نتایج جستجو برای: ژن fhl1

تعداد نتایج: 15963  

Journal: :The Journal of clinical investigation 2011
Qing-Jun Zhang Hou-Zao Chen Lin Wang De-Pei Liu Joseph A Hill Zhi-Ping Liu

Cardiac hypertrophy and failure are accompanied by a reprogramming of gene expression that involves transcription factors and chromatin remodeling enzymes. Little is known about the roles of histone methylation and demethylation in this process. To understand the role of JMJD2A, a histone trimethyl demethylase, in cardiac hypertrophy, we generated mouse lines with heart-specific Jmjd2a deletion...

Journal: :The Journal of clinical investigation 2008
Farah Sheikh Anna Raskin Pao-Hsien Chu Stephan Lange Andrea A Domenighetti Ming Zheng Xingqun Liang Tong Zhang Toshitaka Yajima Yusu Gu Nancy D Dalton Sushil K Mahata Gerald W Dorn Joan Heller Brown Kirk L Peterson Jeffrey H Omens Andrew D McCulloch Ju Chen

The response of cardiomyocytes to biomechanical stress can determine the pathophysiology of hypertrophic cardiac disease, and targeting the pathways regulating these responses is a therapeutic goal. However, little is known about how biomechanical stress is sensed by the cardiomyocyte sarcomere to transduce intracellular hypertrophic signals or how the dysfunction of these pathways may lead to ...

2003
Vinciane Gaussin James E. Tomlinson Christopher L. Antos James N. Topper Eric N. Olson Martin J. Lohse Stephen F. Vatner Dorothy E. Vatner

Background—Although -adrenergic receptor (AR) blockade therapy is beneficial in the treatment of heart failure, little is known regarding the transcriptional mechanisms underlying this salutary action. Methods and Results—In the present study, we screened mice overexpressing Gs , 1AR, 2AR, or protein kinase A to test if a common genomic pathway exists in different models with enhanced -adrenerg...

Journal: :Circulation 2003
Vinciane Gaussin James E Tomlinson Christophe Depre Stefan Engelhardt Christopher L Antos Gen Takagi Lutz Hein James N Topper Stephen B Liggett Eric N Olson Martin J Lohse Stephen F Vatner Dorothy E Vatner

BACKGROUND Although beta-adrenergic receptor (AR) blockade therapy is beneficial in the treatment of heart failure, little is known regarding the transcriptional mechanisms underlying this salutary action. METHODS AND RESULTS In the present study, we screened mice overexpressing Gsalpha, beta1AR, beta2AR, or protein kinase A to test if a common genomic pathway exists in different models with ...

Journal: :Cytogenetics and cell genetics 2000
A Krempler S Kollers R Fries B Brenig

Four and a half LIM domain protein 1 (FHL1) was initially described as an abundant skeletal muscle protein with four LIM domains and a GATA like zinc finger. FHL1 was shown to be expressed in skeletal muscle as well as in a variety of other tissues. Recently, alternatively spliced FHL1 mRNAs were identified coding for C-terminal truncated proteins. The tissue distribution of these variants is m...

Journal: :Genes & development 2014
Britta Knight Slawomir Kubik Bhaswar Ghosh Maria Jessica Bruzzone Marcel Geertz Victoria Martin Nicolas Dénervaud Philippe Jacquet Burak Ozkan Jacques Rougemont Sebastian J Maerkl Félix Naef David Shore

In yeast, ribosome production is controlled transcriptionally by tight coregulation of the 138 ribosomal protein genes (RPGs). RPG promoters display limited sequence homology, and the molecular basis for their coregulation remains largely unknown. Here we identify two prevalent RPG promoter types, both characterized by upstream binding of the general transcription factor (TF) Rap1 followed by t...

Journal: :Chang Gung medical journal 2011
Pao-Hsien Chu Ju Chen

Four and a half LIM domains protein 1 (FHL1) and FHL2, as the name suggests, contain four and a half LIM domain binding proteins. Proteins in this family capable of interacting with many types of proteins, including structural proteins, kinases, and several classes of transcription factors, have been identified. These interactions have been found to have important roles in a variety of fundamen...

Journal: :Journal of Cell Biology 2008

Journal: :Brain : a journal of neurology 2009
Joachim Schessl Ana L Taratuto Caroline Sewry Roberta Battini Steven S Chin Baijayanta Maiti Alberto L Dubrovsky Marcela G Erro Graciela Espada Monica Robertella Maria Saccoliti Patricia Olmos Leslie R Bridges Peter Standring Ying Hu Yaqun Zou Kathryn J Swoboda Mena Scavina Hans-Hilmar Goebel Christina A Mitchell Kevin M Flanigan Francesco Muntoni Carsten G Bönnemann

We recently identified the X-chromosomal four and a half LIM domain gene FHL1 as the causative gene for reducing body myopathy, a disorder characterized by progressive weakness and intracytoplasmic aggregates in muscle that exert reducing activity on menadione nitro-blue-tetrazolium (NBT). The mutations detected in FHL1 affected highly conserved zinc coordinating residues within the second LIM ...

Journal: :Journal of cell science 2011
Laura Leitner Dmitry Shaposhnikov Alexander Mengel Arnaud Descot Sylvia Julien Reinhard Hoffmann Guido Posern

Monomeric actin regulates gene expression through serum response factor (SRF) by inhibiting its transcriptional coactivator myocardin-related transcription factor (MAL/MRTF). Many affected genes encode cytoskeletal components. We have analysed the migratory effects of actin-MAL signalling and of new target genes in non-invasive highly adherent cells. Expression of active MAL impaired migration ...

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