نتایج جستجو برای: a3243g mutation

تعداد نتایج: 291433  

Journal: :Diabetes 2004
Camilla Cervin Brita Liljeström Tiinamaija Tuomi Seija Heikkinen Juha S Tapanainen Leif Groop Corrado M Cilio

The aim of this study was characterization of a family carrying two mutations known to cause monogenic forms of diabetes, the M626K mutation in the HNF1alpha gene (MODY3) and the A3243G in mtDNA. Beta-cell function and insulin sensitivity were assessed with the Botnia clamp. Heteroplasmy of the A3243G mutation and variants in type 2 diabetes susceptibility genes were determined, and transcripti...

Journal: :Chang Gung medical journal 2004
Yung-Nien Chen Chia-Wei Liou Chin-Chang Huang Tsu-Kung Lin Yau-Huei Wei

We report on a case of a 48-year-old woman presenting with maternally inherited diabetes mellitus and deafness (MIDD) syndrome. Molecular genetic analysis and clinical evaluation were conducted in the patient and her 4 children to investigate the interrelation between an MIDD-associated mitochondrial DNA (mtDNA) mutation and clinical manifestations. Various symptoms and markers of MIDD, includi...

Journal: :Graefe's Archive for Clinical and Experimental Ophthalmology 2018

Journal: :Journal of the American Society of Nephrology : JASN 2003
Bruno Guéry Gabriel Choukroun Laure-Hélène Noël Pierre Clavel Agnès Rötig Sophie Lebon Pierre Rustin Christine Bellané-Chantelot Béatrice Mougenot Jean-Pierre Grünfeld Dominique Chauveau

The A3243G mutation of the mitochondrial tRNA(Leu) gene has been recently reported in rare patients with focal and segmental glomerulosclerosis (FSGS). However, the full spectrum of systemic and kidney manifestations in adults presenting with this mutation remains poorly defined. Assessment of renal and nonrenal manifestations was performed in nine patients with A3243G mutation and prominent ki...

2013
Hui Liu Yinan Ma Fang Fang Ying Zhang Liping Zou Yanling Yang Sainan Zhu Songtao Wang Xuefei Zheng Pei Pei Lin Li Hairong Wu Yang Xiao Yufeng Xu Liwen Wang Yanyan Cao Hong Pan Yu Qi

The genotype-phenotype relationship in diseases with mtDNA point mutations is still elusive. The maintenance of wild-type mtDNA copy number is essential to the normal mitochondrial oxidative function. This study examined the relationship between mtDNA copy number in blood and urine and disease severity of the patients harboring A3243G mutation. We recruited 115 A3243G patients, in which 28 were...

Journal: :Genetic Testing and Molecular Biomarkers 2013

Journal: :Clinical chemistry 2002
Diane K Hancock Frederick P Schwarz Fenhong Song Lee-Jun C Wong Barbara C Levin

BACKGROUND Most pathogenic human mitochondrial DNA (mtDNA) mutations are heteroplasmic (i.e., mutant and wild-type mtDNA coexist in the same individual) and are difficult to detect when their concentration is a small proportion of that of wild-type mtDNA molecules. We describe a simple methodology to detect low proportions of the single base pair heteroplasmic mutation, A3243G, that has been as...

2017
Negar Sarhangi Fatemeh Khatami Abbasali Keshtkar Ramin Heshmat Rasha Atlasi Mahsa Mohammadamoli

Diabetic Retinopathy (DR) is the most prevalent health problem, which is influenced by environmental and genetic factors with an increasing prevalence. The current systematic review is focused on mtDNA modification, including polymorphism and mutation/deletion, with a direct effect on DR.This systematic search was initially done through PubMed, Cochrane, EMBASE, SCOPUS, and Web of Science witho...

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