assay. We found that stimulated neutrophils killed epithelial cells by a...

Journal: :American journal of respiratory cell and molecular biology 2008
Rizwan Manzer Charles A Dinarello Glen McConville Robert J Mason

Ozone is known to produce an acute influx of neutrophils, and alveolar epithelial cells can secrete chemokines and modulate inflammatory processes. However, direct exposure of alveolar epithelial cells and macrophages to ozone (O(3)) produces little chemokine response. To determine if cell-cell interactions might be responsible, we investigated the effect of alveolar macrophage-conditioned medi...

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