نتایج جستجو برای: volkmanns ischemic contracture

تعداد نتایج: 91023  

Journal: :Journal of applied physiology 2002
Mirit Eynan Tanya Knubuvetz Uri Meiri Gil Navon Gary Gerstenblith Zohar Bromberg Yonathan Hasin Michal Horowitz

Based on our observations of energy sparing in heat-acclimated (AC) rat hearts, we investigated whether changes in preischemic glycogen level, glycolytic rate, and plasma thyroxine level mediate cardioprotection induced in these hearts during ischemia-reperfusion insults. Control (C) (24 degrees C), AC (34 degrees C, 30 days), acclimated-euthyroid (34 degrees C + 3 ng/ml l-thyroxine), and contr...

Journal: :European Journal of Trauma and Emergency Surgery 2007

Journal: :Proceedings of the National Academy of Sciences of the United States of America 1997
G P Matherne J Linden A M Byford N S Gauthier J P Headrick

Activation of myocardial A1 adenosine receptors (A1AR) protects the heart from ischemic injury. In this study transgenic mice were created using the cardiac-specific alpha-myosin heavy chain promoter and rat A1AR cDNA. Heart membranes from two transgene positive lines displayed approximately 1,000-fold overexpression of A1AR (6,574 +/- 965 and 10,691 +/- 1,002 fmol per mg of protein vs. 8 +/- 5...

Journal: :American journal of physiology. Heart and circulatory physiology 2007
Karla Carvajal Elham Zarrinpashneh Ondrej Szarszoi Frederic Joubert Yoni Athea Philippe Mateo Brigitte Gillet Sophie Vaulont Benoit Viollet Xavier Bigard Luc Bertrand Renée Ventura-Clapier Jacqueline A Hoerter

Because the question "is AMP-activated protein kinase (AMPK) alpha(2)-isoform a friend or a foe in the protection of the myocardium against ischemia-reperfusion injury?" is still in debate, we studied the functional consequence of its deletion on the contractility, the energetics, and the respiration of the isolated perfused heart and characterized the response to low-flow ischemia and reperfus...

Journal: :Circulation research 1990
P G Anderson M F Allard G D Thomas S P Bishop S B Digerness

The purpose of this study was to compare the degree of ischemic and hypoxic injury in normal versus hypertrophied rat hearts to investigate basic mechanisms responsible for irreversible myocardial ischemic injury. Hearts from rats with bands placed on the aortic arch at 23 days of age (BAND) and sham-operated rats (SHAM, 8 weeks postoperative) were isolated, perfused with Krebs buffer, and had ...

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