نتایج جستجو برای: vhl

تعداد نتایج: 1947  

Journal: :Transactions of the American Ophthalmological Society 2004
Chi-Chao Chan Youn-Soo Lee Zhengping Zhuang Joseph Hackett Emily Y Chew

PURPOSE Von Hippel-Lindau (VHL) disease is a hereditary cancer syndrome expressed in multiple organs caused by germline alterations of the VHL gene. We have shown VHL deletion in the "stromal" cells of retinal angiomas. The VHL protein-associated complex is a primary ubiquitin ligase for the ubiquitination of hypoxia-inducible factor (HIF). This study examines VHL and ubiquitin expression in op...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 1998
A Pause S Lee K M Lonergan R D Klausner

The inactivation of the von Hippel-Lindau (VHL) tumor suppressor gene predisposes affected individuals to the human VHL cancer syndrome and is associated with sporadic renal cell carcinomas (RCC) and brain hemangioblastomas. VHL-negative 786-0 RCC cells are tumorigenic in nude mice which is suppressed by the reintroduction of VHL. Remarkably, this occurs without affecting the growth rate and ce...

Journal: :International journal of cancer 2008
Jean-Jacques Patard Patricia Fergelot Pierre I Karakiewicz Tobias Klatte Quoc-Dien Trinh Nathalie Rioux-Leclercq Jonathan W Said Arie S Belldegrun Allan J Pantuck

We attempted to describe, in a series of clear cell renal cell carcinoma (RCC), the relationship between CAIX expression, VHL gene mutations, tumor characteristics and outcome. Radical nephrectomy was performed in 100 patients. Genomic DNA was extracted from frozen tumor samples. Four amplimers covering the whole coding sequence of the VHL gene were synthesized by PCR and sequenced. The monoclo...

Journal: :Blood 2009
Ellen van Rooijen Emile E Voest Ive Logister Jeroen Korving Thorsten Schwerte Stefan Schulte-Merker Rachel H Giles Fredericus J van Eeden

We have generated 2 zebrafish lines carrying inactivating germline mutations in the von Hippel-Lindau (VHL) tumor suppressor gene ortholog vhl. Mutant embryos display a general systemic hypoxic response, including the up-regulation of hypoxia-induced genes by 1 day after fertilization and a severe hyperventilation and cardiophysiologic response. The vhl mutants develop polycythemia with concomi...

Journal: :Journal of clinical oncology : official journal of the American Society of Clinical Oncology 2016
Sarah M Nielsen Lindsay Rhodes Ignacio Blanco Wendy K Chung Charis Eng Eamonn R Maher Stéphane Richard Rachel H Giles

Von Hippel-Lindau disease (VHL) is one of the most common inherited neoplasia syndromes and is characterized by highly vascular tumors of the eyes, brain, and spine, as well as benign and malignant tumors and/or cysts of the kidneys, adrenal medullae and sympathetic paraganglia, endolymphatic sac, epididymis, and broad ligament. Since the discovery of the VHL gene in 1993, more than 900 familie...

2005
Lauren Ashbrook Kevin P Hartman

Many environmental factors, including hypoxia, can induce oxidative stress. Cells that are subjected to oxidative stress often generate reactive oxygen species (ROS), which include hydrogen peroxide (H2O2). Familial von Hippel-Lindau (VHL) disease develops in individuals who are born heterotrophic for the wild-typeVHL gene, and who subsequently lose the remaining wt-VHL allele due to mutation. ...

2016
Haley R Noonan Ana M Metelo Caramai N Kamei Randall T Peterson Iain A Drummond Othon Iliopoulos

Patients with von Hippel-Lindau (VHL) disease harbor a germline mutation in the VHL gene leading to the development of several tumor types including clear cell renal cell carcinoma (ccRCC). In addition, the VHL gene is inactivated in over 90% of sporadic ccRCC cases. 'Clear cell' tumors contain large, proliferating cells with 'clear cytoplasm', and a reduced number of cilia. VHL inactivation le...

Journal: :The Journal of biological chemistry 2002
Takumi Kamura Christopher S Brower Ronald C Conaway Joan W Conaway

The multiprotein von Hippel-Lindau (VHL) tumor suppressor (CBC(VHL), Cul2-Elongin BC-VHL) and SCF (Skp1-Cul1/Cdc53-F-box protein) complexes are members of structurally related families of E3 ubiquitin ligases that use a heterodimeric module composed of a member of the Cullin protein family and the RING finger protein Rbx1 (ROC1/Hrt1) to activate ubiquitylation of target proteins by the E2 ubiqu...

Journal: :Endocrine-related cancer 2010
Zoran Erlic Ursula Ploeckinger Alberto Cascon Michael M Hoffmann Laura von Duecker Aurelia Winter Gerit Kammel Janina Bacher Maren Sullivan Berend Isermann Lars Fischer Andreas Raffel Wolfram Trudo Knoefel Matthias Schott Tobias Baumann Oliver Schaefer Tobias Keck Richard P Baum Ioana Milos Mihaela Muresan Mariola Peczkowska Andrzej Januszewicz Kenko Cupisti Anke Tönjes Mathias Fasshauer Jan Langrehr Peter von Wussow Abbas Agaimy Günter Schlimok Regina Lamberts Thorsten Wiech Kurt Werner Schmid Alexander Weber Mercedes Nunez Mercedes Robledo Charis Eng Hartmut P H Neumann

Pancreatic islet cell tumors (ICTs) occur as sporadic neoplasias or as a manifestation of multiple endocrine neoplasia type 1 (MEN1) and von Hippel-Lindau disease (VHL). Molecular classification of ICTs is mandatory for timely diagnosis and surveillance. Systematic comparison of VHL-ICTs and sporadic ICTs has been lacking. Our registry-based approaches used the German NET-Registry with 259 pati...

2015
Jingyao Zhang Jie Ma Xiaoyun Du Dapeng Wu Hong Ai Jigang Bai Shunbin Dong Qinling Yang Kai Qu Yi Lyu Robert K Valenzuela Chang Liu

BACKGROUND Von Hippel-Lindau (VHL) disease is a hereditary tumor disorder caused by mutations or deletions of the VHL gene. Few studies have documented the clinical phenotype and genetic basis of the occurrence of VHL disease in China. This study armed to present clinical and genetic analyses of VHL within a five-generation VHL family from Northwestern China, and summarize the VHL mutations and...

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