نتایج جستجو برای: tumorigenesis antiangiogenesis

تعداد نتایج: 25649  

2012
Eduardo Lasalvia-Prisco Pablo Goldschmidt Felipe Galmarini Silvia Cucchi Jesús Vázquez Martha Aghazarian Eduardo Lasalvia-Galante Wilson Golomar William Gordon

Studies have shown that cancer requires two conditions for tumor progression: cancer cell proliferation and an environment permissive to and conditioned by malignancy. Chemotherapy aims to control the number and proliferation of cancer cells, but it does not effectively control the two best-known conditions of the tumor-permissive environment: neoangiogenesis and tolerogenic immunity. Many mali...

Journal: :Cancer research 2011
Jie Ma Chong-Sheng Chen Todd Blute David J Waxman

The tumor vasculature delivers nutrients, oxygen, and therapeutic agents to tumor cells. Unfortunately, the delivery of anticancer drugs through tumor blood vessels is often inefficient and can constitute an important barrier for cancer treatment. This barrier can sometimes be circumvented by antiangiogenesis-induced normalization of tumor vasculature. However, such normalizing effects are tran...

Journal: :acta medica iranica 0
mojgan mirakholi department of pathology, faculty of medicine and health sciences, university putra malaysia, 43400 upm serdang, malaysia. touraj mahmoudi department of cancer, research center for gastroenterology and liver diseases (rcgld), shahid beheshti university of medical sciences, tehran, iran. mansour heidari department of medical genetics, faculty of medicine, tehran university of medical sciences, tehran, iran. and stem cell preparation unit, farabi eye hospital, tehran university of medical sciences, tehran, iran.

in the retinoblastoma research, it is of great interest to identify molecular markers associated with the genetics of tumorigenesis. micrornas (mirnas) are small non-coding rna molecules that play a regulatory role in many crucial cellular pathways such as differentiation, cell cycle progression, and apoptosis. a body of evidences showed dysregulation of mirnas in tumor biology and many disease...

2012
Katsuyuki Shirai Michael R. Siedow Arnab Chakravarti

Malignant gliomas have a poor prognosis despite advances in diagnosis and therapy. Although postoperative temozolomide and radiotherapy improve overall survival in glioblastoma patients, most patients experience a recurrence. The prognosis of recurrent malignant gliomas is dismal, and more effective therapeutic strategies are clearly needed. Antiangiogenesis is currently considered an attractiv...

Journal: :JNCI Journal of the National Cancer Institute 2014

2008
Dezheng Dong Min Ni Jianze Li Shigang Xiong Wei Ye Jenilyn J. Virrey Changhui Mao Risheng Ye Miao Wang Louis Dubeau Susan Groshen Florence M. Hofman Amy S. Lee

The unfolded protein response (UPR) is an evolutionarily conserved mechanism that activates both proapoptotic and survival pathways to allow eukaryotic cells to adapt to endoplasmic reticulum (ER) stress. Although the UPR has been implicated in tumorigenesis, its precise role in endogenous cancer remains unclear. A major UPR protective response is the induction of the ER chaperone GRP78/BiP, wh...

Journal: :Cancer control : journal of the Moffitt Cancer Center 1999
Brem

BACKGROUND: There is extraordinary interest in developing angiosuppressive agents for cancer treatment. Several new agents appear promising for the treatment of a variety of human cancers. Current concepts and new agents in clinical trials are the focus of this article. In particular, the introduction of a new treatment for human brain tumors is presented in detail, using an antiangiogenic agen...

Journal: :Cancer research 2008
Dezheng Dong Min Ni Jianze Li Shigang Xiong Wei Ye Jenilyn J Virrey Changhui Mao Risheng Ye Miao Wang Ligaya Pen Louis Dubeau Susan Groshen Florence M Hofman Amy S Lee

The unfolded protein response (UPR) is an evolutionarily conserved mechanism that activates both proapoptotic and survival pathways to allow eukaryotic cells to adapt to endoplasmic reticulum (ER) stress. Although the UPR has been implicated in tumorigenesis, its precise role in endogenous cancer remains unclear. A major UPR protective response is the induction of the ER chaperone GRP78/BiP, wh...

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