[1] Aftershocks follow a well‐defined spatial decay pattern in the intermediate field. Here I investigate the same pattern for foreshocks. Foreshock linear density decays as r over distances r of 0.1–30 km for 15 minutes before magnitude 3–4 mainshocks. This trend is the same as that of the aftershocks within the error of the measurement. This consistency of spatial decay can be explained by th...

background: infectious agents have been suspected as a triggering factor for development of autoimmune thyroid disease (atd). some reports from western countries have suggested association between helicobacter pylori (hp) infection and atd. objective: to investigate the association of atd with cag a seropositivity in a population with high rate and early age of onset of hp infection. methods: i...

TLRs trigger innate immunity by recognizing conserved motifs of microorganisms. Recently, ssRNAs from HIV and influenza virus were shown to trigger TLR7 and 8. Thus, we hypothesized that HIV ssRNA, by triggering TLR7/8, affects HIV pathogenesis. Indeed, HIV ssRNA rendered human lymphoid tissue of tonsillar origin or PBMC barely permissive to HIV replication. The synthetic compound R-848, which ...

The M•,=7.4 Izmit, Turkey earthquake triggered widespread regional seismicity in Greece over a study region extending from 400 km to nearly 1000 km away from the epicenter. Small events began immediately after the passage of the mainshock surface waves suggesting that the transient stresses of the seismic waves were the trigger. The increase in cataloged earthquakes in ordinary continental crus...

Background GnRH agonist triggering is one of the strategies for ovulation triggering and final maturation of oocytes. So , should be notice for luteal phase support in these cycles. At the first it was began for prevention of severe OHSS but it was associated with luteal phase problem and lower pregnancy rate due to luteolysis effects of GnRH agonists. So, two other alternative strategies have ...

We describe a procedure that sensitizes chemotherapy-and tumor necrosis factor-resistant human tumor cell populations in vitro and in nude mouse transplants to the immediate triggering of high rates of cell death by anisomycin, an agent causing activation of stress-activated protein kinases [SAPKs, as defined by P. Cohen (Trends Cell Biol., 7: 353-361, 1997)] including p38/RK and c-jun NH2-term...